Effect of Intravenous Methylprednisolone and Intravenous Erythropoietin in Toxic Optic Neuropathies: Randomized Clinical Trial.

NCT ID: NCT05748561

Last Updated: 2023-02-28

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

PHASE2/PHASE3

Total Enrollment

18 participants

Study Classification

INTERVENTIONAL

Study Start Date

2022-04-05

Study Completion Date

2024-04-05

Brief Summary

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The goal of this double-blind prospective randomized clinical trial is to determine if the effect of intravenous erythropoietin is superior to the effect of intravenous methylprednisolone in cases of toxic optic neuropathy 4 weeks after therapeutic intervention.

The main question it aims to answer:

• Is there a difference in the visual recovery of toxic optic neuropathies treated with intravenous methylprednisolone in comparison with those treated with intravenous erythropoietin?

Detailed Description

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A double-blind prospective randomized clinical trial of treatment for toxic optic neuropathies comparing visual outcome of patients treated by standard treatment (intravenous methylprednisolone) vs intravenous erythropoietin.

Enrollment: 18. Randomized groups (2)

1. Standard treatment (intravenous methylprednisolone)
2. Intravenous erythropoietin

Masking: Double (participant and outcomes assessor) Participants won't be aware to which group they were assigned. Investigator in charge of assessing outcomes and analyzing data won't be aware to which group participants were assigned

Conditions

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Toxic Optic Neuropathy Treatment Methylprednisolone Erythropoietin

Keywords

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Toxic Optic Neuropathy Methylprednisolone Erythropoietin

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Randomized groups (2)

1. Standard treatment (Intravenous methylprednisolone)
2. Intervention (Intravenous erythropoietin)
Primary Study Purpose

TREATMENT

Blinding Strategy

DOUBLE

Participants Outcome Assessors
Participants won't be aware to which group they were assigned. Investigator in charge of assessing outcomes and analyzing data won't be aware to which group participants were assigned.

Study Groups

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Experimental group

The patients will receive intravenous erythropoietin - 10,000 IU every 24 hours for 5 days.

Group Type EXPERIMENTAL

Recombinant human erythropoietin 4,000 UI and 2,000 UI

Intervention Type DRUG

Intravenous recombinant human erythropoietin (10,000 IU every 24 hours for 5 days)

Control group

The patients will receive intravenous methylprednisolone - 1 g every 24 hours for 5 days.

Group Type PLACEBO_COMPARATOR

Methylprednisolone succinate 500 mg

Intervention Type DRUG

Intravenous Methylprednisolone succinate (1 g daily for 5 days)

Interventions

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Recombinant human erythropoietin 4,000 UI and 2,000 UI

Intravenous recombinant human erythropoietin (10,000 IU every 24 hours for 5 days)

Intervention Type DRUG

Methylprednisolone succinate 500 mg

Intravenous Methylprednisolone succinate (1 g daily for 5 days)

Intervention Type DRUG

Other Intervention Names

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Experimental Group Control Group

Eligibility Criteria

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Inclusion Criteria

* Both genres.
* Age between 18 and 75 years.
* Clinical diagnosis of toxic optic neuropathy (afferent pupillary defect, acquired dyschromatopsia, visual loss and bilateral prechiasmatic field defect).
* Exposure with a temporal relationship of less than two weeks to a known toxicant for the function of the optic nerve.
* Up to 21 days from symptom onset.
* Informed consent signature.

Exclusion Criteria

* History of previous optic neuropathy.
* History of additional ophthalmological or neurological pathology that has caused permanent visual loss.
* History of previous treatment with intravenous methylprednisolone or some other experimental treatment since the onset of symptoms.
* Poorly controlled diabetes mellitus.
* Poorly controlled systemic arterial hypertension.
* Hemoglobin \>16 mg/dL
* Patients with a history of thromboembolic event.
* Patients with a history of coronary heart disease, myocardial infarction or cerebral vascular event.
* Pregnancy or lactation.
Minimum Eligible Age

18 Years

Maximum Eligible Age

75 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Asociación para Evitar la Ceguera en México

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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Jorge Cárdenas-Belaunzarán, MD, MSc

Role: STUDY_DIRECTOR

Asociación Para Evitar la Ceguera en México I.A.P

Elsa Hernández-Piñamora, MD

Role: PRINCIPAL_INVESTIGATOR

Asociación Para Evitar la Ceguera en México I.A.P

Octavio Turcio-Aceves, MD

Role: PRINCIPAL_INVESTIGATOR

Asociación Para Evitar la Ceguera en México I.A.P

Locations

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Jorge Cárdenas Belaunzarán

Mexico City, , Mexico

Site Status RECRUITING

Countries

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Mexico

Central Contacts

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Jorge Cárdenas-Belaunzarán, MD, MSc

Role: CONTACT

Phone: 5544600113

Email: [email protected]

Octavio Turcio-Aceves, MD

Role: CONTACT

Phone: 5526951290

Email: [email protected]

Facility Contacts

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Jorge Cárdenas-Belaunzarán, MD, MSc

Role: primary

References

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Tan H, Kang X, Zhong Y, Shen X, Cheng Y, Jiao Q, Deng L. Erythropoietin upregulates growth associated protein-43 expression and promotes retinal ganglion cell axonal regeneration in vivo after optic nerve crush. Neural Regen Res. 2012 Feb 5;7(4):295-301. doi: 10.3969/j.issn.1673-5374.2012.04.010.

