Effect of Food Temperature and Diet Composition on Satiety, Satiety Hormones, Chewing Time and Neuronal Activity

NCT ID: NCT05822167

Last Updated: 2024-07-24

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Clinical Phase

NA

Total Enrollment

13 participants

Study Classification

INTERVENTIONAL

Study Start Date

2023-04-20

Study Completion Date

2023-08-28

Brief Summary

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Obesity and its related illnesses have become serious health issues, obesity is today the fifth most common cause of death. Obesity rate has dramatically enhanced in both male and female, and across all ages. Food and energy intake during habitual meals, energy balance, energy expended during physical activity, all play an important role in management of weight.

Lifestyle changes and nutritional strategies are emerging as the best line of treatment for obesity. The achievement of satiety along with, the reduction in dietary intake is the primary goal of nutritionists and food scientists. Appetite control can be defined by two terms; satiety and satiation. The interaction between appetite, food intake, and hormones secreted by the gastrointestinal tract, which are secreted in response to macronutrients like carbohydrates, fats and proteins are the satiety regulators. The gut hormones including glucagon like peptide 1(GLP1), cholecystokinin (CCK) are anorexigenic in action, cause slowing of meal digestion and reduce food intake thus inducing satiation and satiety. The CCK hormone plays a key role in delaying of stomach emptying by fundus relaxation and antral inhibition, ultimately causing major satiation. The incretin hormone GLP-1's main action is to stimulate insulin secretion, inhibit glucagon secretion, regulating postprandial glucose and provide negative feedback to the stomach thereby controlling appetite. Research is needed in meal properties and different diets which may affect gut-brain signaling and altering the mechanisms of gut hormonal secretion, thus further influencing appetite satiation and satiety scores. This knowledge can be utilized in energy expenditure and weight management.

Serving temperatures alter perceived intensities, flavor and acceptances of food as well. Brain areas work in close association with the thermal perception and emotions. In neuroimaging studies neural changes have been when body is exposed to different temperatures either environmental or oral cavity. Temperature of food play an important role in the palatability and affective value of food and, consequently, in appetite regulation. Limited research has been done so far how food temperature is related to sensory perception and satiety Chewing and food texture also affect satiety and satiation. The oral processing, eating rate and physical forms of food i.e., solid versus liquid or semi-solid are all physiologically related to satiety and an individual's behavior to understand this oral sensory satiety effect, requires further studies. The number of chews has been studied showing an association between reduction in food intake with increasing number of chews. EEG is a noninvasive neuroimaging technique, helping in evaluating the cognitive part of food stimuli and food ingestion in relation to gut hormones. Sensory properties of previously identified as drivers of refreshing perception, enhance alpha and beta brain oscillations as observed in prior EEG studies.

Many factors influence satiety including food composition, temperature, environment, last meal and preload. Limited literature is available about temperature of food and its influence on satiety. My study aims to find 1) the effect of temperature of high carbohydrate, high fat meal and high protein meal on the satiety scores, satiety-related hormones, EEG and EMG. 2. To find the effect of chewing time of food on the satiety scores, satiety- related hormones, EEG and EMG.

Detailed Description

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Conditions

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Obesity, Overweight

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

CROSSOVER

3 interventional Studies 9 arms
Primary Study Purpose

PREVENTION

Blinding Strategy

NONE

Study Groups

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High Protein Meal with cold temperature

Group Type EXPERIMENTAL

High Protein Meal at Cold Temperature

Intervention Type OTHER

High Protein Meal providing 500kcal with 60% energy from protein, 30% fat, 10% carbohydrate.

Cold meal will be served at 25 degree C and below.

High Protein Meal with warm temperature

Group Type EXPERIMENTAL

High Protein Meal at Warm Temperarute

Intervention Type OTHER

High Protein Meal providing 500kcal with 60% energy from protein, 30% fat, 10% carbohydrate.

Meal will be served at warm temperature i.e., between 40 degree C and 60 degree C.

High Protein Meal with Hot temperature

Group Type EXPERIMENTAL

High Protein Meal at Hot Temperature

Intervention Type OTHER

High Protein Meal providing 500kcal with 60% energy from protein, 30% fat, 10% carbohydrate.

Meal will be served at hot temperature i.e., 60 degree C and above.

High Carbohydrate Meal with cold temperature

Group Type EXPERIMENTAL

High Carbohydrate Meal at Cold Temperature

Intervention Type OTHER

High carbohydrate meal providing 500 kcal with 65% of energy from carbohydrates, 25% from proteins and 10% from fats.

Cold meal will be served at 25 degree C and below.

High Carbohydrate Meal with warm temperature

Group Type EXPERIMENTAL

High Carbohydrate Meal at Warm Temperature

Intervention Type OTHER

High carbohydrate meal providing 500 kcal with 65% of energy from carbohydrates, 25% from proteins and 10% from fats.

