Bacteria in Dental Plaque and Atheroma Plaque

NCT ID: NCT07100574

Last Updated: 2025-08-03

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

RECRUITING

Total Enrollment

60 participants

Study Classification

OBSERVATIONAL

Study Start Date

2024-12-10

Study Completion Date

2026-03-30

Brief Summary

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Periodontitis is considered a focus of infection where oral bacteria, bacterial by-products or inflammatory mediators can interact with other parts of the organism via the blood. Periodontal pathogen components such as DNA, RNA or specific antigens of P. gingivalis, Aggregatibacter actinomycetemcomitans (A. actinomycetemcomitans), Treponema denticola (T. denticola), Fusobacterium nucleatum (F. nucleatum) or Campylobacter rectus have been isolated from different tissues such as atheroma plaques, placenta, amniotic sac and respiratory tract. It has been reported that periodontitis may contribute to endothelial dysfunction leading to the formation of atherosclerosis.

Detailed Description

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Periodontitis is considered a focus of infection where oral bacteria, bacterial by-products or inflammatory mediators can interact with other parts of the organism via the blood. Periodontal pathogen components such as DNA, RNA or specific antigens of P. gingivalis, Aggregatibacter actinomycetemcomitans (A. actinomycetemcomitans), Treponema denticola (T. denticola), Fusobacterium nucleatum (F. nucleatum) or Campylobacter rectus have been isolated from different tissues such as atheroma plaques, placenta, amniotic sac and respiratory tract. It has been reported that periodontitis may contribute to endothelial dysfunction leading to the formation of atherosclerosis.

Severe chronic generalized periodontitis may result in systemic inflammation and endothelial dysfunction. Periodontal bacteria produce endotoxins in the form of lipopolysaccharide (LPS). Circulating LPS causes an early systemic inflammatory response in the intima.

The aim of this study is to assess the relationship between bacteria in atheroma plaque, and dental plaque in patients grouped using the new periodontal classification. In addition, the relationship between demographic findings and bacterial count and frequency will be evaluated.

Conditions

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Gingival Inflammation Dental Plaque Atheroma Plaque Oral Bacteria

Study Design

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Observational Model Type

OTHER

Study Time Perspective

CROSS_SECTIONAL

Study Groups

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Patients diagnosed with endarterectomy

atheroma plaque

Intervention Type OTHER

During the endarterectomy procedure, samples will be collected for study from some of the atheroma plaques routinely taken for pathology from patients.

dental plaque

Intervention Type OTHER

Subgingival plaque samples will be collected from patients before the endarterectomy procedure.

Interventions

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atheroma plaque

During the endarterectomy procedure, samples will be collected for study from some of the atheroma plaques routinely taken for pathology from patients.

Intervention Type OTHER

dental plaque

Subgingival plaque samples will be collected from patients before the endarterectomy procedure.

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* Diagnosed with endarterectomy
* Not receiving chemotherapy
* Not having received this treatment before

Exclusion Criteria

* Having systemic infection
* Patients with severe heart disease
Minimum Eligible Age

18 Years

Maximum Eligible Age

85 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Istanbul Saglik Bilimleri University

OTHER

Sponsor Role lead

Responsible Party

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Ayşe Toraman

associate professor

Responsibility Role PRINCIPAL_INVESTIGATOR

Locations

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Health Sciences University, Hamidiye Faculty of Dentistry, Department of Periodontology

Istanbul, , Turkey (Türkiye)

Site Status RECRUITING

Countries

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Turkey (Türkiye)

Central Contacts

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Ayşe Toraman

Role: CONTACT

05385025422

Facility Contacts

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Ayşe Toraman

Role: primary

05385025422

Other Identifiers

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1417

Identifier Type: -

Identifier Source: org_study_id

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