Reference Type BACKGROUND
PMID: 25806072 (View on PubMed)

Sharpe JA, Hostovsky M, Bilbao JM, Rewcastle NB. Methanol optic neuropathy: a histopathological study. Neurology. 1982 Oct;32(10):1093-100. doi: 10.1212/wnl.32.10.1093.

Reference Type BACKGROUND
PMID: 6889696 (View on PubMed)

Naeser P. Optic nerve involvement in a case of methanol poisoning. Br J Ophthalmol. 1988 Oct;72(10):778-81. doi: 10.1136/bjo.72.10.778.

Reference Type BACKGROUND
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Sun Y, Calvert JW, Zhang JH. Neonatal hypoxia/ischemia is associated with decreased inflammatory mediators after erythropoietin administration. Stroke. 2005 Aug;36(8):1672-8. doi: 10.1161/01.STR.0000173406.04891.8c. Epub 2005 Jul 21.

Reference Type BACKGROUND
PMID: 16040592 (View on PubMed)

Pakdel F, Sanjari MS, Naderi A, Pirmarzdashti N, Haghighi A, Kashkouli MB. Erythropoietin in Treatment of Methanol Optic Neuropathy. J Neuroophthalmol. 2018 Jun;38(2):167-171. doi: 10.1097/WNO.0000000000000614.

Reference Type BACKGROUND
PMID: 29300238 (View on PubMed)

Sharma R, Marasini S, Sharma AK, Shrestha JK, Nepal BP. Methanol poisoning: ocular and neurological manifestations. Optom Vis Sci. 2012 Feb;89(2):178-82. doi: 10.1097/OPX.0b013e31823ee128.

Reference Type BACKGROUND
PMID: 22127151 (View on PubMed)

Pakravan M, Esfandiari H, Sanjari N, Ghahari E. Erythropoietin as an adjunctive treatment for methanol-induced toxic optic neuropathy. Am J Drug Alcohol Abuse. 2016 Nov;42(6):633-639. doi: 10.1080/00952990.2016.1198800. Epub 2016 Jul 27.

Reference Type BACKGROUND
PMID: 27463192 (View on PubMed)

Kashkouli MB, Pakdel F, Sanjari MS, Haghighi A, Nojomi M, Homaee MH, Heirati A. Erythropoietin: a novel treatment for traumatic optic neuropathy-a pilot study. Graefes Arch Clin Exp Ophthalmol. 2011 May;249(5):731-6. doi: 10.1007/s00417-010-1534-3. Epub 2010 Oct 2.

Reference Type BACKGROUND
PMID: 20890611 (View on PubMed)

Entezari M, Esmaeili M, Yaseri M. A pilot study of the effect of intravenous erythropoetin on improvement of visual function in patients with recent indirect traumatic optic neuropathy. Graefes Arch Clin Exp Ophthalmol. 2014 Aug;252(8):1309-13. doi: 10.1007/s00417-014-2691-6. Epub 2014 Jul 2.

Reference Type BACKGROUND
PMID: 24986593 (View on PubMed)

Shayegannejad V, Shahzamani S, Dehghani A, Dast Borhan Z, Rahimi M, Mirmohammadsadeghi A. A double-blind, placebo-controlled trial of adding erythropoietin to intravenous methylprednisolone for the treatment of unilateral acute optic neuritis of unknown or demyelinative origin. Graefes Arch Clin Exp Ophthalmol. 2015 May;253(5):797-801. doi: 10.1007/s00417-014-2925-7. Epub 2015 Jan 22.

Reference Type BACKGROUND
PMID: 25605544 (View on PubMed)

Behbehani R. Clinical approach to optic neuropathies. Clin Ophthalmol. 2007 Sep;1(3):233-46.

Reference Type BACKGROUND
PMID: 19668477 (View on PubMed)

Grzybowski A, Zulsdorff M, Wilhelm H, Tonagel F. Toxic optic neuropathies: an updated review. Acta Ophthalmol. 2015 Aug;93(5):402-410. doi: 10.1111/aos.12515. Epub 2014 Aug 27.

Reference Type BACKGROUND
PMID: 25159832 (View on PubMed)

Karimi S, Arabi A, Shahraki T. Alcohol and the Eye. J Ophthalmic Vis Res. 2021 Apr 29;16(2):260-270. doi: 10.18502/jovr.v16i2.9089. eCollection 2021 Apr-Jun.

Reference Type BACKGROUND
PMID: 34055263 (View on PubMed)

Kraut JA, Kurtz I. Toxic alcohol ingestions: clinical features, diagnosis, and management. Clin J Am Soc Nephrol. 2008 Jan;3(1):208-25. doi: 10.2215/CJN.03220807. Epub 2007 Nov 28.

Reference Type BACKGROUND
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Feizi S, Alemzadeh-Ansari M, Karimian F, Esfandiari H. Use of erythropoietin in ophthalmology: a review. Surv Ophthalmol. 2022 Mar-Apr;67(2):427-439. doi: 10.1016/j.survophthal.2021.06.002. Epub 2021 Jun 23.

Reference Type BACKGROUND
PMID: 34157346 (View on PubMed)

Other Identifiers

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NEU-22-02

Identifier Type: -

Identifier Source: org_study_id