Aberrant Expression of CD56 in Patients With Hematologic Malignancies.

NCT ID: NCT04546945

Last Updated: 2022-07-22

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Total Enrollment

38 participants

Study Classification

OBSERVATIONAL

Study Start Date

2020-07-20

Study Completion Date

2024-12-30

Brief Summary

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CD56(cluster of differentiation 56) was found to be ectopically expressed in multiple myeloma . A met analysis indicated that CD56 over expression may be an adverse prognostic factor in AML. To the best of our knowledge, no available data the expression pattern of CD56 in other Hematologic malignancies. This work is designed to evaluate the expression pattern of CD56 in hematologic malignancies.

Detailed Description

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CD56 is the archetypal phenotypic marker of natural killer cells but can actually be expressed by many more immune cells, including alpha, beta T cells, gamma, delta T cells, dendritic cells, and monocytes. Common to all these CD56 expressing cell types have strong immunostimulatory effector functions, including T helper 1 cytokine production and an efficient cytotoxic capacity. Interestingly, both numerical , functional deficiencies and phenotypic alterations of the CD56 immune cell fraction have been reported in patients with various infectious, autoimmune, or malignant diseases.CD56 is also known as neural cell adhesion molecule (NCAM).

Hematologic malignancies are a heterogeneous group of diseases of diverse incidence, prognosis and etiology that arise from malignant transformation of cells from the bone marrow or the lymphatic system.There are two major groups of Hematologic malignancies according to their cell lineage: Myeloid and lymphoid. Lymphoid neoplasms are a varied group that includes non-Hodgkin lymphoma (NHL), Hodgkin lymphoma (HL), leukemia and multiple myeloma (MM).myeloid neoplasms and acute leukaemia include: Myeloproliferative neoplasms (MPN), Myelodysplastic/myeloproliferative neoplasms (MDS/MPN), Myelodysplastic syndromes (MDS), Acute myeloid leukaemia (AML), Acute leukemias of ambiguous lineage, B-lymphoblastic leukaemia/lymphoma, T-lymphoblastic leukaemia/lymphoma.The exact causes of Hematologic malignancies are still unknown although multiple epidemiological studies have reported an association between the development of Hematologic malignancies and several risk factors. Some factors are well documented to increase the risk of some types of leukaemia such as benzene exposure and ionizing radiation.However, many other factors were observed to have an association with Hematologic malignancies such as age, gender, tobacco smoking , obesity , hepatitis C virus (HCV) infection , family history , and environmental exposure to pesticides but with no clear evidence.

CD56 was found to be ectopically expressed in multiple myeloma. A met analysis indicated that CD56 over expression may be an adverse prognostic factor in AML. To the best of our knowledge, no available data the expression pattern of CD56 in other Hematologic malignancies. This work is designed to evaluate the expression pattern of CD56 in hematologic malignancies.

Conditions

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Hematologic Malignancies

Study Design

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Observational Model Type

CASE_CONTROL

Study Time Perspective

CROSS_SECTIONAL

Study Groups

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control cases

normal healthy person

CD56

Intervention Type DIAGNOSTIC_TEST

detection of CD56 by flow cytometry

Patients

Patients with hematologic malignancies. Newly diagnosed.

CD56

Intervention Type DIAGNOSTIC_TEST

detection of CD56 by flow cytometry

Interventions

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CD56

detection of CD56 by flow cytometry

Intervention Type DIAGNOSTIC_TEST

Eligibility Criteria

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Inclusion Criteria

* Patients with hematologic malignancies.
* Newly diagnosed

Exclusion Criteria

* Patients with hematologic disorders other than hematologic malignancies.
Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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Asmaa Hassan mohamed Abdel Mawjoud

