Lysosomal Acid Lipase (LAL) Deficiency Registry

NCT ID: NCT01633489

Last Updated: 2025-11-13

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

RECRUITING

Total Enrollment

300 participants

Study Classification

OBSERVATIONAL

Study Start Date

2013-05-30

Study Completion Date

2029-08-30

Brief Summary

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This is an observational, multi-center, international disease registry designed to collect longitudinal data and create a knowledge base that will be utilized to improve the care and treatment of patients with LAL Deficiency. Participation in the Registry by both physicians and patients is voluntary.

Detailed Description

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Lysosomal Acid Lipase (LAL) Deficiency is a rare autosomal recessive lysosomal storage disorder (LSD) that is caused by a marked decrease of lysosomal acid lipase (LAL), the enzyme that breaks down cholesteryl esters and triglycerides in the lysosomes.

Lysosomal Acid Lipase Deficiency presenting in infants (historically called Wolman Disease) is a medical emergency with rapid disease progression over a period of weeks that is typically fatal within the first 6 months of life. More commonly, LAL Deficiency presents in children and adults and this presentation has been historically called Cholesteryl Ester Storage Disease (CESD). In general, data on the prevalence of LAL Deficiency are limited, and the overall prevalence of the disease in the population is unclear.

For all presentations, LAL Deficiency is associated with significant morbidity and mortality. Deficient LAL enzyme activity results in the lysosomal accumulation of cholesteryl esters and triglycerides. In the liver, this accumulation leads to hepatomegaly, increased hepatic fat content, transaminase elevation signaling chronic liver injury, and progression to fibrosis, cirrhosis, and complications of end stage liver disease. In the spleen, LAL Deficiency results in splenomegaly, anemia, and thrombocytopenia. Lipid accumulation in the intestinal wall leads to malabsorption and growth failure. Dyslipidemia is common with elevated low density lipoprotein (LDL) and triglycerides and low high density lipoprotein (HDL), associated with increased liver fat content and transaminase elevations. In addition to liver disease, patients with LAL Deficiency experience increased risk for cardiovascular disease and accelerated atherosclerosis.

The LAL Deficiency Registry is a global registry, established to help improve care for patients through improved understanding of the disease and long-term effectiveness of therapeutic interventions including sebelipase alfa.

As with other registries, which are becoming increasingly valuable for collecting information in large, heterogeneous, 'real world' populations, the LAL Deficiency Registry aims to provide evidence to help support patient care and inform clinical practice. This Registry is also being conducted, in part, to fulfill post-marketing commitments and requirements agreed to by the Sponsor as a condition for sebelipase alfa approval in the EU and the USA.

Conditions

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Lysosomal Acid Lipase Deficiency Cholesterol Ester Storage Disease Wolman Disease Acid Cholesteryl Ester Hydrolase Deficiency, Type 2 Acid Lipase Deficiency LIPA Deficiency LAL-Deficiency

Study Design

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Observational Model Type

COHORT

Study Time Perspective

PROSPECTIVE

Study Groups

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LAL Deficiency patients

Patients are those with a diagnosis of LAL Deficiency (living and deceased), irrespective of treatment status or treatment choice.

No interventions assigned to this group

Eligibility Criteria

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Inclusion Criteria

Patients must have a confirmed diagnosis of LAL Deficiency. An Informed Consent and Authorization must be obtained prior to patient enrollment where required under applicable laws and regulations, or a waiver must be obtained by the Institutional Review Board/Independent Ethics Committee.

Patients cannot be currently participating in an Alexion-sponsored clinical trial. Patients who have concluded participation in an Alexion-sponsored sebelipase alfa clinical trial are eligible to enroll in this Registry, and enrollment in the Registry will not exclude a patient from enrolling in a future clinical trial.
Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Alexion Pharmaceuticals, Inc.