Meal will be served at warm temperature i.e., between 40 degree C and 60 degree C.

High Carbohydrate Meal with hot temperature

Group Type EXPERIMENTAL

High Carbohydrate Meal at Hot Temperature

Intervention Type OTHER

High carbohydrate meal providing 500 kcal with 65% of energy from carbohydrates, 25% from proteins and 10% from fats.

Meal will be served at hot temperature i.e., 60 degree C and above.

High Fat Meal with cold temperature

Group Type EXPERIMENTAL

High Fat Meal at Cold Temperature

Intervention Type OTHER

High fat meal will contain 60% fat, 30% protein, 10% carbohydrate. Cold meal will be served at 25 degree C and below.

High Fat Meal with warm temperature

Group Type EXPERIMENTAL

High Fat Meal at Warm Temperature

Intervention Type OTHER

High fat meal will contain 60% fat, 30% protein, 10% carbohydrate. Meal will be served at warm temperature i.e., between 40 degree C and 60 degree C.

High Fat Meal with hot temperature

Group Type EXPERIMENTAL

High Fat Meal at Hot Temperature

Intervention Type OTHER

High fat meal will contain 60% fat, 30% protein, 10% carbohydrate. Meal will be served at hot temperature i.e., 60 degree C and above.

Interventions

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High Protein Meal at Cold Temperature

High Protein Meal providing 500kcal with 60% energy from protein, 30% fat, 10% carbohydrate.

Cold meal will be served at 25 degree C and below.

Intervention Type OTHER

High Protein Meal at Warm Temperarute

High Protein Meal providing 500kcal with 60% energy from protein, 30% fat, 10% carbohydrate.

Meal will be served at warm temperature i.e., between 40 degree C and 60 degree C.

Intervention Type OTHER

High Protein Meal at Hot Temperature

High Protein Meal providing 500kcal with 60% energy from protein, 30% fat, 10% carbohydrate.

Meal will be served at hot temperature i.e., 60 degree C and above.

Intervention Type OTHER

High Carbohydrate Meal at Cold Temperature

High carbohydrate meal providing 500 kcal with 65% of energy from carbohydrates, 25% from proteins and 10% from fats.

Cold meal will be served at 25 degree C and below.

Intervention Type OTHER

High Carbohydrate Meal at Warm Temperature

High carbohydrate meal providing 500 kcal with 65% of energy from carbohydrates, 25% from proteins and 10% from fats.

Meal will be served at warm temperature i.e., between 40 degree C and 60 degree C.

Intervention Type OTHER

High Carbohydrate Meal at Hot Temperature

High carbohydrate meal providing 500 kcal with 65% of energy from carbohydrates, 25% from proteins and 10% from fats.

Meal will be served at hot temperature i.e., 60 degree C and above.

Intervention Type OTHER

High Fat Meal at Cold Temperature

High fat meal will contain 60% fat, 30% protein, 10% carbohydrate. Cold meal will be served at 25 degree C and below.

Intervention Type OTHER

High Fat Meal at Warm Temperature

High fat meal will contain 60% fat, 30% protein, 10% carbohydrate. Meal will be served at warm temperature i.e., between 40 degree C and 60 degree C.

Intervention Type OTHER

High Fat Meal at Hot Temperature

High fat meal will contain 60% fat, 30% protein, 10% carbohydrate. Meal will be served at hot temperature i.e., 60 degree C and above.

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* Healthy individuals of age 25- 35 years
* BMI ranged between 18.5 and 24.9

Exclusion Criteria

* Chronic diseases, diabetes, endocrine disorder,
* Any bariatric surgery that interfered with gastrointestinal functions,
* Smoking,
* Dieting,
* Pregnancy, lactation,
* Taking medication or supplements.
* Psychiatry illnesses or dental problems will be excluded.
* Females with the history of premenstrual syndrome will be excluded.
* Gastrointestinal Complications
* Following Special diets
* Food allergy to food used in the trial
Minimum Eligible Age

25 Years

Maximum Eligible Age

35 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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Khyber Medical University Peshawar

OTHER

Sponsor Role lead

Responsible Party

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Bibi Hajira

Assistant Professor

Responsibility Role PRINCIPAL_INVESTIGATOR

Locations

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Khyber Medical University

Peshawar, KPK, Pakistan

Site Status

Countries

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Pakistan

References

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Rexrode KM, Carey VJ, Hennekens CH, Walters EE, Colditz GA, Stampfer MJ, Willett WC, Manson JE. Abdominal adiposity and coronary heart disease in women. JAMA. 1998 Dec 2;280(21):1843-8. doi: 10.1001/jama.280.21.1843.

