Glivec in Ph Positive Lymphoblastic Leukemia

NCT ID: NCT00388895

Last Updated: 2008-11-19

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Clinical Phase

PHASE2

Total Enrollment

35 participants

Study Classification

INTERVENTIONAL

Study Start Date

2002-06-30

Study Completion Date

2007-10-31

Brief Summary

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% positive Ph LLA with RC alter the Glivec and induction chemotherapy treatment

Detailed Description

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Pilot phase II clinical trial, prospective, multicentric and opened

Conditions

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Acute Lymphoblastic Leukemia Cromosome Philadelphia Positive

Keywords

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Acute lymphoblastic Leukemia

Study Design

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Allocation Method

NON_RANDOMIZED

Intervention Model

SINGLE_GROUP

Primary Study Purpose

TREATMENT

Blinding Strategy

NONE

Interventions

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chemotherapy

Intervention Type DRUG

Glivec

Intervention Type DRUG

Eligibility Criteria

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Inclusion Criteria

* New diagnosis LLA Ph+ (BCR/ABL) patients ≤ 65 years old
* Fertile age women must do a pregnancy test in the 7 days previous at the beginning of clinical trial medication
* Performance status 0-2 (Appendix B); Is allowed performance status \> 2 because of LLA
* Patients without organ alteration: hepatic function: global bilirubin, AST, ALT, gamma-GT and alkaline phosphatase less than 2 times LSN; renal function: Creatinine \< 1,5 mg/dl o Clearance creatinine \> 60 ml/min; anormal renal function caused by LLA ; normal heart function (Appendix B): FEV \> 50%; No Chronic respiratory illness. If the anormal values are secondary of the experimental illness the investigator can decide himself if the patient can be included at the clinical trial.
* Negative HIV serology
* Written, oral or with witness informed consent. In patients \< 18 years old must be signed written and legal representative informed consent.
* No experimental chemotherapy or other experimental treatment. Allowed to begin induction chemotherapy from the diagnosis to confirm Ph. No major surgical process in the previous 14 days of the treatment Start.

Exclusion Criteria

* Other LLA variability
* Previous history of coronary valvular, hypertensive cardiopathy illness
* Chronic hepatic illness
* Chronic respiratory insufficiency
* Renal insufficiency not caused by LLA
* Severe neurological problems not caused by LLA
* Severe affection of the performance status (grade 3-4 OMS gradation) not caused by LLA
* Pregnancy and women
* Blastic crisis LMC
Maximum Eligible Age

65 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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PETHEMA Foundation

OTHER

Sponsor Role lead

Responsible Party

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pethema

Principal Investigators

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Ribera Josep Mª, Dr

Role: STUDY_CHAIR

Germans Trias i Pujol Hospital

Locations

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Hospital General de Alicante

Alicante, Alicante, Spain

Site Status

Hospital "Duran I Reynals"

Barcelona, Barcelona, Spain

Site Status

Hospital "Santa Creu i Sant Pau"

Barcelona, Barcelona, Spain

Site Status

Hospital Clínico y Provincial de Barcelona

Barcelona, Barcelona, Spain

Site Status

Hospital del Mar

Barcelona, Barcelona, Spain

Site Status

Hospital Germans Trias i Pujol

Barcelona, Barcelona, Spain

Site Status

Hospital Universitario "Germans Trias i Pujol"

Barcelona, Barcelona, Spain

Site Status

Hospital Valle Hebrón-Materno Infantil

Barcelona, Barcelona, Spain

Site Status

Hospital Valle Hebrón

Barcelona, Barcelona, Spain

Site Status

Hospital Universitario Marqués de Valdecilla

Santander, Cantabria, Spain

Site Status

Hospital Puerta del Mar

Cadiz, Cádiz, Spain

Site Status

Hospital Juan Canalejo

A Coruña, La Coruña, Spain

Site Status

Fundación Jiménez Díaz

Madrid, Madrid, Spain

Site Status

Hospital Clínico San Carlos de Madrid

Madrid, Madrid, Spain

Site Status

Hospital Doce de Octubre

Madrid, Madrid, Spain

Site Status

Hospital Gregorio Marañón

Madrid, Madrid, Spain

Site Status

Hospital Ramón y Cajal

Madrid, Madrid, Spain

Site Status

Hospital General Universitario Morales Meseguer.

Murcia, Murcia, Spain

Site Status

. Hospital Clínico Universitario Virgen de la Victoria

Málaga, Málaga, Spain

Site Status

Hospital Carlos Haya

Málaga, Málaga, Spain

Site Status

Clínica Universitaria de Navarra

Pamplona, Navarre, Spain

Site Status

Hospital Central de Asturias

Oviedo, Oviedo, Spain

Site Status

Hospital Son Dureta

Palma de Mallorca, Palma de Mallorca, Spain

Site Status

Hospital Son Llàtzer

Palma de Mallorca, Palma de Mallorca, Spain

Site Status

Hospital Universitario Virgen del Rocío

Seville, Sevilla, Spain

Site Status

Hospital Clínico de Valencia

Valencia, Valencia, Spain

Site Status

Hospital La Fe

Valencia, Valencia, Spain

Site Status

Hospital Clínico Universitario de Salamanca

Salamanca, , Spain

Site Status

Countries

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Spain

References

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Schrappe M, Reiter A, Zimmermann M, Harbott J, Ludwig WD, Henze G, Gadner H, Odenwald E, Riehm H. Long-term results of four consecutive trials in childhood ALL performed by the ALL-BFM study group from 1981 to 1995. Berlin-Frankfurt-Munster. Leukemia. 2000 Dec;14(12):2205-22. doi: 10.1038/sj.leu.2401973.