INDUSTRY

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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Alexion Pharmaceuticals

Role: STUDY_DIRECTOR

Sponsor GmbH

Locations

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Clinical Trial Site

Phoenix, Arizona, United States

Site Status RECRUITING

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Stanford, California, United States

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Miramar, Florida, United States

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Orlando, Florida, United States

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Atlanta, Georgia, United States

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Chicago, Illinois, United States

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Boston, Massachusetts, United States

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Detroit, Michigan, United States

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Minneapolis, Minnesota, United States

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Jackson, Mississippi, United States

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Hackensack, NJ, 7601, New Jersey, United States

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New York, New York, United States

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The Bronx, New York, United States

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Philadelphia, Pennsylvania, United States

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Philadelphia, Pennsylvania, United States

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Philadelphia, Pennsylvania, United States

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Houston, Texas, United States

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Houston, Texas, United States

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Snyder, Texas, United States

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Tacoma, Washington, United States

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New Lambton Heights, New South Wales, Australia

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Ghent, East Flanders, Belgium

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Campinas, São Paulo, Brazil

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São Paulo, , Brazil

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São Paulo, , Brazil

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Sofia, Sofia-Grad, Bulgaria

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Edmonton, Alberta, Canada

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Halifax, Nova Scotia, Canada

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London, Ontario, Canada

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Québec, , Canada

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Zagreb, , Croatia

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Prague, Central Bohemia, Czechia

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Olomouc, , Czechia

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Bron, Auvergne-Rhône-Alpes, France

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Clermont-Ferrand, Auvergne-Rhône-Alpes, France

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Clermont-Ferrand, Auvergne-Rhône-Alpes, France

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Grenoble, Auvergne-Rhône-Alpes, France

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Caen, Normandy, France

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La Rochelle, Nouvelle-Aquitaine, France

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Niort, Nouvelle-Aquitaine, France

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Nancy, Vandœuvre-lès-Nancy, France

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Dijon, , France

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Nantes, , France

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Paris, Île-de-France Region, France

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Munich, Bavaria, Germany

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Essen, North Rhine-Westphalia, Germany

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Mainz, Rhineland-Palatinate, Germany

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Berlin, , Germany

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Athens, Attica, Greece

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Ioannina, Epirus, Greece

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Thessaloniki, Macedonia, Greece

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Dublin, Leinster, Ireland

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Petah Tikva, Central District, Israel

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Haifa, , Israel

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Jerusalem, , Israel

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Jerusalem, , Israel

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Bari, Apulia, Italy

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Napoli, Campania, Italy

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Udine, Friuli Venezia Giulia, Italy

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Genoa, Liguria, Italy

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Bergamo, Lombardy, Italy

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Milan, Lombardy, Italy

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Turin, Piedmont, Italy

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Florence, Tuscany, Italy

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Padua, Veneto, Italy

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Zapopan, Jalisco, Mexico

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Aguascalientes, , Mexico

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Mexico City, , Mexico

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Amsterdam, North Holland, Netherlands

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Amsterdam, North Holland, Netherlands

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Guimarães, Braga District, Portugal

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Lisbon, , Portugal

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Petersburg, Leningradskaya Oblast', Russia

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Moscow, Moscow Oblast, Russia

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Moscow, Moscow Oblast, Russia

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Moscow, Moscow Oblast, Russia

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Moscow, Moscow Oblast, Russia

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Nizhny Novgorod, Nizhny Novgorod Oblast, Russia

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Riyadh, , Saudi Arabia

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Riyadh, , Saudi Arabia

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Ljubljana, , Slovenia

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Santiago de Compostela, A Coruña, Spain

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Barakaldo, Biscay, Spain

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Logroño, La Rioja, Spain

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Burela de Cabo, Lugo, Spain

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Málaga, Málaga, Spain

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Oviedo, Principality of Asturias, Spain

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Reus, Tarragona, Spain

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A Coruña, , Spain

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Albacete, , Spain

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Alicante, , Spain

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Barcelona, , Spain

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Barcelona, , Spain

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Las Palmas, , Spain

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Madrid, , Spain

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Madrid, , Spain

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Murcia, , Spain

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Valladolid, , Spain

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Zaragoza, , Spain

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Cambridge, Cambridgeshire, United Kingdom