OTHER

Sponsor Role lead

Responsible Party

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Asmaa Hassan mohamed Abdel Mawjoud

resident doctor clinical pathology

Responsibility Role SPONSOR_INVESTIGATOR

Principal Investigators

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Maged S Mahmoud, prof

Role: STUDY_DIRECTOR

Assiut University

Locations

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Assiut University

Asyut, , Egypt

Site Status COMPLETED

Assiut university

Asyut, , Egypt

Site Status RECRUITING

Countries

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Egypt

Central Contacts

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asmaa H mohamed, R.DR

Role: CONTACT

+201024850070

Dina A Mohareb, a.prof

Role: CONTACT

+201062427707

Facility Contacts

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Assiut

Role: primary

Related Links

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3601173/

Roothans D, Smits E, Lion E, Tel J, Anguille S. CD56 marks human dendritic cell subsets with cytotoxic potential. Oncoimmunology (2013)

https://pubmed.ncbi.nlm.nih.gov/27686372/

(3) Van Acker HH, Anguille S, Willemen Y, Van den Bergh JM, Berneman ZN, Lion E, et al. Interleukin-15 enhances the proliferation, stimulatory phenotype, and antitumor effector functions of human gamma delta T cells. J Hematol Oncol (2016)

https://www.frontiersin.org/articles/10.3389/fimmu.2017.00892/full

Heleen H. Van Acker,\* Anna Capsomidis, Evelien L. Smits, and Viggo F. Van Tendeloo ( CD56 in the Immune System: More Than a Marker for Cytotoxicity?) Published 2017 frontiers in immunology.

https://pubmed.ncbi.nlm.nih.gov/20664057/

(6) Sant M, Allemani C, Tereanu C, De Angelis R, Capocaccia R, Visser O, Marcos-Gragera R, Maynadie M, Simonetti A, Lutz JM, et al: Incidence of hematologic malignancies in Europe by morphologic subtype: Results of the HAEMACARE project. Blood.

https://pubmed.ncbi.nlm.nih.gov/19357394/

(8)Vardiman JW, Thiele J, Arber DA, Brunning RD, Borowitz MJ, Porwit A, Harris NL, Le Beau MM, Hellström-Lindberg E, Tefferi A and Bloomfield CD: The 2008 revision of the World Health Organization (WHO)

https://pubmed.ncbi.nlm.nih.gov/26980727/

(9) Swerdlow SH, Campo E, Pileri SA, Harris NL, Stein H, Siebert R, Advani R, Ghielmini M, Salles GA, Zelenetz AD and Jaffe ES: The 2016 revision of the World Health Organization classification of lymphoid neoplasmsBlood.

https://pubmed.ncbi.nlm.nih.gov/27069254/

(10) A. Arber, Attilio Orazi,Hasserjian, J ¨urgen Thiele,J. Borowitz,Le Beau, D. Bloomfield, Cazzola, and W. Vardiman : The 2016 revision to the World Health Organization classification of myeloid neo

https://pubmed.ncbi.nlm.nih.gov/17311819/

(11) Rodriguez-Abreu D, Bordoni A, Zucca E. Epidemiology of hematological malignancies. Ann Oncol 2007.

https://pubmed.ncbi.nlm.nih.gov/8218879/

(12) Floderus B, Persson T, Stenlund C, Wennberg A, Ost A, Knave B. Occupational exposure to electromagnetic fields in relation to leukemia and brain tumors: a case-control study in Sweden. Cancer Causes Control 1993

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4635414/

(13) Kedia S, Bhatt VR, Rajan SK, Tandra PK, El Behery RA, Akhtari M. Benign and malignant hematological manifestations of chronic hepatitis C virus infection. Int J Prev Med 2014.

https://pubmed.ncbi.nlm.nih.gov/26597998/

(14) Pan Y, Wang H, Tao Q, Zhang C, Yang D, Qin H, et al. Absence of both CD56 and CD117 expression on malignant plasma cells is related with a poor prognosis in patients with newly diagnosed multiple myeloma. Leuk Res (2016)

https://pubmed.ncbi.nlm.nih.gov/25924702/

(15) Xu S, Li X, Zhang J, Chen J. Prognostic value of CD56 in patients with acute myeloid leukemia: a meta-analysis. J Cancer Res Clin Oncol (2015)

http://jtd.amegroups.com/article/view/16406/html

(16) Hazra A. Using the confidence interval confidently. J Thorac Dis 2017.

Other Identifiers

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Asmaa Hassan

Identifier Type: -

Identifier Source: org_study_id

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