Physical Training and the Incretin Effect

NCT ID: NCT01698502

Last Updated: 2014-04-24

Study Results

Results available

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Basic Information

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Recruitment Status

COMPLETED

Total Enrollment

21 participants

Study Classification

OBSERVATIONAL

Study Start Date

2012-08-31

Study Completion Date

2013-06-30

Brief Summary

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It is well known that continuous physical exercise leads to a number of changes in the body. Maximal oxygen uptake; the heart's pumping ability and muscle mass and strength increases. Also the metabolism adapts: The ability to oxidize fat increase and the insulin sensitivity in primarily in muscle, but also in the liver increase.

Also endocrine glands adapts according to the level of physical activity. It is known that in healthy, younger people the insulin secretion from the pancreas after administration of sugar consumed orally or given directly into a vein, is significantly lower in trained individuals compared with untrained. This change does, however, not only apply to glucose, as also stimulation by the amino acid arginine, shows the same pattern.

It seems plausible that the endocrine glands/cells adapts to the level of physical training, but this has not yet been investigated.

The gastrointestinal tract is the birthplace of a variety of hormones. One group of these is called incretin hormones. They stimulate the glucose dependant insulin secretion in the pancreas and affect hunger/satiety. Whether the incretin production and thus their concentration in the blood is regulated by physical training is unknown.

Obese and patients with type 2 diabetes, has, in contrast to well-trained, decreased insulin sensitivity. As a consequence their (type 2 diabetics, at least early in their disease course) meal stimulated insulin release is greater than in healthy, normal weight individuals. This in spite of the fact that the incretin effect is reduced in obese people and patients with type 2 diabetes compared to healthy, normal weight.

Whether physical training affects both the secretion of incretins and the incretin effect has not yet been studied.

The purpose of this study is to investigate whether incretin hormones in physical well-trained young men have a changed effect on insulin secretion from the pancreas compared to untrained young men. A difference may indicate that the body's endocrine glands adapts to training mode.

The investigators hypothesis is that incretin hormones have a decreased effect on the glucose dependant insulin release in physically trained persons and thus results in a lower insulin release at any given plasma glucose level.

Detailed Description

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Conditions

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Healthy

Study Design

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Observational Model Type

CASE_CROSSOVER

Study Time Perspective

CROSS_SECTIONAL

Study Groups

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Trained

Healthy, Endurance trained (Maximal oxygen uptake (VO2max), ml\*min-1\*kg-1\>60), 20-30 year, BMI: 18,5-25kg/m2, males.

No interventions assigned to this group

Untrained

Healthy, sedentary (Maximal oxygen uptake (VO2max), ml\*min-1\*kg-1\<50), 20-30 year, BMI: 18,5-25kg/m2, males.

No interventions assigned to this group

Eligibility Criteria

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Inclusion Criteria

* Sedentary: 50\>Maximal oxygen uptake (VO2max), ml\*min-1\*kg-1 OR endurance trained VO2max, ml\*min-1\*kg-1\>60
* 20-30 years
* BMI: 18,5-25kg/m2
* Caucasian
* Healthy

Exclusion Criteria

* Any kind of medication or diabetes in the family (parents, siblings),
* Non-caucasian
Minimum Eligible Age

20 Years

Maximum Eligible Age

30 Years

Eligible Sex

MALE

Accepts Healthy Volunteers

Yes

Sponsors

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University of Copenhagen

OTHER

Sponsor Role lead

Responsible Party

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Michael Taulo Lund

MD. PhD. Student

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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Flemming Dela, Prof. MD

Role: STUDY_CHAIR

University of Copenhagen, Dep. of Biomedical Sciences, Center of healthy Ageing, XLAB

Locations

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University of Copenhagen, Faculty of Health Sciences

Copenhagen, North, Denmark

Site Status

Countries

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Denmark

References

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Dela F, Handberg A, Mikines KJ, Vinten J, Galbo H. GLUT 4 and insulin receptor binding and kinase activity in trained human muscle. J Physiol. 1993 Sep;469:615-24. doi: 10.1113/jphysiol.1993.sp019833.

Reference Type BACKGROUND
PMID: 8271219 (View on PubMed)

Dela F, Mikines KJ, von Linstow M, Secher NH, Galbo H. Effect of training on insulin-mediated glucose uptake in human muscle. Am J Physiol. 1992 Dec;263(6):E1134-43. doi: 10.1152/ajpendo.2006.263.6.E1134.

Reference Type BACKGROUND
PMID: 1476187 (View on PubMed)

Mikines KJ, Sonne B, Farrell PA, Tronier B, Galbo H. Effect of training on the dose-response relationship for insulin action in men. J Appl Physiol (1985). 1989 Feb;66(2):695-703. doi: 10.1152/jappl.1989.66.2.695.

Reference Type BACKGROUND
PMID: 2651385 (View on PubMed)

Dela F, Mikines KJ, Von Linstow M, Galbo H. Effect of training on response to a glucose load adjusted for daily carbohydrate intake. Am J Physiol. 1991 Jan;260(1 Pt 1):E14-20. doi: 10.1152/ajpendo.1991.260.1.E14.

Reference Type BACKGROUND
PMID: 1987787 (View on PubMed)

King DS, Dalsky GP, Staten MA, Clutter WE, Van Houten DR, Holloszy JO. Insulin action and secretion in endurance-trained and untrained humans. J Appl Physiol (1985). 1987 Dec;63(6):2247-52. doi: 10.1152/jappl.1987.63.6.2247.

Reference Type BACKGROUND
PMID: 3325486 (View on PubMed)

Mikines KJ, Sonne B, Tronier B, Galbo H. Effects of training and detraining on dose-response relationship between glucose and insulin secretion. Am J Physiol. 1989 May;256(5 Pt 1):E588-96. doi: 10.1152/ajpendo.1989.256.5.E588.

Reference Type BACKGROUND
PMID: 2655469 (View on PubMed)

Dupre J, Ross SA, Watson D, Brown JC. Stimulation of insulin secretion by gastric inhibitory polypeptide in man. J Clin Endocrinol Metab. 1973 Nov;37(5):826-8. doi: 10.1210/jcem-37-5-826. No abstract available.

Reference Type BACKGROUND
PMID: 4749457 (View on PubMed)

Holst JJ, Orskov C, Nielsen OV, Schwartz TW. Truncated glucagon-like peptide I, an insulin-releasing hormone from the distal gut. FEBS Lett. 1987 Jan 26;211(2):169-74. doi: 10.1016/0014-5793(87)81430-8.

Reference Type BACKGROUND
PMID: 3542566 (View on PubMed)

Nauck MA, Heimesaat MM, Orskov C, Holst JJ, Ebert R, Creutzfeldt W. Preserved incretin activity of glucagon-like peptide 1 [7-36 amide] but not of synthetic human gastric inhibitory polypeptide in patients with type-2 diabetes mellitus. J Clin Invest. 1993 Jan;91(1):301-7. doi: 10.1172/JCI116186.

Reference Type BACKGROUND
PMID: 8423228 (View on PubMed)

Knop FK, Aaboe K, Vilsboll T, Volund A, Holst JJ, Krarup T, Madsbad S. Impaired incretin effect and fasting hyperglucagonaemia characterizing type 2 diabetic subjects are early signs of dysmetabolism in obesity. Diabetes Obes Metab. 2012 Jun;14(6):500-10. doi: 10.1111/j.1463-1326.2011.01549.x. Epub 2012 Jan 17.

Reference Type BACKGROUND
PMID: 22171657 (View on PubMed)

Other Identifiers

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EXINT2_FD

Identifier Type: -

Identifier Source: org_study_id

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