Effect of Acarbose and Vildagliptin on Visceral Fat Distribution in Newly Diagnosed Type 2 Diabetes Patients

NCT ID: NCT02999841

Last Updated: 2016-12-21

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

PHASE4

Total Enrollment

100 participants

Study Classification

INTERVENTIONAL

Study Start Date

2016-03-31

Study Completion Date

2018-01-31

Brief Summary

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Focusing on newly diagnosed type 2 diabetes participants with overweight and obesity (24kg/m2 ≤ body mass index ≤ 30kg/m2).

50 participants per arm (acarbose \& lifestyle combination / vildagliptin \& lifestyle combination), using abdominal computed tomography scans and other methods to evaluate the effects of acarbose and vildagliptin on visceral fat distribution in overweight and obesity patients with newly diagnosed type 2 diabetes.

Detailed Description

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Conditions

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Diabetes Mellitus, Type 2 Obesity

Keywords

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Visceral fat

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

TREATMENT

Blinding Strategy

NONE

Study Groups

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Group A

Acarbose

Group Type EXPERIMENTAL

Acarbose

Intervention Type DRUG

1-2 week: 50mg tid; 3-24 week: 100mg tid.

Group B

Vildagliptin

Group Type ACTIVE_COMPARATOR

Vildagliptin

Intervention Type DRUG

1-24 week: 50mg bid

Interventions

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Acarbose

1-2 week: 50mg tid; 3-24 week: 100mg tid.

Intervention Type DRUG

Vildagliptin

1-24 week: 50mg bid

Intervention Type DRUG

Other Intervention Names

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Glucobay (Acarbose Tablets) Galvus (Vidagliptin Tablets)

Eligibility Criteria

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Inclusion Criteria

* All patients was diagnosed within the past 12 months with type 2 diabetes patients (WHO, 1999 criteria ).
* Not received oral anti-diabetic drugs or has been on short-term(1month) treatment that had been discontinued 3 months before enrollment.
* 30 ≤ Age ≤ 70 years old, male or female.
* HbA1c between 7% and 9% (7.0% ≤ HbA1c ≤9.0%).
* 24 ≤ BMI ≤ 30 kg/m2.
* Written Informed consent.

Exclusion Criteria

* Subject with type 1 diabetes or gestational diabetes mellitus and other specific types DM.
* Those who can not tolerate AGI or who is suffering GI disease.
* Subject with repeated severe hypoglycemia and/or unawareness of hypoglycemia.
* Known or suspected allergy to trial product(s) or related products.
* Females of child bearing potential who are pregnant, breast-feeding or have the intention of becoming pregnant or not using adequate contraceptive methods throughout the trial
* Impaired liver function, defined as ALT≥2 or AST≥ 2 times upper referenced limit times upper normal limit.
* Any other clinically significant condition or major systemic diseases, including serious coronary heart disease, cardiovascular disease, cancer, TB, acute infection.
* Endocrine diseases (hypo thyroidism, hyperthyroidism,Cushing's syndrome).
* Uncontrolled hypertension(SBP≥180mmHg and/or DBP≥100mmHg).
* Diabetic ketoacidosis; or hyperosmolar non-ketotic coma.
* Concomitant treatment which influences blood glucose and bodyweight.
* Impaired renal function(Cr≥ 1.5 mg/dl in male or Cr≥1.4 mg/dl in female).
* Mental disorders; drug or other substance misuse.
* Participation in any drug clinical trials during the past 3 months before enrolment.
Minimum Eligible Age

30 Years

Maximum Eligible Age

70 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Peking University

OTHER

Sponsor Role lead

Responsible Party

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Linong Ji

Director of Department of Endocrinology and Metabolism, Peking University People's Hospital.

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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Linong Ji, MD

Role: PRINCIPAL_INVESTIGATOR

Peking University People's Hospital

Locations

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Beijing Pinggu Hospital

Beijing, Beijing Municipality, China

Site Status RECRUITING

Countries

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China

Central Contacts

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Linong Ji, MD

Role: CONTACT

Phone: +86 10 8832 4108

Facility Contacts

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Yufeng Li, MD

Role: primary

References

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Ibrahim MM. Subcutaneous and visceral adipose tissue: structural and functional differences. Obes Rev. 2010 Jan;11(1):11-8. doi: 10.1111/j.1467-789X.2009.00623.x. Epub 2009 Jul 28.

Reference Type RESULT
PMID: 19656312 (View on PubMed)

Schernthaner GH, Schernthaner G. Insulin resistance and inflammation in the early phase of type 2 diabetes: potential for therapeutic intervention. Scand J Clin Lab Invest Suppl. 2005;240:30-40. doi: 10.1080/00365510500236119.

Reference Type RESULT
PMID: 16112958 (View on PubMed)

Monami M, Iacomelli I, Marchionni N, Mannucci E. Dipeptydil peptidase-4 inhibitors in type 2 diabetes: a meta-analysis of randomized clinical trials. Nutr Metab Cardiovasc Dis. 2010 May;20(4):224-35. doi: 10.1016/j.numecd.2009.03.015. Epub 2009 Jun 9.

Reference Type RESULT
PMID: 19515542 (View on PubMed)

Kodama N, Tahara N, Tahara A, Honda A, Nitta Y, Mizoguchi M, Kaida H, Ishibashi M, Abe T, Ikeda H, Narula J, Fukumoto Y, Yamagishi S, Imaizumi T. Effects of pioglitazone on visceral fat metabolic activity in impaired glucose tolerance or type 2 diabetes mellitus. J Clin Endocrinol Metab. 2013 Nov;98(11):4438-45. doi: 10.1210/jc.2013-2920. Epub 2013 Sep 12.

Reference Type RESULT
PMID: 24030946 (View on PubMed)

Yang X, Zhang X, Sun C, Zhao C, Kong X, Zhao M, Ji L, Li Y. Effect of acarbose and vildagliptin on plasma trimethylamine N-oxide levels in patients with type 2 diabetes mellitus: a 6-month, two-arm randomized controlled trial. Front Endocrinol (Lausanne). 2025 May 6;16:1575087. doi: 10.3389/fendo.2025.1575087. eCollection 2025.

Reference Type DERIVED
PMID: 40395816 (View on PubMed)

Zhang X, Ren H, Zhao C, Shi Z, Qiu L, Yang F, Zhou X, Han X, Wu K, Zhong H, Li Y, Li J, Ji L. Metagenomic analysis reveals crosstalk between gut microbiota and glucose-lowering drugs targeting the gastrointestinal tract in Chinese patients with type 2 diabetes: a 6 month, two-arm randomised trial. Diabetologia. 2022 Oct;65(10):1613-1626. doi: 10.1007/s00125-022-05768-5. Epub 2022 Aug 5.

Reference Type DERIVED
PMID: 35930018 (View on PubMed)

Other Identifiers

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2119000273

Identifier Type: -

Identifier Source: org_study_id