Does Green Tea Affect Postprandial Glucose, Insulin and Satiety in Healthy Subjects

NCT ID: NCT01086189

Last Updated: 2010-03-12

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Total Enrollment

14 participants

Study Classification

OBSERVATIONAL

Study Start Date

2009-12-31

Study Completion Date

2010-02-28

Brief Summary

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Green tea consumption could lower the risk of type II diabetes, as suggested by epidemiological studies. There is also evidence from intervention studies that green tea can decrease blood glucose levels and contribute to weight loss.

The aim with this study is therefore to examine the postprandial effects of green tea on glycemic index, insulin levels and satiety in healthy individuals after the consumption of a meal.

Detailed Description

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The study was conducted on 14 healthy volunteers, with a crossover design. Participants were randomized to either green tea or water. This was consumed together with a breakfast consisting of white bread. Blood samples were drawn at 0, 15, 30, 45, 60, 90 and 120 minutes. Participants completed two different satiety scores at the same time intervals.

Conditions

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Diabetes Type 2 Obesity

Study Design

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Observational Model Type

CASE_CROSSOVER

Study Time Perspective

CROSS_SECTIONAL

Eligibility Criteria

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Inclusion Criteria

* Healthy subjects

Exclusion Criteria

* Diabetes
Minimum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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Skane University Hospital

OTHER

Sponsor Role lead

Locations

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Skane University Hospital

Malmo, Skåne County, Sweden

Site Status

Countries

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Sweden

References

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Josic J, Olsson AT, Wickeberg J, Lindstedt S, Hlebowicz J. Does green tea affect postprandial glucose, insulin and satiety in healthy subjects: a randomized controlled trial. Nutr J. 2010 Nov 30;9:63. doi: 10.1186/1475-2891-9-63.

Reference Type DERIVED
PMID: 21118565 (View on PubMed)

Other Identifiers

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2009/66

Identifier Type: -

Identifier Source: org_study_id

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