Establishment of a Prospective Clinical-biological Database

NCT ID: NCT04458792

Last Updated: 2025-02-12

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

RECRUITING

Clinical Phase

NA

Total Enrollment

450 participants

Study Classification

INTERVENTIONAL

Study Start Date

2016-11-30

Study Completion Date

2032-11-30

Brief Summary

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A Clinical and Biological Database will provide to the scientific community a collection of blood and tissues with clinical data to improve knowledge about cancer and help to develope new cancer treatments. This database is specific to Sarcoma

Detailed Description

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Liposarcomas (LPS) are soft tissue tumours of mesenchymal origin. The most common histological subtypes, represented by well differenciated Lipsarcomas (WD-LPS) and dedifferenciated (DD-LPS), are characterized by the quasi-systematic amplification of the q13-15 of chromosome 12 containing the Mdm2 gene.

The systematic amplification of the Mdm2 gene is such that it is used clinically to distinguish WD/DD-LPS from other types of sarcomas. These observations raise key questions about the high selection pressure that leads to the almost systematic amplification of Mdm2 during the development of these LPS.

Mdm2 is an oncoprotein whose roles in p53 tumour suppressor degradation are broadly described. However, the different inhibitors targeting this interaction were disappointing in clinical trials.

Recently a team from the U1194 INSERM unit of the IRCM which collaborates in this project showed by a pan-genomics analysis, that Mdm2 is recruited to chromatin within a multiproteic complex having as target a transcriptional program involved in the metabolism and in particular in the biosynthesis of the serin.

This clinical-biological basis will allow the continuation of this research project on liposarcomas and the development of new research projects on other histological types of sarcomas.

Conditions

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Sarcoma

Study Design

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Allocation Method

NA

Intervention Model

SINGLE_GROUP

Primary Study Purpose

OTHER

Blinding Strategy

NONE

Study Groups

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Experimental: Biological collection

For all the patients include in the study :

* Paraffin tissue samples collected during surgery (neoplasic tissue and normal tissue)
* MDM2 project : Blood samples collected at different times : Before the surgery, and 1 month after the surgery
* circulant DNA project : Blood samples collected at different times : Before the surgery, and 1 month after the surgery
* radiotherapy toxicity : Blood samples collected before the radiotherapy

In parallel to this biological collection, standardized clinical data will be entered into a database

Group Type EXPERIMENTAL

Paraffin tissue samples

Intervention Type OTHER

Paraffin tissue samples collected during surgery (neoplasic tissue and normal tissue

blood sample

Intervention Type OTHER

MDM2 project : Blood samples collected at different times : Before the surgery, and 1 month after the surgery circulant DNA project : Blood samples collected at different times : Before the surgery, and 1 month after the surgery radiotherapy toxicity : Blood samples collected before the radiotherapy

Interventions

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Paraffin tissue samples

Paraffin tissue samples collected during surgery (neoplasic tissue and normal tissue

Intervention Type OTHER

blood sample

MDM2 project : Blood samples collected at different times : Before the surgery, and 1 month after the surgery circulant DNA project : Blood samples collected at different times : Before the surgery, and 1 month after the surgery radiotherapy toxicity : Blood samples collected before the radiotherapy

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* men or women \> 18 years
* Patient treated by surgery for : a primitive sarcoma and/or local relapse sarcoma and/or metastatic localisation of sarcoma
* patient accepted blood sample
* patient treated by radiotherapy before or after the surgery
* Patient signed informed consent

Exclusion Criteria

* Patient not affiliated to french Social Protection system
* Patient under guardianship
* Patient unable to understand or comply with study instructions or requirements for psychological, family, social or geographical reasons
* Pregnancy and/or feeding
* Patient take care in an Emergency Situation
Minimum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Institut du Cancer de Montpellier - Val d'Aurelle

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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FIRMIN Nelly, MD

Role: STUDY_CHAIR

Institut régional du Cancer Montpellier

Locations

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Icm Val D'Aurelle

Montpellier, Herault, France

Site Status RECRUITING

Countries

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France

Central Contacts

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Aurore MOUSSION

Role: CONTACT

4 67 61 31 02 ext. +33

References

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Ducimetiere F, Lurkin A, Ranchere-Vince D, Decouvelaere AV, Peoc'h M, Istier L, Chalabreysse P, Muller C, Alberti L, Bringuier PP, Scoazec JY, Schott AM, Bergeron C, Cellier D, Blay JY, Ray-Coquard I. Incidence of sarcoma histotypes and molecular subtypes in a prospective epidemiological study with central pathology review and molecular testing. PLoS One. 2011;6(8):e20294. doi: 10.1371/journal.pone.0020294. Epub 2011 Aug 3.

Reference Type RESULT
PMID: 21826194 (View on PubMed)

Mastrangelo G, Coindre JM, Ducimetiere F, Dei Tos AP, Fadda E, Blay JY, Buja A, Fedeli U, Cegolon L, Frasson A, Ranchere-Vince D, Montesco C, Ray-Coquard I, Rossi CR. Incidence of soft tissue sarcoma and beyond: a population-based prospective study in 3 European regions. Cancer. 2012 Nov 1;118(21):5339-48. doi: 10.1002/cncr.27555. Epub 2012 Apr 19.

Reference Type RESULT
PMID: 22517534 (View on PubMed)

Crago AM, Singer S. Clinical and molecular approaches to well differentiated and dedifferentiated liposarcoma. Curr Opin Oncol. 2011 Jul;23(4):373-8. doi: 10.1097/CCO.0b013e32834796e6.

Reference Type RESULT
PMID: 21552124 (View on PubMed)

Lokka S, Scheel AH, Dango S, Schmitz K, Hesterberg R, Ruschoff J, Schildhaus HU. Challenging dedifferentiated liposarcoma identified by MDM2-amplification, a report of two cases. BMC Clin Pathol. 2014 Jul 28;14:36. doi: 10.1186/1472-6890-14-36. eCollection 2014.

Reference Type RESULT
PMID: 25126005 (View on PubMed)

Marine JC, Lozano G. Mdm2-mediated ubiquitylation: p53 and beyond. Cell Death Differ. 2010 Jan;17(1):93-102. doi: 10.1038/cdd.2009.68.

Reference Type RESULT
PMID: 19498444 (View on PubMed)

Ray-Coquard I, Blay JY, Italiano A, Le Cesne A, Penel N, Zhi J, Heil F, Rueger R, Graves B, Ding M, Geho D, Middleton SA, Vassilev LT, Nichols GL, Bui BN. Effect of the MDM2 antagonist RG7112 on the P53 pathway in patients with MDM2-amplified, well-differentiated or dedifferentiated liposarcoma: an exploratory proof-of-mechanism study. Lancet Oncol. 2012 Nov;13(11):1133-40. doi: 10.1016/S1470-2045(12)70474-6. Epub 2012 Oct 17.

Reference Type RESULT
PMID: 23084521 (View on PubMed)

Other Identifiers

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ICM-BDD 2016/03

Identifier Type: -

Identifier Source: org_study_id

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