Ventilatory Efficiency in Critically Ill COVID-19 Patients

NCT ID: NCT04694742

Last Updated: 2021-01-05

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Total Enrollment

100 participants

Study Classification

OBSERVATIONAL

Study Start Date

2020-09-01

Study Completion Date

2021-04-15

Brief Summary

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The new severe acute respiratory syndrome coronavirus 2019 (SARS-CoV-2) causes the illness named COVID-19, which is primarily characterized by pneumonia. As of 27 December, there have been over 79.2 million cases and over 1.7 million deaths reported since the start of the pandemic. In many cases, pneumonia evolves to acute respiratory distress syndrome (ARDS) with the need for mechanical ventilation and patient admission to intensive care unit, determining a marked increase in the need for intensive care beds worldwide.

Pulmonary involvement causes predominantly hypoxemic respiratory failure. Although COVID-19 pneumonia often falls within the diagnostic criteria of ARDS, it differs from it for some peculiar pathophysiological characteristics. In particular, patients with ARDS secondary to COVID-19 often have the compliance of the respiratory system within the normal range. A significant role in the pathophysiology of hypoxemia seems to depend on vascular alterations such as altered pulmonary vascular self-regulation, pulmonary capillary leakage, and microvascular thrombosis in a complex process known as "immunothrombosis". All together they act by altering the relationship between ventilation and perfusion and increasing the dead space, which ultimately results in impaired efficiency of the pulmonary ventilation. Among the various markers associated with the prognosis of patients with COVID-19, D-dimer is linked to both the inflammatory state and thrombotic phenomena and could help to identify patients at greater risk of developing early ventilation-perfusion changes.

This study aims at measuring the ventilatory efficiency, assessed by Ventilatory Ratio, in critically ill, mechanically ventilated, COVID-19 patients and its correlation with plasma D-dimer and quasi-static respiratory compliance.

Detailed Description

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Conditions

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ARDS

Study Design

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Observational Model Type

COHORT

Study Time Perspective

PROSPECTIVE

Study Groups

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ARDS COVID-19

Patients who meet Berlin's ARDS diagnostic criteria, with confirmed SARS-CoV-2 infection, requiring invasive mechanical ventilation.

data collecting

Intervention Type OTHER

Within 24h from ICU admission, the ventilatory efficiency will be assessed by the following Ventilatory Ratio equation:

Ventilatory Ratio = \[minute ventilation (ml/min) × PaCO2 (mm Hg)\]/(predicted body weight × 100 × 37.5).

Where PaCO2 is the partial pressure of carbon dioxide in mmHg in the arterial blood.

Tha quasi-static compliance will be calculated according to the equation:

C=Tidal Volume/(Paw plateau - PEEP total)

where Paw plateau is the airway pressure measured during 4 seconds of inspiratory pause, PEEP total is the airway pressure measured during 4 seconds of expiratory pause.

In the same time frame, complete blood count, d-dimer, sequential organ failure assessment score, blood gas analysis, haemodynamic and ventilatory parameters will be collected.

Interventions

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data collecting

Within 24h from ICU admission, the ventilatory efficiency will be assessed by the following Ventilatory Ratio equation:

Ventilatory Ratio = \[minute ventilation (ml/min) × PaCO2 (mm Hg)\]/(predicted body weight × 100 × 37.5).

Where PaCO2 is the partial pressure of carbon dioxide in mmHg in the arterial blood.

Tha quasi-static compliance will be calculated according to the equation:

C=Tidal Volume/(Paw plateau - PEEP total)

where Paw plateau is the airway pressure measured during 4 seconds of inspiratory pause, PEEP total is the airway pressure measured during 4 seconds of expiratory pause.

In the same time frame, complete blood count, d-dimer, sequential organ failure assessment score, blood gas analysis, haemodynamic and ventilatory parameters will be collected.

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

All of the following:

* confirmed SARS-CoV-2 infection by RT-PCR on a nasopharyngeal swab
* severe hypoxemia due to COVID-19 who meets the diagnostic criteria of ARDS (Berlin's definition)
* invasive mechanical ventilation
* patients receiving neuromuscular blocking drugs

Exclusion Criteria

* history of preexisting severe hypoxemia (i.e. primary pulmonary hypertension, COPD in therapy with O2 supplementation, pulmonary fibrosis, etc.)
* severe haemodynamic instability defined as:

* Mean arterial pressure \< 65 mmHg despite the infusion of norepinephrine, or epinephrine, or dobutamine, or levosimendan
* severe left ventricular dysfunction with ejection fraction \<20%
* right ventricular failure due to pulmonary embolism
Minimum Eligible Age

18 Years

Maximum Eligible Age

90 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Ospedale Infermi Rimini

UNKNOWN

Sponsor Role collaborator

S. Anna Hospital

OTHER

Sponsor Role collaborator

Azienda Ospedaliero, Universitaria Ospedali Riuniti

OTHER

Sponsor Role collaborator

ASST Fatebenefratelli Sacco

OTHER

Sponsor Role lead

Responsible Party

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Riccardo Colombo

MD

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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Riccardo Colombo, M.D.

Role: PRINCIPAL_INVESTIGATOR

ASST Fatebenefratelli Sacco - Ospedale Luigi Sacco

Locations

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Arcispedale Sant'Anna

Ferrara, Emilia-Romagna, Italy

Site Status NOT_YET_RECRUITING

Ospedale Infermi

Rimini, Emilia-Romagna, Italy

Site Status NOT_YET_RECRUITING

ASST Fatebenefratelli Sacco

Milan, Lombardy, Italy

Site Status RECRUITING

Azienda Ospedaliero Universitaria Ospedali Riuniti

Ancona, The Marches, Italy

Site Status RECRUITING

Countries

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Italy

Central Contacts

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Riccardo Colombo, M.D.

Role: CONTACT

+390239043023

Andrea Agarossi, M.D.

Role: CONTACT

Facility Contacts

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Alberto Fogagnolo, M.D.

Role: primary

Jonathan Montomoli, M.D.

Role: primary

Riccardo Colombo, M.D.

Role: primary

0239043023

Andrea Carsetti, M.D.

Role: primary

References

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WHO Weekly epidemiological update - 29 December 2020 - https://www.who.int/publications/m/item/weekly-epidemiological-update---29-december-2020

Reference Type BACKGROUND

Sinha P, Calfee CS, Beitler JR, Soni N, Ho K, Matthay MA, Kallet RH. Physiologic Analysis and Clinical Performance of the Ventilatory Ratio in Acute Respiratory Distress Syndrome. Am J Respir Crit Care Med. 2019 Feb 1;199(3):333-341. doi: 10.1164/rccm.201804-0692OC.

Reference Type BACKGROUND
PMID: 30211618 (View on PubMed)

ARDS Definition Task Force; Ranieri VM, Rubenfeld GD, Thompson BT, Ferguson ND, Caldwell E, Fan E, Camporota L, Slutsky AS. Acute respiratory distress syndrome: the Berlin Definition. JAMA. 2012 Jun 20;307(23):2526-33. doi: 10.1001/jama.2012.5669.

Reference Type BACKGROUND
PMID: 22797452 (View on PubMed)

Other Identifiers

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VentRatio-19

Identifier Type: -

Identifier Source: org_study_id

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