Long-term Investigation of Resveratrol on Fat Metabolism in Obese Men With Nonalcoholic Fatty Liver Disease

NCT ID: NCT01446276

Last Updated: 2014-04-28

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Clinical Phase

NA

Total Enrollment

26 participants

Study Classification

INTERVENTIONAL

Study Start Date

2011-11-30

Study Completion Date

2014-04-30

Brief Summary

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The purpose of this study is to investigate potential metabolic effects of resveratrol in obese healthy men with non-alcoholic fatty liver disease.

The investigators hypothesize that resveratrol will:

* decrease hepatic very-low-density-lipoprotein-triglyceride (VLDL-TG) secretion
* decrease liver fat content
* increase insulin sensitivity

The investigators will look at changes in:

* lipid turnover (VLDL-TG kinetics, palmitate kinetics, indirect calorimetry)
* liver fat content (MR liver spectroscopy)
* insulin sensitivity (glucose kinetics during hyperinsulinaemic euglycaemic clamp)
* body composition (DXA and MRI)
* lipase activity and fat cell size (fat biopsy from abdominal and femoral adipose tissue)

Detailed Description

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Conditions

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Obesity Nonalcoholic Fatty Liver Disease

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

BASIC_SCIENCE

Blinding Strategy

QUADRUPLE

Participants Caregivers Investigators Outcome Assessors

Study Groups

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Resveratrol

Resveratrol 500mg 3 times daily for six month

Group Type EXPERIMENTAL

Resveratrol

Intervention Type DIETARY_SUPPLEMENT

500 mg 3 times daily for six month

Placebo

Placebo 1 tablet 3 times daily for six month

Group Type PLACEBO_COMPARATOR

Placebo

Intervention Type OTHER

1 placebo tablet 3 times daily for six month

Control group

Men without non-alcoholic fatty liver disease

Group Type NO_INTERVENTION

No interventions assigned to this group

Interventions

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Resveratrol

500 mg 3 times daily for six month

Intervention Type DIETARY_SUPPLEMENT

Placebo

1 placebo tablet 3 times daily for six month

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* Male
* 25-65 years
* Obesity (BMI \> 28 kg/m2, waist/hip ratio \> 0,95)
* Have nonalcoholic fatty liver disease (NAFLD)(intervention group) or do not have NAFLD (control group)
* May have hypertension and/or hypercholesterolemia
* Written informed consent

Exclusion Criteria

* Any other disease than NAFLD (e.g. diabetes, thyroid or parathyroid disease, heart, liver or kidney disease)
* Present and previous malignancy
* Alcohol dependency (more than 21 units of alcohol per week)
* History of smoking
* Participation in studies with radioactive isotopes within the last six months
* Hemoglobin under normal range regarding to sex (under 8.3 mmol/l for men)
Minimum Eligible Age

25 Years

Maximum Eligible Age

65 Years

Eligible Sex

MALE

Accepts Healthy Volunteers

No

Sponsors

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The Ministry of Science, Technology and Innovation, Denmark

OTHER_GOV

Sponsor Role collaborator

University of Aarhus

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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Søren Nielsen, MD, associate professor, DMSc

Role: STUDY_CHAIR

Department of Endocrinology and Internal Medicine

Locations

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Department of Endocrinology and Internal Medicine

Aarhus C, , Denmark

Site Status

Countries

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Denmark

References

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Poulsen MK, Nellemann B, Bibby BM, Stodkilde-Jorgensen H, Pedersen SB, Gronbaek H, Nielsen S. No effect of resveratrol on VLDL-TG kinetics and insulin sensitivity in obese men with nonalcoholic fatty liver disease. Diabetes Obes Metab. 2018 Oct;20(10):2504-2509. doi: 10.1111/dom.13409. Epub 2018 Jul 5.

Reference Type DERIVED
PMID: 29885082 (View on PubMed)

Poulsen MK, Nellemann B, Stodkilde-Jorgensen H, Pedersen SB, Gronbaek H, Nielsen S. Impaired Insulin Suppression of VLDL-Triglyceride Kinetics in Nonalcoholic Fatty Liver Disease. J Clin Endocrinol Metab. 2016 Apr;101(4):1637-46. doi: 10.1210/jc.2015-3476. Epub 2016 Feb 1.

Reference Type DERIVED
PMID: 26829441 (View on PubMed)

Other Identifiers

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M-20110172A

Identifier Type: -

Identifier Source: org_study_id

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