Efficacy of PLDD Versus ESI in the Treatment of Lumbar Radicular Pain

NCT ID: NCT05601791

Last Updated: 2022-11-03

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

NA

Total Enrollment

116 participants

Study Classification

INTERVENTIONAL

Study Start Date

2021-11-30

Study Completion Date

2024-07-30

Brief Summary

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The most common cause of lumbar radicular pain is intervertebral disc herniation with or without pressure on the nerve root, which leads to inflammation and pain. Just as the mechanical component is important, so is the inflammatory component in the etiology of lumbar radicular pain. Numerous pro-inflammatory and anti-inflammatory proteins were found in serum, cerebrospinal fluid and disc biopsies from patients with lumbar radicular pain. Interleukin(IL)-1β, IL-6, IL-8, and tumor necrosis factor TNF-α are the most frequently investigated (8, 9).

Elevated levels of IL-6 and TNF-α were found in patients with lumbar pain caused by intervertebral disc herniation.

In order to avoid systemic and unwanted effects of analgesics, undergoing anesthesia and long-term and extensive operations, minimally invasive procedures are increasingly used in the treatment of lumbar radicular pain. Epidural administration of steroids and local anesthetic through a transforaminal approach (ESI TF) and percutaneous laser disc decompression (PLDD) are some of these methods.

Lumbar radicular pain occurs due to inflammation and/or disc-radicular contact. Corticosteroids interrupt the inflammatory process, the transmission of pain signals via nociceptive C fibers and reduce capillary permeability. Along with the corticosteroid, a local anesthetic is also applied, which leads to immediate analgesia by blocking the conduction of painful impulses by blocking sodium channels. Percutaneous laser disc decompression (PLDD) is a minimally invasive method of treating lumbar radicular pain performed under local anesthesia under fluoroscopic control. The laser energy leads to the heating of the tissue of the nucleus pulposus, which leads to the evaporation of a small volume of water inside the disc. Viewing the disc as a closed hydraulic system, a small decrease in the water content within the disc leads to a disproportionate decrease in intradiscal pressure, which results in retraction of the herniated disc. Thermal energy leads to protein denaturation, which causes structural changes and thus prevents further retention of water in the disc, and a stable scar is created at the point of laser action. On the basis of current knowledge, an attempt is made to establish a link between inflammatory parameters as predictive and prognostic biomarkers in the treatment of patients with lumbar radicular pain caused by intervertebral disc herniation.

Detailed Description

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The hypothesis is that:

* there will be no difference in effectiveness in reducing the intensity of pain, improving the quality of life, reducing the degree of disability, reducing neuropathic pain, reducing anxiety and depression, and improving the quality of sleep in the treatment of lumbar radicular pain caused by disc herniation without discoradicular contact, but PLDD will be more effective in patients where there is discorradicular contact
* PLDD will lead to a greater reduction in serum levels of inflammatory markers in patients with nerve compression by intervertebral disc herniation
* ESI TF will lead to a greater reduced serum level of inflammatory markers where there is no nerve compression by intervertebral disc herniation
* The group of patients in whom PLDD was performed will have a significantly greater retraction of the disc herniation compared to ESI TF

Conditions

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Herniated Disc Low Back Pain

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

The research is designed as a randomized controlled trial, and will be conducted at the Clinic for Anesthesiology, Reanimatology and Intensive Medicine in the Department of Pain Management of the Osijek Clinical Hospital Center after obtaining the approval of the Ethics Committee. All patients will receive a written informed consent in which the procedure to be performed will be described, and the procedure will also be explained to them orally. After signing the informed consent, the patients will be divided into 2 groups using a random number generator, depending on whether there is discorradicular contact or not, and then according to the treatment method (ESI TF or PLDD). All subjects will be selected at the Institute for Pain Management of KBC Osijek.
Primary Study Purpose

TREATMENT

Blinding Strategy

NONE

Study Groups

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Discoradicular contact+ESI TF

Patients with discorradicular contact who underwent ESI TF

Group Type ACTIVE_COMPARATOR

Epidural Steroid Injection

Intervention Type PROCEDURE

Epidural Steroid Injection transforaminal approach Drug: midazolam, fentanyl, lidocaine, levobupivacaine, methilprednisoloneacetat

