Glucagon-like Peptide 1, Glucose Metabolism and Gastric Bypass

NCT ID: NCT01803451

Last Updated: 2025-09-09

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

RECRUITING

Clinical Phase

EARLY_PHASE1

Total Enrollment

80 participants

Study Classification

INTERVENTIONAL

Study Start Date

2005-11-30

Study Completion Date

2027-08-31

Brief Summary

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The overall goal of this project is to understand the mechanisms by which gastric bypass surgery improves glucose metabolism.

The central hypothesis guiding this project is that the reconfiguration of intestinal transit with the Roux-en-Y will increase the release of insulinotropic GI hormones, termed incretins that improve insulin secretion and glucose metabolism. The study is divided into three specific aims.

1. To determine the role of incretin hormones on insulin secretion in patients with gastric bypass surgery using intravenous-oral hyperglycemic clamp.
2. To compare incretin effect and glucose tolerance among patient who suffer from hypoglycemia after RYGB and asymptomatic surgical and non-surgical individuals.
3. To quantify the contribution of GLP-1 to incretin effect enhancement following surgery.

Detailed Description

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The overall goal of this project is to understand the mechanisms by which gastric bypass surgery improves glucose metabolism.

The central hypothesis guiding this project is that the reconfiguration of intestinal transit with the Roux-en-Y will increase the release of insulinotropic GI hormones, termed incretins that improve insulin secretion and glucose metabolism. The study is divided into three specific aims.

1. To determine the role of incretin hormones on insulin secretion in patients with gastric bypass surgery using intravenous-oral hyperglycemic clamp.
2. To compare incretin effect and glucose tolerance among patient who suffer from hypoglycemia after RYGB and asymptomatic surgical and non-surgical individuals.
3. To quantify the contribution of GLP-1 to incretin effect enhancement following surgery.

Conditions

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Post-bariatric Surgery

Study Design

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Allocation Method

NON_RANDOMIZED

Intervention Model

FACTORIAL

Primary Study Purpose

OTHER

Blinding Strategy

SINGLE

Participants

Study Groups

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hyperglycemic clamp-Meal tolerance test

these studies are to evaluate the effect of exendin-9 on insulin secretion before and after meal ingestion in patients after bariatric surgeries compared to non-surgical controls

Group Type EXPERIMENTAL

exendin-(9-39)

Intervention Type DRUG

hyperglycemic clamp-meal tolerance test is designed to assess insulin secretion before and after meal ingestion

Labeled meal tolerance test

The effect of GLP-1 receptor blockade on glucose tolerance and glucose kinetics are evaluated in the group patients with bariatric surgery vs. nonsurgical using exendin-9-39 infusion during one of the the 2-day dual tracer studies of meal tolerance test

Group Type EXPERIMENTAL

exendin -(9-39)

Intervention Type DRUG

2-day meal tolerance tests with labeled oral and IV glucose using exendin-(9-39) infusion are designed to evaluate the role of GLP-1 signaling on glucose tolerance and glucose kinetics.

Interventions

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exendin-(9-39)

hyperglycemic clamp-meal tolerance test is designed to assess insulin secretion before and after meal ingestion

Intervention Type DRUG

exendin -(9-39)

2-day meal tolerance tests with labeled oral and IV glucose using exendin-(9-39) infusion are designed to evaluate the role of GLP-1 signaling on glucose tolerance and glucose kinetics.

Intervention Type DRUG

Other Intervention Names

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no other name for exendin-(9-39) no other name for exendin-(9-39)

Eligibility Criteria

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Inclusion Criteria

* age 18-65
* healthy control without diabetes or active organ disease
* Individuals with bariatric surgery
* recurrent hypoglycemia post gastric bypass

Exclusion Criteria

* pregnancy
* significant anemia
* diabetes currently unless pre-op for bariatric surgery procedure
* GI obstruction
Minimum Eligible Age

18 Years

Maximum Eligible Age

65 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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The University of Texas Health Science Center at San Antonio

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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Marzieh Salehi, MD,MS

Role: PRINCIPAL_INVESTIGATOR

Marzieh Salehi

Locations

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Texas Diabetes Institute - University Health System

San Antonio, Texas, United States

Site Status RECRUITING

South Texas Veterans Health Care System

San Antonio, Texas, United States

Site Status RECRUITING

Countries

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United States

Central Contacts

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Marzieh Salehi, MD MS

Role: CONTACT

210-567-6691

Jennifer Foster, MSN

Role: CONTACT

210-450-8696

Facility Contacts

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Jennifer Foster, MSN

Role: primary

210-450-8696

Marzieh Salehi, MD, MS

Role: backup

210-567-6691

Marzieh Salehi, MD MS

Role: primary

210-567-6691

Jennifer Foster

Role: backup

210-450-8696

References

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Salehi M, Gastaldelli A, D'Alessio DA. Blockade of glucagon-like peptide 1 receptor corrects postprandial hypoglycemia after gastric bypass. Gastroenterology. 2014 Mar;146(3):669-680.e2. doi: 10.1053/j.gastro.2013.11.044. Epub 2013 Dec 4.

Reference Type DERIVED
PMID: 24315990 (View on PubMed)

Other Identifiers

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DK083554

Identifier Type: OTHER

Identifier Source: secondary_id

HSC20180070H

Identifier Type: -

Identifier Source: org_study_id

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