Potential Role of Inflammasome NLRP3 And IL-1B Gene Expression in COVID-19 Patients: Impact of Ferretin and D -Dimer.

NCT ID: NCT06080750

Last Updated: 2023-10-12

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Total Enrollment

75 participants

Study Classification

OBSERVATIONAL

Study Start Date

2023-01-01

Study Completion Date

2025-01-01

Brief Summary

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2.1 Study the role of NLRP3 inflammasome in COVID-19 patients. 2.2 Study the gene expression of NLRP3 and IL-1β in blood samples of COVID-19 patients and compare to apparently healthy subjects.

2.3 Correlation between NLRP3, IL-1β, IL-6 and severity of the disease. 2.4 Impact of ferritin and D-dimer on inflammasome componnets NLRP3, IL-1β IL-6 .

Detailed Description

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Coronavirus Disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a viral infection that results in respiratory disease, which can evolve into multiorgan failure (MOF), leading to death (Aida A Abdelmaksoud et al., 2021).

The first cases of COVID-19 were detected in Wuhan, China, in 2019. Since then, the illness has spread rapidly around the country and the world reaching a pandemic level (Rocklöv J et al., 2020). Cases have been reported in more than 180 countries to the World Health Organization (WHO), including more than two million deaths (WHO, 2021).

Several biochemical alterations have been described in COVID-19 patients as lymphopenia, thrombocytopenia and increased levels of C-reactive protein. The hallmark of severe COVID-19 is the cytokine storm accompanied by a hyperinflammatory response in the host due to the release of large amounts of pro-inflammatory cytokines IL-1β, IL-6, IL-2, IL-7, TNF-α, interferon-γ (IFN)-γ, CRP, procalcitonin (PCT), lactate dehydrogenase (LDH), erythrocyte sedimentation rate (ESR) and ferritin (Marcello Ciaccio and Agnello, 2020; Shah A., 2020).

The inflammasome is a multiprotein complex, known for its role in the innate immune response against viral diseases and a regulator of processing of the pro-inflammatory cytokines interleukin (IL)-1β and IL-18. Inflammasome contains a NOD-like receptor (NLR) sensor protein and pyrin domain-containing 3 , after which, NLRP3 Inflammasome was named (de Rivero Vaccari et al., 2020). The presence of inflammasome-derived products such as IL-1β, IL-18, and LDH in patients' sera suggests inflammasome engagement (Chen G et al., 2020).

Increased IL-6, IL-10, and IL-4 were found in patients when compared with controls, indicating that SARS-CoV-2 infects human monocytes and triggers NLRP3 inflammasome activation. However, the definitive demonstration of NRLP3 inflammasome participation is still required because these products can be produced via inflammasome-independent pathways (Rodrigues et al., 2021). But, the possible contribution of NLRP3 inflammasome activation and their ability to induce IL-1β production is still not clear .

The pronounced inflammatory characteristics of COVID-19 prompted us to investigate the expression of the NRLP3 inflammasome and its relation to IL-1B in disease development and severity and to determine the best anti-inflammatory will be used in COVID-19.

Conditions

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Inflammasome NLRP3 And IL-1B Gene Expression in COVID-19 Patients

Study Design

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Observational Model Type

CASE_CONTROL

Study Time Perspective

CROSS_SECTIONAL

Study Groups

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healthy

as a control group

Quantitative Real Time Polymerase chain reaction (qRT-PCR)

Intervention Type GENETIC

to study gene expression of NLRP3 inflammasome and IL-1B in blood

ELISA and colorimeter

Intervention Type DIAGNOSTIC_TEST

IL-6 and total LDH

mild to moderate covid-19

mild-moderate symptoms

Quantitative Real Time Polymerase chain reaction (qRT-PCR)

Intervention Type GENETIC

to study gene expression of NLRP3 inflammasome and IL-1B in blood

ELISA and colorimeter

Intervention Type DIAGNOSTIC_TEST

IL-6 and total LDH

sever covid-19.

severe symptoms

Quantitative Real Time Polymerase chain reaction (qRT-PCR)