Discoradicular contact+PLDD

Patients with discorradicular contact who underwent PLDD

Group Type ACTIVE_COMPARATOR

Percutaneous Laser Disc Decompression

Intervention Type PROCEDURE

Percutaneous Laser Disc Decompression Drug: midazolam, fentanyl, lidocaine

Without discoradicular contact+ESI TF

Patients without discorradicular contact who underwent ESI TF

Group Type ACTIVE_COMPARATOR

Epidural Steroid Injection

Intervention Type PROCEDURE

Epidural Steroid Injection transforaminal approach Drug: midazolam, fentanyl, lidocaine, levobupivacaine, methilprednisoloneacetat

Without discoradicular contact+PLDD

Patients without discorradicular contact who underwent PLDD

Group Type ACTIVE_COMPARATOR

Percutaneous Laser Disc Decompression

Intervention Type PROCEDURE

Percutaneous Laser Disc Decompression Drug: midazolam, fentanyl, lidocaine

Interventions

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Epidural Steroid Injection

Epidural Steroid Injection transforaminal approach Drug: midazolam, fentanyl, lidocaine, levobupivacaine, methilprednisoloneacetat

Intervention Type PROCEDURE

Percutaneous Laser Disc Decompression

Percutaneous Laser Disc Decompression Drug: midazolam, fentanyl, lidocaine

Intervention Type PROCEDURE

Eligibility Criteria

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Inclusion Criteria

* Age between 18 and 60
* Signing informed consent
* Unilateral lumbar radicular pain
* Subjects who do not responds to conservative treatment
* Disc herniation at one level
* MR verified disc herniation
* Pain intensity measured by VAS scale, from 0 - 10, \>5

Exclusion Criteria

* Subjects younger than 18 and older than 65 years
* Refusal of the subjects to participate in the research
* Central stenosis of the lumbar canal
* Lumbar radicular pain caused by causes other than intervertebral disc herniation
* Pregnancy
* Allergy to steroids, local anesthetics, fentanyl, midazolam and contrast medium
* Positive history of prolonged bleeding
* Local or systemic infection
* Previous lumbar spine surgery7
* Opioid abuse
* Proven inflammatory rheumatic disease and inflammatory bowel disease in the active phase
* Other acute infections
Minimum Eligible Age

18 Years

Maximum Eligible Age

65 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Osijek University Hospital

OTHER

Sponsor Role lead

Responsible Party

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Dino Budrovac

MD, Specialist in anesthesiology, resuscitation and intensive care medicine

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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Dino Budrovac, MD

Role: PRINCIPAL_INVESTIGATOR

UH Osijek

Locations

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UH Osijek

Osijek, , Croatia

Site Status RECRUITING

Countries

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Croatia

Central Contacts

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Dino Budrovac, MD

Role: CONTACT

+385995162489

Ivan Radoš, Prof.MD.PhD

Role: CONTACT

+385917974306

Facility Contacts

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Ivan Radoš, Prof.MD,PhD

Role: primary

+385917974306

References

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Hoy D, March L, Brooks P, Blyth F, Woolf A, Bain C, Williams G, Smith E, Vos T, Barendregt J, Murray C, Burstein R, Buchbinder R. The global burden of low back pain: estimates from the Global Burden of Disease 2010 study. Ann Rheum Dis. 2014 Jun;73(6):968-74. doi: 10.1136/annrheumdis-2013-204428. Epub 2014 Mar 24.

Reference Type BACKGROUND
PMID: 24665116 (View on PubMed)

Khan AN, Jacobsen HE, Khan J, Filippi CG, Levine M, Lehman RA Jr, Riew KD, Lenke LG, Chahine NO. Inflammatory biomarkers of low back pain and disc degeneration: a review. Ann N Y Acad Sci. 2017 Dec;1410(1):68-84. doi: 10.1111/nyas.13551.

Reference Type BACKGROUND
PMID: 29265416 (View on PubMed)

Porchet F, Wietlisbach V, Burnand B, Daeppen K, Villemure JG, Vader JP. Relationship between severity of lumbar disc disease and disability scores in sciatica patients. Neurosurgery. 2002 Jun;50(6):1253-9; discussion 1259-60. doi: 10.1097/00006123-200206000-00014.

