Corticosteroid-induced Lipodystrophy and Adipokines

NCT ID: NCT00822042

Last Updated: 2014-02-27

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Total Enrollment

32 participants

Study Classification

OBSERVATIONAL

Study Start Date

2006-08-31

Study Completion Date

2008-11-30

Brief Summary

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Hypothesis: systemic therapy with corticosteroid induces morphological changes (e.g., moon face, buffalo neck) called lipodystrophy (LD). We hypothesize that this LD is associated with variation of adipocytokines (e.g., adiponectin, leptine, IL6) levels

Primary objective: To show a 50% decrease in adipocytes adiponectin's expression in patients who developed LD versus those who did not developed LD during the first 3 months of a systemic therapy with corticosteroids

Secondary objectives: To look for differences in the mRNA expression of 11bHSD1, SREBP1c and PPARg in fat samples of patients before and after treatment with systemic corticosteroids and between LD+ and LD-patients To compare the fat morphology before and after treatment with glucosteroids

Detailed Description

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Design: Monocentric, cross-sectional analytical study

Subjects: 32 HIV-free and Cushing disease-free adult patients for whom a prolonged treatment (³3months) with glucosteroids (³ 0.5 mg/kg/day) is initiated

Methods: At treatment initiation and 3 months after: comparison of fat sample mRNA expression of adipokines (adiponectin, leptin, IL6, TNFa), 11bHSD1, SREBP1c and PPARg, fat morphology and seric concentrations of adiponectin, leptin, IL6, sTNFR1 between patients LD+ and patients LD-. The diagnosis of LD will be performed by 3 experts using patients photographs

Aims of this study:

* To gain a better understanding of the pathophysiology of glucosteroids-induced LD
* To compare this pathophysiology to the one of HIV-associated LD for which the hypothesis of a local, cellular, hypercorticism has been put forward and for which related treatment have been prescribed.

Conditions

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Lipodystrophy

Study Design

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Observational Model Type

COHORT

Study Time Perspective

PROSPECTIVE

Study Groups

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1

Patients with corticotherapy lasting more than 3 months

Samples and procedures

Intervention Type OTHER

* Scanner, histomorphometry : at the inclusion and M3 visits
* whole blood samples, cytoponction : at the inclusion and M3 visits

Interventions

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Samples and procedures

* Scanner, histomorphometry : at the inclusion and M3 visits
* whole blood samples, cytoponction : at the inclusion and M3 visits

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* adult patient
* starting therapy with prednisone
* corticosteroid therapy lasting more than 3 months
* baseline prednisone dosage \>= 0.5 mg/kg/d

Exclusion Criteria

* Cushing disease
* HIV +Pregnancy
* Recent weight lost (\> 5% of the usual weight)
* Therapy with glucocorticosteroids during the past 6 months
Minimum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Assistance Publique - Hôpitaux de Paris

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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Laurence FARDET, MD PhD

Role: PRINCIPAL_INVESTIGATOR

Assistance Publique - Hôpitaux de Paris

Locations

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Hopital Saint-Antoine

Paris, , France

Site Status

Countries

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France

Other Identifiers

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CIRC 05147

Identifier Type: -

Identifier Source: secondary_id

P 051037

Identifier Type: -

Identifier Source: org_study_id

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