Intervention Type GENETIC

to study gene expression of NLRP3 inflammasome and IL-1B in blood

ELISA and colorimeter

Intervention Type DIAGNOSTIC_TEST

IL-6 and total LDH

Interventions

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Quantitative Real Time Polymerase chain reaction (qRT-PCR)

to study gene expression of NLRP3 inflammasome and IL-1B in blood

Intervention Type GENETIC

ELISA and colorimeter

IL-6 and total LDH

Intervention Type DIAGNOSTIC_TEST

Other Intervention Names

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qRT-PCR

Eligibility Criteria

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Inclusion Criteria

* Adult Patient aged 18years and over presented to Assiut University Hospital
* Suspected of covid19 because of the presence of symptoms suggestive of pneumonia (fever\>38) and at least one of the following symptoms ;cough, dyspnea, tachpnea or hypoxia
* having RT-PCR (confirmed positive PCR)
* Chest CT within 5 days of initial PCR

Exclusion Criteria

* Cases less than 18 years old
* Cancer patients (affect result of study as they have disrubted level of these biomarker)
Minimum Eligible Age

18 Years

Maximum Eligible Age

90 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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Assiut University

OTHER

Sponsor Role lead

Responsible Party

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Tasneem Abdel-Baset Ahmed Aly

Pharmacist

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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Ayat A Sayed, M.D

Role: STUDY_DIRECTOR

associate professor

Mona A AlBaz, M.D

Role: STUDY_CHAIR

Professor

Locations

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Biochemistry department,Faculty of medicine

Asyut, , Egypt

Site Status RECRUITING

Countries

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Egypt

Central Contacts

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Tasneem A Alsanory, Master

Role: CONTACT

+20106852153

Marwa A Gaber, M.D

Role: CONTACT

+201001883544

References

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Abdelmaksoud AA, Ghweil AA, Hassan MH, Rashad A, Khodeary A, Aref ZF, Sayed MAA, Elsamman MK, Bazeed SES. Olfactory Disturbances as Presenting Manifestation Among Egyptian Patients with COVID-19: Possible Role of Zinc. Biol Trace Elem Res. 2021 Nov;199(11):4101-4108. doi: 10.1007/s12011-020-02546-5. Epub 2021 Jan 7.

Reference Type BACKGROUND
PMID: 33409924 (View on PubMed)

Chen G, Wu D, Guo W, Cao Y, Huang D, Wang H, Wang T, Zhang X, Chen H, Yu H, Zhang X, Zhang M, Wu S, Song J, Chen T, Han M, Li S, Luo X, Zhao J, Ning Q. Clinical and immunological features of severe and moderate coronavirus disease 2019. J Clin Invest. 2020 May 1;130(5):2620-2629. doi: 10.1172/JCI137244.

Reference Type BACKGROUND
PMID: 32217835 (View on PubMed)

de Rivero Vaccari JC, Dietrich WD, Keane RW, de Rivero Vaccari JP. The Inflammasome in Times of COVID-19. Front Immunol. 2020 Oct 8;11:583373. doi: 10.3389/fimmu.2020.583373. eCollection 2020.

Reference Type BACKGROUND
PMID: 33149733 (View on PubMed)

Ciaccio M, Agnello L. Biochemical biomarkers alterations in Coronavirus Disease 2019 (COVID-19). Diagnosis (Berl). 2020 Nov 18;7(4):365-372. doi: 10.1515/dx-2020-0057.

Reference Type BACKGROUND
PMID: 32589600 (View on PubMed)

Rocklov J, Sjodin H, Wilder-Smith A. COVID-19 outbreak on the Diamond Princess cruise ship: estimating the epidemic potential and effectiveness of public health countermeasures. J Travel Med. 2020 May 18;27(3):taaa030. doi: 10.1093/jtm/taaa030.

Reference Type BACKGROUND
PMID: 32109273 (View on PubMed)

Rodrigues TS, de Sa KSG, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Goncalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, de Lima MHF, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, de Barros FC, do Amaral NB, Giannini MC, Bonjorno LP, Lopes MIF, Santana RC, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, de Almeida SCL, de Oliveira FR, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves-Filho JC, Cunha FQ, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, de Oliveira RDR, Louzada-Junior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. J Exp Med. 2021 Mar 1;218(3):e20201707. doi: 10.1084/jem.20201707.

Reference Type BACKGROUND
PMID: 33231615 (View on PubMed)

Shah A. Novel Coronavirus-Induced NLRP3 Inflammasome Activation: A Potential Drug Target in the Treatment of COVID-19. Front Immunol. 2020 May 19;11:1021. doi: 10.3389/fimmu.2020.01021. eCollection 2020. No abstract available.

Reference Type BACKGROUND
PMID: 32574259 (View on PubMed)

Other Identifiers

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NLRP3 & IL-1B in covid19

Identifier Type: -

Identifier Source: org_study_id

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