Intravenous Mepolizumab In Subjects With Hypereosinophilic Syndromes (HES)

NCT ID: NCT00086658

Last Updated: 2020-05-05

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Clinical Phase

PHASE3

Total Enrollment

86 participants

Study Classification

INTERVENTIONAL

Study Start Date

2004-03-23

Study Completion Date

2006-05-01

Brief Summary

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Hypereosinophilic syndrome (HES) is a rare disease with broad clinical signs and symptoms which is diagnosed based on a persistent blood eosinophil count of greater than 1500 cells, various end-organ damages (including skin, heart, lung, nervous system and digestive system etc.), and with exclusion of known secondary causes of hypereosinophilia.

HES has a high morbidity/mortality rate. The major treatment of HES has been systemic corticosteroid and other chemotherapeutic drugs (for example, hydroxyurea and interferon) with the intention to lower eosinophil counts and therefore to slow down the progression of disease. Even though corticosteroid and other therapies can effectively reduce eosinophilia in some patients, some may eventually become nonresponsive and intolerable to the amount of side effects of the long-term therapy with these medications.

Mepolizumab is a humanized monoclonal antibody that binds specifically to human interleukin 5 (hIL-5) and inhibits its activity. Previous human experience has shown it has been effective in reducing blood eosinophilia in atopic and HES patients and has alleviated some HES clinical signs and symptoms. This study intends to further evaluate the corticosteroid-sparing and clinical benefit of mepolizumab in HES.

Detailed Description

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A Multicenter, Randomized, Double-blind, Placebo-controlled, Parallel Group Phase III Study to Evaluate Corticosteroid-reduction and -sparing effects of Mepolizumab 750 mg intravenously in Subjects with Hypereosinophilic Syndromes (HES) and to evaluate the Efficacy and Safety of Mepolizumab in controlling the Clinical Signs and Symptoms of HES over Nine Months

Conditions

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Hypereosinophilia Hypereosinophilic Syndrome

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

TREATMENT

Blinding Strategy

DOUBLE

Interventions

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mepolizumab

Intervention Type DRUG

Eligibility Criteria

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Inclusion Criteria

* Documented history of Hypereosinophilic Syndrome (HES)
* Eosinophil count greater than 1500 cells for 6 months
* Signs and symptoms of organ system involvement
* No evidence of parasitic, allergic or other causes of eosinophilia after comprehensive evaluation.
* Achieve and maintain a stable prednisone (corticosteroid) dose prior to starting study medication.
* Not pregnant or nursing.

Exclusion Criteria

* Churg-Strauss Syndrome
* Wegener's Granulomatosis
* Lymphoma, hematological malignancy, advanced and metastatic solid tumors
* Chemotherapy, radiotherapy or interleukin 2 treatment.
Minimum Eligible Age

18 Years

Maximum Eligible Age

75 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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GlaxoSmithKline

INDUSTRY

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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GSK Clinical Trials

Role: STUDY_DIRECTOR

GlaxoSmithKline

Locations

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GSK Investigational Site

San Diego, California, United States

Site Status

GSK Investigational Site

Denver, Colorado, United States

Site Status

GSK Investigational Site

Bethesda, Maryland, United States

Site Status

GSK Investigational Site

Boston, Massachusetts, United States

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GSK Investigational Site

Rochester, Minnesota, United States

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GSK Investigational Site

Cincinnati, Ohio, United States

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GSK Investigational Site

Nashville, Tennessee, United States

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GSK Investigational Site

Houston, Texas, United States

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GSK Investigational Site

Salt Lake City, Utah, United States

Site Status

GSK Investigational Site

Richmond, Virginia, United States

Site Status

GSK Investigational Site

Madison, Wisconsin, United States

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GSK Investigational Site

St Leonards, New South Wales, Australia

Site Status

GSK Investigational Site

South Brisbane, Queensland, Australia

Site Status

GSK Investigational Site

Melbourne, Victoria, Australia

Site Status

GSK Investigational Site

West Perth, Western Australia, Australia

Site Status

GSK Investigational Site

Leuven, , Belgium

Site Status

GSK Investigational Site

Edmonton, Alberta, Canada

Site Status

GSK Investigational Site

Winnipeg, Manitoba, Canada

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GSK Investigational Site

Halifax, Nova Scotia, Canada

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GSK Investigational Site

Hamilton, Ontario, Canada

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GSK Investigational Site

Toronto, Ontario, Canada

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GSK Investigational Site

Montreal, Quebec, Canada

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GSK Investigational Site

Lille, , France

Site Status

GSK Investigational Site

Suresnes, , France

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GSK Investigational Site

Munich, Bavaria, Germany

Site Status

GSK Investigational Site

Hanover, Lower Saxony, Germany

Site Status

GSK Investigational Site

Bad Bramstedt, Schleswig-Holstein, Germany

Site Status

GSK Investigational Site

Bologna, Emilia-Romagna, Italy

Site Status

GSK Investigational Site

Bern, , Switzerland

Site Status

Countries

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United States Australia Belgium Canada France Germany Italy Switzerland

References

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Roufosse F, de Lavareille A, Schandene L, Cogan E, Georgelas A, Wagner L, Xi L, Raffeld M, Goldman M, Gleich GJ, Klion A. Mepolizumab as a corticosteroid-sparing agent in lymphocytic variant hypereosinophilic syndrome. J Allergy Clin Immunol. 2010 Oct;126(4):828-835.e3. doi: 10.1016/j.jaci.2010.06.049.

Reference Type DERIVED
PMID: 20810155 (View on PubMed)

Rothenberg ME, Klion AD, Roufosse FE, Kahn JE, Weller PF, Simon HU, Schwartz LB, Rosenwasser LJ, Ring J, Griffin EF, Haig AE, Frewer PI, Parkin JM, Gleich GJ; Mepolizumab HES Study Group. Treatment of patients with the hypereosinophilic syndrome with mepolizumab. N Engl J Med. 2008 Mar 20;358(12):1215-28. doi: 10.1056/NEJMoa070812. Epub 2008 Mar 16.

Reference Type DERIVED
PMID: 18344568 (View on PubMed)

Other Identifiers

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100185

Identifier Type: -

Identifier Source: org_study_id

NCT00081445

Identifier Type: -

Identifier Source: nct_alias

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