Myeloid Cells in Patients With Covid-19 Pneumonia

NCT ID: NCT04590261

Last Updated: 2022-10-25

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

NOT_YET_RECRUITING

Clinical Phase

NA

Total Enrollment

120 participants

Study Classification

INTERVENTIONAL

Study Start Date

2022-12-31

Study Completion Date

2025-12-31

Brief Summary

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The purpose of this study is to analyze in depth the relationship of myeloid cell subpopulations during infection by Severe acute respiratory syndrome coronavirus 2 (SARS-Cov2), the virus mediating Covid-19. Myeloid cells include neutrophils, monocytes and dendritic cells, each divided into subpopulations with different functions in immune defense and immune pathologies.

The study is based on the following hypotheses:

* Infection and the interferon response to infection may induce hyperactive or immunosuppressive differentiation of myeloid cells, that may be treated by specific inhibitors.
* Some myeloid cell subpopulations currently identified in our laboratories might be markers for Covid-19 prognosis.
* Alternative receptors may be present on myeloid cells, inducing the cytokine storm, a target for therapy.
* The expression of Interferon (IFN) receptor and IFN responding genes on myeloid cells and on respiratory epithelial cells may correlate with prognosis and indicate potential treatment targets.
* Interferon responses are known to be skewed during Covid-19, but some IFN subtype polymorphisms may correlate with prognosis and these subtypes migt be supplemented or inhibited for therapy.

Detailed Description

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Infection by SARS-Cov2 drives to pneumonia in most cases, 30 percent of which require hospitalization in a pneumology ward, among which 30 percent with severe acute respiratory syndrome (SARS) must go to critical care units, with a high mortality rate.

This infection drives a strong cytokine response. In patients developing SARS, a profound, paradoxical defect in IFN alpha and in the expression of genes responding to IFN alpha was discovered. IFNs are strong anti-viral proteins, used for the treatment of viral hepatitis. Type I IFNs, including IFN alpha, have ubiquitous receptors on almost every cell type. Type III IFNs, or IFN lambda, have a more restricted receptor expression, including on neutrophils. Their polymorphisms were already related to the prognosis of another ribonucleic acid (RNA) virus with mucosal entry, hepatitis C virus (HCV), especially in people with African origins.

Coronaviruses responsible for the previous SARS-Cov or Middle East respiratory syndrome coronavirus (MERS-Cov) epidemics induce a defective IFN signal transduction. Many other viral infection lead to desensitization. Moreover, IFN alpha by itself can lead to defective antiviral responses. At the immune cell level, lymphopenia with an increased neutrophil/lymphocyte ratio were noted in severe SARS-Cov2 case. New subpopulations of neutrophils have been characterized by phenotypic and proteomic studies, with inflammatory or suppressive functions.

It will be important to know if

* hyperactive or immunosuppressive myeloid cell differentiation is caused by SARS-Cov2 and can be inhibited specifically.
* some myeloid subpopulations
* correlate with the prognosis of the disease,
* myeloid cells have alternative receptors for SARS-Cov2,
* some IFN polymorphisms may correlate with prognosis and might be supplemented or inhibited for therapy.

The answers will be obtained through the primary and secondary outcome measures, as described below.

Conditions

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Covid-19; SARS-Cov2

Study Design

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Allocation Method

NON_RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

SCREENING

Blinding Strategy

NONE

Study Groups

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group 1

Former mild SARS-Cov2 Pneumonia, 2 to 12 moths before, ≤ 5 L/mn Oxygen treatment

Group Type OTHER

Blood sampling

Intervention Type OTHER

Peripheral Blood sampling, 25 mL

Nasal Brushing

Intervention Type OTHER

Nasal Brushing, facultative

Group 2

Former severe SARS-Cov2 Pneumonia, 2 to 12 moths before, \> 5 L/mn Oxygen treatment

Group Type OTHER

Blood sampling

Intervention Type OTHER

Peripheral Blood sampling, 25 mL

Nasal Brushing

Intervention Type OTHER

Nasal Brushing, facultative

Group 3

Physician examination in the Pneumology ward, Cochin Hospital

Group Type OTHER

Blood sampling

Intervention Type OTHER

Peripheral Blood sampling, 25 mL

Nasal Brushing

Intervention Type OTHER

Nasal Brushing, facultative

Group 4

Current hospitalization for Sars-Cov2 Pneumonia at Cochin Hospital

Group Type OTHER

Blood sampling

Intervention Type OTHER

Peripheral Blood sampling, 25 mL

Nasal Brushing

Intervention Type OTHER

Nasal Brushing, facultative

Interventions

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Blood sampling

Peripheral Blood sampling, 25 mL

Intervention Type OTHER

Nasal Brushing

Nasal Brushing, facultative

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* Age \> 18 years
* Sex : male or female
* French Social Security insurance
* Information and consent dated and signed \*
* Group 1 : inclusion 2 to 12 months after hospitalization for Covid-19 pneumonia with mild severity (oxygen treatment ≤5L/mn);
* Group 2 : 2 to 12 months after hospitalization for Covid-19 pneumonia with high severity (oxygen treatment \>5L/mn);
* Group 3 : external visit at Cochin Hospital, age- and sex -matched with Groups 1, 2, 4.
* Group 4 : inclusion during hospitalization for Covid-19, within the first month of symptoms.

Exclusion Criteria

* Tuberculosis or other evolutive bacterial infection
* Chronic evolutive viral Infections (Hepatitis B or C, HIV)
* Ongoing chemotherapy or radiotherapy
* Participation in another research protocol with current exclusion period at the time of pre-inclusion (possible inclusion in an observational study
* Vulnerable person (pregnant, parturient woman, breastfeeding woman, person Under tutorship, person under arrest through judiciary or administrative decision )
Minimum Eligible Age

18 Years

Maximum Eligible Age

70 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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Institut National de la Santé Et de la Recherche Médicale, France

OTHER_GOV

Sponsor Role collaborator

Assistance Publique - Hôpitaux de Paris

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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Pierre-Régis Burgel, MD, PhD

Role: PRINCIPAL_INVESTIGATOR

Cochin Hospital

Central Contacts

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Pierre-Régis Burgel, MD, PhD

Role: CONTACT

01 58 41 23 49 ext. +33

Marie BENHAMMANI-Godard

Role: CONTACT

01 58 41 12 11 ext. +33

Related Links

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Other Identifiers

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2020-A02700-39

Identifier Type: OTHER

Identifier Source: secondary_id

APHP201138

Identifier Type: -

Identifier Source: org_study_id

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