Antioxidant Supplementation in Patients With Kashin-Beck Disease

NCT ID: NCT00376025

Last Updated: 2008-02-22

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

PHASE2

Total Enrollment

300 participants

Study Classification

INTERVENTIONAL

Study Start Date

2007-07-31

Study Completion Date

2009-04-30

Brief Summary

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The purpose of this study is to determine whether antioxidant supplementation can have a positive health effect on patients suffering from Kashin-Beck disease.

Detailed Description

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Current research regarding Kashin-Beck disease, (KBD) have identified dramatic deficiencies of both selenium and iodine in patients with this disease. Initial supplementation of these trace minerals provided no measurable benefit to the affected population. Research conducted by Innovative Humanitarian Solutions, suggests that such deficiencies may not be causal, but markers of an underlying condition of extreme oxidative stress brought on by the improper functioning of the Glutathione Peroxidase enzyme in synthesizing H202 during periods of critical cellular development, primarily in mesenchymal cell development.

The purpose of this trial is to determine the efficacy of antioxidant supplementation in aiding the Glutathione Peroxidase enzyme in its proper function and thereby reducing oxidative stress and enabling the uptake of selenium and iodine which are necessary for proper bone growth and development.

Conditions

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Kashin-Beck Disease

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

SINGLE_GROUP

Primary Study Purpose

TREATMENT

Blinding Strategy

TRIPLE

Participants Caregivers Investigators

Interventions

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Biological Antioxidant Supplementation

Supplementation of affected population with the Phytochemical antioxidant, Garcinia Mangostata in addition to sodium selenate.

Intervention Type DIETARY_SUPPLEMENT

Eligibility Criteria

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Inclusion Criteria

* Clinically diagnosed patients with Kashin-Beck disease

Exclusion Criteria

* less than 24 hours from admission to ICU
* Patients who are moribund
* Lack of commitment to program
* Absolute contraindication to enteral nutrients
* Severe acquired brain injury
* Pregnant or lactating patients
* Previous randomization in this study
* Enrollment in a related interventional study
* Child's class C liver disease Metastatic cancer with life expectancy \< 6 months Seizure disorder requiring anticonvulsant medication
Minimum Eligible Age

9 Years

Maximum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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University of Houston - Victoria

OTHER

Sponsor Role collaborator

Innovative Humanitarian Solutions

OTHER

Sponsor Role lead

Responsible Party

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University of Houston - Victoria

Principal Investigators

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Richard Gunasekera, Ph.D.

Role: STUDY_CHAIR

University of Houston - Victoria

Jeff C Cokenour, B.S.

Role: PRINCIPAL_INVESTIGATOR

Innovative Humanitarian Solutions

Minh Han, M.D.

Role: PRINCIPAL_INVESTIGATOR

Innovative Humanitarian Solutions

Locations

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Lhasa Prefecture and surrounding villages

Lhasa, Tibet, China

Site Status RECRUITING

Countries

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China

Central Contacts

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Jeff C Cokenour, B.S.

Role: CONTACT

832-863-5690

Don Heath, B.S.

Role: CONTACT

References

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Moreno-Reyes R, Egrise D, Boelaert M, Goldman S, Meuris S. Iodine deficiency mitigates growth retardation and osteopenia in selenium-deficient rats. J Nutr. 2006 Mar;136(3):595-600. doi: 10.1093/jn/136.3.595.

Reference Type BACKGROUND
PMID: 16484530 (View on PubMed)

Kohrle J, Jakob F, Contempre B, Dumont JE. Selenium, the thyroid, and the endocrine system. Endocr Rev. 2005 Dec;26(7):944-84. doi: 10.1210/er.2001-0034. Epub 2005 Sep 20.

Reference Type BACKGROUND
PMID: 16174820 (View on PubMed)

Xia Y, Hill KE, Byrne DW, Xu J, Burk RF. Effectiveness of selenium supplements in a low-selenium area of China. Am J Clin Nutr. 2005 Apr;81(4):829-34. doi: 10.1093/ajcn/81.4.829.

Reference Type BACKGROUND
PMID: 15817859 (View on PubMed)

Esworthy RS, Yang L, Frankel PH, Chu FF. Epithelium-specific glutathione peroxidase, Gpx2, is involved in the prevention of intestinal inflammation in selenium-deficient mice. J Nutr. 2005 Apr;135(4):740-5. doi: 10.1093/jn/135.4.740.

Reference Type BACKGROUND
PMID: 15795427 (View on PubMed)

Beckett GJ, Arthur JR. Selenium and endocrine systems. J Endocrinol. 2005 Mar;184(3):455-65. doi: 10.1677/joe.1.05971.

Reference Type BACKGROUND
PMID: 15749805 (View on PubMed)

Moreno-Reyes R, Mathieu F, Boelaert M, Begaux F, Suetens C, Rivera MT, Neve J, Perlmutter N, Vanderpas J. Selenium and iodine supplementation of rural Tibetan children affected by Kashin-Beck osteoarthropathy. Am J Clin Nutr. 2003 Jul;78(1):137-44. doi: 10.1093/ajcn/78.1.137.

Reference Type BACKGROUND
PMID: 12816783 (View on PubMed)

Stamler J, Stamler R, Neaton JD, Wentworth D, Daviglus ML, Garside D, Dyer AR, Liu K, Greenland P. Low risk-factor profile and long-term cardiovascular and noncardiovascular mortality and life expectancy: findings for 5 large cohorts of young adult and middle-aged men and women. JAMA. 1999 Dec 1;282(21):2012-8. doi: 10.1001/jama.282.21.2012.

Reference Type BACKGROUND
PMID: 10591383 (View on PubMed)

Harris NS, Crawford PB, Yangzom Y, Pinzo L, Gyaltsen P, Hudes M. Nutritional and health status of Tibetan children living at high altitudes. N Engl J Med. 2001 Feb 1;344(5):341-7. doi: 10.1056/NEJM200102013440504.

Reference Type BACKGROUND
PMID: 11172165 (View on PubMed)

Utiger RD. Kashin-Beck disease--expanding the spectrum of iodine-deficiency disorders. N Engl J Med. 1998 Oct 15;339(16):1156-8. doi: 10.1056/NEJM199810153391611. No abstract available.

Reference Type BACKGROUND
PMID: 9770565 (View on PubMed)

Chanoine JP. Selenium and thyroid function in infants, children and adolescents. Biofactors. 2003;19(3-4):137-43. doi: 10.1002/biof.5520190306.

Reference Type BACKGROUND
PMID: 14757964 (View on PubMed)

Molecular Biology of selenium with its implications for metabolism. Raymond F. Burke, Division of Gastroenterology, division of Medicine and center in molecular toxicology, Vanderbilt University school of medicine, Nashville, Tenn 37232, USA

Reference Type BACKGROUND

Other Identifiers

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KB001

Identifier Type: -

Identifier Source: org_study_id

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