Reference Type BACKGROUND
PMID: 11187912 (View on PubMed)

Thomas X, Thiebaut A, Olteanu N, Danaila C, Charrin C, Archimbaud E, Fiere D. Philadelphia chromosome positive adult acute lymphoblastic leukemia: characteristics, prognostic factors and treatment outcome. Hematol Cell Ther. 1998 Jun;40(3):119-28.

Reference Type BACKGROUND
PMID: 9698220 (View on PubMed)

Snyder DS, Nademanee AP, O'Donnell MR, Parker PM, Stein AS, Margolin K, Somlo G, Molina A, Spielberger R, Kashyap A, Fung H, Slovak ML, Dagis A, Negrin RS, Amylon MD, Blume KG, Forman SJ. Long-term follow-up of 23 patients with Philadelphia chromosome-positive acute lymphoblastic leukemia treated with allogeneic bone marrow transplant in first complete remission. Leukemia. 1999 Dec;13(12):2053-8. doi: 10.1038/sj.leu.2401589.

Reference Type BACKGROUND
PMID: 10602428 (View on PubMed)

Arico M, Valsecchi MG, Camitta B, Schrappe M, Chessells J, Baruchel A, Gaynon P, Silverman L, Janka-Schaub G, Kamps W, Pui CH, Masera G. Outcome of treatment in children with Philadelphia chromosome-positive acute lymphoblastic leukemia. N Engl J Med. 2000 Apr 6;342(14):998-1006. doi: 10.1056/NEJM200004063421402.

Reference Type BACKGROUND
PMID: 10749961 (View on PubMed)

Druker BJ, Tamura S, Buchdunger E, Ohno S, Segal GM, Fanning S, Zimmermann J, Lydon NB. Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells. Nat Med. 1996 May;2(5):561-6. doi: 10.1038/nm0596-561.

Reference Type BACKGROUND
PMID: 8616716 (View on PubMed)

Druker BJ, Sawyers CL, Kantarjian H, Resta DJ, Reese SF, Ford JM, Capdeville R, Talpaz M. Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia with the Philadelphia chromosome. N Engl J Med. 2001 Apr 5;344(14):1038-42. doi: 10.1056/NEJM200104053441402.

Reference Type BACKGROUND
PMID: 11287973 (View on PubMed)

Saffroy R, Lemoine A, Brezillon P, Frenoy N, Delmas B, Goldschmidt E, Souleau B, Nedellec G, Debuire B. Real-time quantitation of bcr-abl transcripts in haematological malignancies. Eur J Haematol. 2000 Oct;65(4):258-66. doi: 10.1034/j.1600-0609.2000.065004258.x.

Reference Type BACKGROUND
PMID: 11073166 (View on PubMed)

Mitterbauer G, Nemeth P, Wacha S, Cross NC, Schwarzinger I, Jaeger U, Geissler K, Greinix HT, Kalhs P, Lechner K, Mannhalter C. Quantification of minimal residual disease in patients with BCR-ABL-positive acute lymphoblastic leukaemia using quantitative competitive polymerase chain reaction. Br J Haematol. 1999 Sep;106(3):634-43. doi: 10.1046/j.1365-2141.1999.01605.x.

Reference Type BACKGROUND
PMID: 10468851 (View on PubMed)

Tabernero MD, Bortoluci AM, Alaejos I, Lopez-Berges MC, Rasillo A, Garcia-Sanz R, Garcia M, Sayagues JM, Gonzalez M, Mateo G, San Miguel JF, Orfao A. Adult precursor B-ALL with BCR/ABL gene rearrangements displays a unique immunophenotype based on the pattern of CD10, CD34, CD13 and CD38 expresssion. Leukemia. 2001 Mar;15(3):406-14. doi: 10.1038/sj.leu.2402060.

Reference Type BACKGROUND
PMID: 11237064 (View on PubMed)

le Coutre P, Tassi E, Varella-Garcia M, Barni R, Mologni L, Cabrita G, Marchesi E, Supino R, Gambacorti-Passerini C. Induction of resistance to the Abelson inhibitor STI571 in human leukemic cells through gene amplification. Blood. 2000 Mar 1;95(5):1758-66.

Reference Type BACKGROUND
PMID: 10688835 (View on PubMed)

Mahon FX, Deininger MW, Schultheis B, Chabrol J, Reiffers J, Goldman JM, Melo JV. Selection and characterization of BCR-ABL positive cell lines with differential sensitivity to the tyrosine kinase inhibitor STI571: diverse mechanisms of resistance. Blood. 2000 Aug 1;96(3):1070-9.

Reference Type BACKGROUND
PMID: 10910924 (View on PubMed)

Weisberg E, Griffin JD. Mechanism of resistance to the ABL tyrosine kinase inhibitor STI571 in BCR/ABL-transformed hematopoietic cell lines. Blood. 2000 Jun 1;95(11):3498-505.

Reference Type BACKGROUND
PMID: 10828035 (View on PubMed)

Gambacorti-Passerini C, Barni R, le Coutre P, Zucchetti M, Cabrita G, Cleris L, Rossi F, Gianazza E, Brueggen J, Cozens R, Pioltelli P, Pogliani E, Corneo G, Formelli F, D'Incalci M. Role of alpha1 acid glycoprotein in the in vivo resistance of human BCR-ABL(+) leukemic cells to the abl inhibitor STI571. J Natl Cancer Inst. 2000 Oct 18;92(20):1641-50. doi: 10.1093/jnci/92.20.1641.

Reference Type BACKGROUND
PMID: 11036109 (View on PubMed)

Related Links

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http://www.aehh.org

Spanish association of Haematology

Other Identifiers

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02-0207 (nº AEMPS)

Identifier Type: -

Identifier Source: secondary_id

CSTI571BES02

Identifier Type: -

Identifier Source: org_study_id