Relationship Between Thoracic Aortic Structure Assessed B PET/CT Scan and Arterial Stiffness in Elderly Patients

NCT ID: NCT01963221

Last Updated: 2013-10-16

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Clinical Phase

NA

Total Enrollment

30 participants

Study Classification

INTERVENTIONAL

Study Start Date

2010-01-31

Study Completion Date

2013-09-30

Brief Summary

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The purpose of this study is to determine the relationship between thoracic aortic inflammation and arterial stiffness in elderly patients.

Vascular-aging is accompanied by a gradual remodeling affecting both cardiac and arterial walls. Arterial hypertension, an established cardiovascular risk factor, has been suggested to exert pro-inflammatory actions threw several biological mediators enhancing arterial stiffness. Both effects of aging and hypertension are associated with higher levels of arterial stiffness, but their respective role is not well established in the pathophysiology of arterial stiffening.

Few data are available neither on the real anatomic aortic impact of aging and hypertension on aortic compliance and ventricular function and its relationship to arterial stiffness assessed by carotid-femoral pulse wave velocity, nor on the reliability of cine phase contrast magnetic resonance imaging arterial stiffness measurements.

Recent studies using positron emission tomography imaging (PET) with 18 F fluorodeoxyglucose (FDG) has been advocated as a means of measuring arterial wall inflammation in various population referred for oncology staging. FDG uptake is correlated with the number of cardiovascular risk factors and even the risk of future cardiovascular events. This method, combined with X-ray computed tomography (CT), has also demonstrated that aortic calcifications quantified by CT and local signs of inflammation detected by FDG uptake contribute to arterial stiffness. A strong relationship between large vessels stiffening assessed by carotid-femoral pulse wave velocity measurement, aortic calcifications quantified by CT and inflammation evaluated by FDG uptake has been demonstrated.

Therefore, in the current study, we use FDG PET associated to CT to characterize aortic inflammation and aortic calcifications coupled to pulse wave velocity measurement and cardiac function in elderly individuals.

In fact, if vascular aging promoting a local inflammatory process is a risk factor for cardiovascular disease, then vascular changes assessed by non-invasive vascular imaging (MRI,FDG PET) could represent a potential target for treatment and prevention Thirty individuals ≥ 65 years of age were examined, 15 hypertensive subjects and 15 controls. Pulse wave velocity, a surrogate for aortic stiffness, was measured both by cine phase contrast magnetic resonance imaging and applanation tonometry. Brachial pulse pressure, carotid calculated pulse pressure and pulse pressure amplification (brachial to carotid ratio), predictors of cardiovascular mortality were also quantified. Thoracic aorta local inflammation and calcification were measured by 18 F-fluorodeoxyglucose positron emission tomography/computed tomography imaging. Moreover, biomarkers of low grade inflammation (high-sensitivity C-reactive protein, interleukin 6 were also determined).

Detailed Description

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Conditions

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Aging Inflammation

Keywords

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elderly aorta inflammation arterial stiffness PET/CT Imaging hypertension MRI

Study Design

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Allocation Method

NA

Intervention Model

SINGLE_GROUP

Primary Study Purpose

DIAGNOSTIC

Blinding Strategy

NONE

Study Groups

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fluodeoxyglucose (18f)

Group Type OTHER

fludeoxyglucose(18f)

Intervention Type RADIATION

blood sample

Intervention Type OTHER

cardiac and aortic magnetic resonance imaging

Intervention Type OTHER

carotid femoral pulse wave velocity

Intervention Type OTHER

positron emission tomography

Intervention Type RADIATION

computed tomography

Intervention Type RADIATION

Interventions

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fludeoxyglucose(18f)

Intervention Type RADIATION

blood sample

Intervention Type OTHER

cardiac and aortic magnetic resonance imaging

Intervention Type OTHER

carotid femoral pulse wave velocity

Intervention Type OTHER

positron emission tomography

Intervention Type RADIATION

computed tomography

Intervention Type RADIATION

Other Intervention Names

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GLUSCAN, solution for injection 600 MBq/ml : 564 461-8 ADVANCED ACCELERATOR APPLICATIONS (AAA) Laboratory

Eligibility Criteria

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Inclusion Criteria

* age \>=65 years old

Exclusion Criteria

* blood glucose\> 200 mg/dl before the PET/CT scanning
* inflammatory disease
* cancer
* all forms of secondary hypertension
* renal hepatic or pulmonary insufficiency
* absence of cardiac sinus rhythm
* diabetes mellitus
* contraindication to MRI
Minimum Eligible Age

65 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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French Cardiology Society

OTHER

Sponsor Role collaborator

Central Hospital, Nancy, France

OTHER

Sponsor Role lead

Responsible Party

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Laure JOLY

MCU-PH, MD, PhD

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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Laure JOLY, MD, PhD

Role: PRINCIPAL_INVESTIGATOR

Institut National de la Santé Et de la Recherche Médicale, France

Locations

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University Hospital of Nancy

Vandœuvre-lès-Nancy, , France

Site Status

Countries

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France

References

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Joly L, Mandry D, Verger A, Labat C, Watfa G, Roux V, Karcher G, Marie PY, Benetos A. Influence of Thoracic Aortic Inflammation and Calcifications on Arterial Stiffness and Cardiac Function in Older Subjects. J Nutr Health Aging. 2016 Mar;20(3):347-54. doi: 10.1007/s12603-015-0574-0.

Reference Type DERIVED
PMID: 26892585 (View on PubMed)

Other Identifiers

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CentralHNF

Identifier Type: -

Identifier Source: org_study_id