Pirfenidone: A New Drug to Treat Kidney Disease in Patients With Diabetes

NCT ID: NCT00063583

Last Updated: 2009-11-04

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Clinical Phase

PHASE1/PHASE2

Total Enrollment

77 participants

Study Classification

INTERVENTIONAL

Study Start Date

2003-06-30

Study Completion Date

2009-03-31

Brief Summary

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The purpose of this study is to determine whether a new investigational drug, pirfenidone, will be an effective therapy for diabetic patients with kidney dysfunction. Our hypothesis is that administration of pirfenidone to type 1 and type 2 diabetic patients with advanced kidney disease will lead to preservation of kidney function.

Detailed Description

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Diabetic kidney disease is the leading cause of new cases of kidney failure in the United States. In the kidneys of diabetic patients, there is accumulation of protein that leads to the formation of scar tissue and poor kidney function. Because of this many patients eventually require dialysis or kidney transplantation. A new investigational drug, pirfenidone, has been shown to be beneficial in a number of diseases in which scar formation leads to disease progression. It is our goal to examine whether pirfenidone is effective at stabilizing or reducing progressive diabetic kidney dysfunction.

Conditions

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Diabetes Mellitus Diabetic Nephropathy

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

TREATMENT

Blinding Strategy

TRIPLE

Participants Caregivers Investigators

Study Groups

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Placebo

Placebo

Group Type PLACEBO_COMPARATOR

Pirfenidone

Intervention Type DRUG

Pirfenidone will be administered orally at 1200 or 2400 mg day in divided doses

Pirfenidone 1200 mg/day

Pirfenidone will be administered at a dose of 1200 mg/day

Group Type EXPERIMENTAL

Pirfenidone

Intervention Type DRUG

Pirfenidone will be administered orally at 1200 or 2400 mg day in divided doses

Pirfenidone 2400 mg/day

Pirfenidone will be administered at 2400 mg/day

Group Type EXPERIMENTAL

Pirfenidone

Intervention Type DRUG

Pirfenidone will be administered orally at 1200 or 2400 mg day in divided doses

Interventions

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Pirfenidone

Pirfenidone will be administered orally at 1200 or 2400 mg day in divided doses

Intervention Type DRUG

Eligibility Criteria

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Inclusion Criteria

* Type 1 or type 2 diabetes
* Males and females greater than or equal to 18 years.
* Abnormal kidney function determined by glomerular filtration rate
* History of proteinuria
* Blood pressure controlled to \<140/90 on anti-hypertensive medication

Exclusion

* Cancer, liver disease, hepatitis, HIV+
* History of heart attack, unstable angina, stroke or peptic ulcer in the past 6 months
* Pregnant or planning to become pregnant during the study period
* Other known kidney disease besides diabetic nephropathy
* Expect to begin dialysis or receive a kidney transplant within 1 year of study enrollment
Minimum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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National Institutes of Health (NIH)

NIH

Sponsor Role collaborator

National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)

NIH

Sponsor Role collaborator

Sharma, Kumar, M.D.

INDIV

Sponsor Role lead

Responsible Party

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UCSD

Principal Investigators

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Kumar Sharma, M.D.

Role: PRINCIPAL_INVESTIGATOR

UCSD

Locations

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National Institute of Diabetes and Digestive and Kidney Disease (NIDDK)

Bethesda, Maryland, United States

Site Status

Mayo Clinic

Rochester, Minnesota, United States

Site Status

The Center for Diabetic Kidney Disease at Thomas Jefferson University

Philadelphia, Pennsylvania, United States

Site Status

Countries

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United States

References

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Sharma K, Ziyadeh FN, Alzahabi B, McGowan TA, Kapoor S, Kurnik BR, Kurnik PB, Weisberg LS. Increased renal production of transforming growth factor-beta1 in patients with type II diabetes. Diabetes. 1997 May;46(5):854-9. doi: 10.2337/diab.46.5.854.

Reference Type BACKGROUND
PMID: 9133555 (View on PubMed)

Shimizu F, Fukagawa M, Yamauchi S, Taniyama M, Komemushi S, Margolin SB, Kurokawa K: Pirfenidone prevents the progression of irreversible glomerular sclerotic lesions in rats. Nephrology 3:315-322, 1997

Reference Type BACKGROUND

Shimizu T, Fukagawa M, Kuroda T, Hata S, Iwasaki Y, Nemoto M, Shirai K, Yamauchi S, Margolin SB, Shimizu F, Kurokawa K. Pirfenidone prevents collagen accumulation in the remnant kidney in rats with partial nephrectomy. Kidney Int Suppl. 1997 Dec;63:S239-43.

Reference Type BACKGROUND
PMID: 9407470 (View on PubMed)

Iyer SN, Gurujeyalakshmi G, Giri SN. Effects of pirfenidone on transforming growth factor-beta gene expression at the transcriptional level in bleomycin hamster model of lung fibrosis. J Pharmacol Exp Ther. 1999 Oct;291(1):367-73.

Reference Type BACKGROUND
PMID: 10490926 (View on PubMed)

McGowan T, Dunn SR, Sharma K: Treatment of db/db mice with pirfenidone leads to improved histology and serum creatinine. J Am Soc Nephrology 11:A2814, 2000

Reference Type BACKGROUND

Raghu G, Johnson WC, Lockhart D, Mageto Y. Treatment of idiopathic pulmonary fibrosis with a new antifibrotic agent, pirfenidone: results of a prospective, open-label Phase II study. Am J Respir Crit Care Med. 1999 Apr;159(4 Pt 1):1061-9. doi: 10.1164/ajrccm.159.4.9805017.

Reference Type BACKGROUND
PMID: 10194146 (View on PubMed)

Other Identifiers

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R01DK063017

Identifier Type: NIH

Identifier Source: secondary_id

View Link

1R01DK063017-01

Identifier Type: NIH

Identifier Source: org_study_id

View Link

NCT00105391

Identifier Type: -

Identifier Source: nct_alias

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