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London, Greater London, United Kingdom

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Manchester, Greater Manchester, United Kingdom

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Salford, Greater Manchester, United Kingdom

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Birmingham, West Midlands, United Kingdom

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Countries

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Denmark Poland Switzerland United States Australia Belgium Brazil Bulgaria Canada Croatia Czechia France Germany Greece Ireland Israel Italy Mexico Netherlands Portugal Russia Saudi Arabia Slovenia Spain United Kingdom

Central Contacts

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Alexion Pharmaceuticals, Inc. (Sponsor)

Role: CONTACT

Phone: 1-855-752-2356

Email: [email protected]

Facility Contacts

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Role: primary

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Chris Mitrevski

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Francois Feillet

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Michelle Santos Rondon

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Marcelo Yamamoto

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Rodrigo Rocco

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Mila Baycheva

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Katherine Schellevis

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Linda Hastie

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Lindsey McLeish

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Davy Eng

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Ivo Baric

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Miguel Angel Barba

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Jana Kňourková

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Lioara Vanstraelen

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Corinne Borderon

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Francois Feillet

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Patricio Aguiar

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Patricio Aguiar

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Miguel Angel Barba

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Angelique Hubert

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Patricio Aguiar

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Isabelle Simoneau

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Caroline Chevalier

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Miguel Angel Barba

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Miguel Angel Barba

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Friederike Spengler

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Adriana Gutierrez

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Elisabeth Steinhagen-Thiessen

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Elissavet Georgiadou

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Charalampos Milionis

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Maria Fotoulaki

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Ann Loughnane

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Yael Becker Ilani

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Liat Pritzker

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Tehila Ben-Moshe

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Irina Rabkin

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Albina Tummolo

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Valentina Pecorella

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Enrica Epifani

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Sharon Giuliana

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Marina Ferrario

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Domenico Azzolino

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Giulia Massini

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Giuseppe Indolfi

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Chiara Cazzorla

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Jorge c

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Jaime Lopez

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Alejandra Consuelo-Sanchez

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Nanet Sons

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Patricio Aguiar

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Olga Azevedo

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Patricio Aguiar

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Nikolay Vlasov

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Francois Feillet

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Natalia Pechatnikova

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Maria Zharkova

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Alexandr Potapov

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Alla Lavrova

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Hadeel Binomar

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Hala Al Anizi

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Damjana Nikovski

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Liz Morris

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Ana Amado Fondo

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Miguel Angel Barba

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Michaela Smyth

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Rachel Smith

Role: primary

References

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Soll D, Spira D, Hollstein T, Haberbosch L, Demuth I, Steinhagen-Thiessen E, Bobbert T, Spranger J, Kassner U. Clinical outcome of a patient with lysosomal acid lipase deficiency and first results after initiation of treatment with Sebelipase alfa: A case report. Mol Genet Metab Rep. 2019 Jun 18;20:100479. doi: 10.1016/j.ymgmr.2019.100479. eCollection 2019 Sep.

Reference Type DERIVED
PMID: 31249784 (View on PubMed)

D'Antiga L, Evans J, Ros E, Abel F, Balwani M, Wilson DP, Balistreri W. Sebelipase Alfa Improves Aminotransferase Levels in Lysosomal Acid Lipase Deficiency: Data From an International Registry. Liver Int. 2025 Sep;45(9):e70279. doi: 10.1111/liv.70279.

Reference Type DERIVED
PMID: 40781810 (View on PubMed)

Balwani M, Balistreri W, D'Antiga L, Evans J, Ros E, Abel F, Wilson DP. Lysosomal acid lipase deficiency manifestations in children and adults: Baseline data from an international registry. Liver Int. 2023 Jul;43(7):1537-1547. doi: 10.1111/liv.15620. Epub 2023 May 24.

Reference Type DERIVED
PMID: 37222260 (View on PubMed)

Related Links

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Other Identifiers

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ALX-LALD-501

Identifier Type: -

Identifier Source: org_study_id