Reference Type BACKGROUND
PMID: 12015843 (View on PubMed)

el Barzouhi A, Vleggeert-Lankamp CL, Lycklama a Nijeholt GJ, Van der Kallen BF, van den Hout WB, Jacobs WC, Koes BW, Peul WC; Leiden-The Hague Spine Intervention Prognostic Study Group. Magnetic resonance imaging in follow-up assessment of sciatica. N Engl J Med. 2013 Mar 14;368(11):999-1007. doi: 10.1056/NEJMoa1209250.

Reference Type BACKGROUND
PMID: 23484826 (View on PubMed)

Wuertz K, Haglund L. Inflammatory mediators in intervertebral disk degeneration and discogenic pain. Global Spine J. 2013 Jun;3(3):175-84. doi: 10.1055/s-0033-1347299. Epub 2013 May 21.

Reference Type BACKGROUND
PMID: 24436868 (View on PubMed)

Jungen MJ, Ter Meulen BC, van Osch T, Weinstein HC, Ostelo RWJG. Inflammatory biomarkers in patients with sciatica: a systematic review. BMC Musculoskelet Disord. 2019 Apr 9;20(1):156. doi: 10.1186/s12891-019-2541-0.

Reference Type BACKGROUND
PMID: 30967132 (View on PubMed)

Kraychete DC, Sakata RK, Issy AM, Bacellar O, Santos-Jesus R, Carvalho EM. Serum cytokine levels in patients with chronic low back pain due to herniated disc: analytical cross-sectional study. Sao Paulo Med J. 2010;128(5):259-62. doi: 10.1590/s1516-31802010000500003.

Reference Type BACKGROUND
PMID: 21181064 (View on PubMed)

Patel VB, Wasserman R, Imani F. Interventional Therapies for Chronic Low Back Pain: A Focused Review (Efficacy and Outcomes). Anesth Pain Med. 2015 Aug 22;5(4):e29716. doi: 10.5812/aapm.29716. eCollection 2015 Aug.

Reference Type BACKGROUND
PMID: 26484298 (View on PubMed)

Harris JD. Management of expected and unexpected opioid-related side effects. Clin J Pain. 2008 May;24 Suppl 10:S8-S13. doi: 10.1097/AJP.0b013e31816b58eb.

Reference Type BACKGROUND
PMID: 18418226 (View on PubMed)

Momenzadeh S, Koosha A, Kazempoor Monfared M, Bairami J, Zali A, Ommi D, Hosseini B, Hashemi M, Sayadi S, Aryani R, Nematollahi F, Nematollahi L, Barati M. The Effect of Percutaneous Laser Disc Decompression on Reducing Pain and Disability in Patients With Lumbar Disc Herniation. J Lasers Med Sci. 2019 Winter;10(1):29-32. doi: 10.15171/jlms.2019.04. Epub 2018 Dec 15.

Reference Type BACKGROUND
PMID: 31360365 (View on PubMed)

Schneider B, Zheng P, Mattie R, Kennedy DJ. Safety of epidural steroid injections. Expert Opin Drug Saf. 2016 Aug;15(8):1031-9. doi: 10.1080/14740338.2016.1184246. Epub 2016 May 13.

Reference Type BACKGROUND
PMID: 27148630 (View on PubMed)

Choy DS. Percutaneous laser disc decompression. J Clin Laser Med Surg. 1995 Jun;13(3):125-6. doi: 10.1089/clm.1995.13.125.

Reference Type BACKGROUND
PMID: 10150634 (View on PubMed)

Budrovac D, Rados I, Hnatesen D, Harsanji-Drenjancevic I, Tot OK, Katic F, Lukic I, Skiljic S, Neskovic N, Dimitrijevic I. Effectiveness of Epidural Steroid Injection Depending on Discoradicular Contact: A Prospective Randomized Trial. Int J Environ Res Public Health. 2023 Feb 19;20(4):3672. doi: 10.3390/ijerph20043672.

Reference Type DERIVED
PMID: 36834367 (View on PubMed)

Provided Documents

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Document Type: Study Protocol, Statistical Analysis Plan, and Informed Consent Form

View Document

Other Identifiers

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2158-61-07-20-05

Identifier Type: -

Identifier Source: org_study_id

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