Reference Type BACKGROUND
PMID: 9846779 (View on PubMed)

McMillan DC, Sattar N, McArdle CS. ABC of obesity. Obesity and cancer. BMJ. 2006 Nov 25;333(7578):1109-11. doi: 10.1136/bmj.39042.565035.BE1. No abstract available.

Reference Type BACKGROUND
PMID: 17124223 (View on PubMed)

Chambers L, Mc Crickerd K, Yeomans M. R. Optimising foods for satiety.Trends Food Sci. Technol. 2015; 41: 149-160. https ://doi.org/10.1016/j.tifs.2014.10.007

Reference Type BACKGROUND

Blundell J. E, De Graaf K , Finlayson G, Halford J. C, Hetherington M, KingN, & Stubbs J. Assessment Methods for Eating Behaviour and Weight-Related Problems: Measures, Theory and Research. 2009; 283-325 (Sage, Thousand Oaks).

Reference Type BACKGROUND

Camilleri M. Peripheral mechanisms in appetite regulation. Gastroenterology. 2015 May;148(6):1219-33. doi: 10.1053/j.gastro.2014.09.016. Epub 2014 Sep 21.

Reference Type BACKGROUND
PMID: 25241326 (View on PubMed)

van der Klaauw AA, Keogh JM, Henning E, Trowse VM, Dhillo WS, Ghatei MA, Farooqi IS. High protein intake stimulates postprandial GLP1 and PYY release. Obesity (Silver Spring). 2013 Aug;21(8):1602-7. doi: 10.1002/oby.20154. Epub 2013 May 13.

Reference Type BACKGROUND
PMID: 23666746 (View on PubMed)

Strader AD, Woods SC. Gastrointestinal hormones and food intake. Gastroenterology. 2005 Jan;128(1):175-91. doi: 10.1053/j.gastro.2004.10.043.

Reference Type BACKGROUND
PMID: 15633135 (View on PubMed)

Gibbons C, Finlayson G, Caudwell P, Webb DL, Hellstrom PM, Naslund E, Blundell JE. Postprandial profiles of CCK after high fat and high carbohydrate meals and the relationship to satiety in humans. Peptides. 2016 Mar;77:3-8. doi: 10.1016/j.peptides.2015.09.010. Epub 2015 Sep 30.

Reference Type BACKGROUND
PMID: 26429068 (View on PubMed)

Tanaka M, Nagashima K, McAllen RM, Kanosue K. Role of the medullary raphe in thermoregulatory vasomotor control in rats. J Physiol. 2002 Apr 15;540(Pt 2):657-64. doi: 10.1113/jphysiol.2001.012989.

Reference Type BACKGROUND
PMID: 11956351 (View on PubMed)

Rolls ET. The affective and cognitive processing of touch, oral texture, and temperature in the brain. Neurosci Biobehav Rev. 2010 Feb;34(2):237-45. doi: 10.1016/j.neubiorev.2008.03.010. Epub 2008 Apr 3.

Reference Type BACKGROUND
PMID: 18468687 (View on PubMed)

Miquel-Kergoat S, Azais-Braesco V, Burton-Freeman B, Hetherington MM. Effects of chewing on appetite, food intake and gut hormones: A systematic review and meta-analysis. Physiol Behav. 2015 Nov 1;151:88-96. doi: 10.1016/j.physbeh.2015.07.017. Epub 2015 Jul 15.

Reference Type BACKGROUND
PMID: 26188140 (View on PubMed)

Robinson E, Almiron-Roig E, Rutters F, de Graaf C, Forde CG, Tudur Smith C, Nolan SJ, Jebb SA. A systematic review and meta-analysis examining the effect of eating rate on energy intake and hunger. Am J Clin Nutr. 2014 Jul;100(1):123-51. doi: 10.3945/ajcn.113.081745. Epub 2014 May 21.

Reference Type BACKGROUND
PMID: 24847856 (View on PubMed)

Schlogl H, Horstmann A, Villringer A, Stumvoll M. Functional neuroimaging in obesity and the potential for development of novel treatments. Lancet Diabetes Endocrinol. 2016 Aug;4(8):695-705. doi: 10.1016/S2213-8587(15)00475-1. Epub 2016 Jan 30.

Reference Type BACKGROUND
PMID: 26838265 (View on PubMed)

Hallschmid M, Molle M, Fischer S, Born J. EEG synchronization upon reward in man. Clin Neurophysiol. 2002 Jul;113(7):1059-65. doi: 10.1016/s1388-2457(02)00142-6.

Reference Type BACKGROUND
PMID: 12088700 (View on PubMed)

Other Identifiers

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KMU/IBMS/IERB/2022/9303-7

Identifier Type: -

Identifier Source: org_study_id

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