"Manual Dexterity and Oculomotor Control in Schizophrenia"

NCT ID: NCT02826629

Last Updated: 2017-10-19

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

NA

Total Enrollment

105 participants

Study Classification

INTERVENTIONAL

Study Start Date

2016-07-26

Study Completion Date

2019-01-31

Brief Summary

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The investigators recently showed that visuomotor integration was significantly altered in schizophrenic patients during: (i) a grip force task (Teremetz et al., 2014), and (ii) a saccadic paradigm (oculomotor task)(Amado et al., 2008). Given this findings, the investigators propose a combined study of oculomotor and grip force control to better characterize the sensorimotor integration deficit. This approach may allow for identification of behavioural biomarkers of vulnerability to develop schizophrenia.

Detailed Description

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1 - Scientific background and rational Use of sensory cues is essential for execution and correction of voluntary movements. The motor areas and their regulation is of special interest in patients with schizophrenia as there is clear evidence of motor abnormalities independent of the effects of antipsychotic medication, even before the onset of the disorder. Sensorimotor abnormalities have been proposed as a valid endophenotype in schizophrenia. Our global objective is to study and provide vulnerability markers for schizophrenia.

1. Control of manual dexterity will be assessed by a force sensor (Power Grip Manipulandum, PGM)
2. Oculomotor movements during behavioral task will be recorded using a video-oculography device
3. The involvement of cortical inhibition in this volitional inhibition task will be studied by neuronavigation guided TMS coupled to EMG recording

2 - Description of the project methodology There is strong evidence for schizophrenia being a neuro-developmental disorder (Rapoport et al., 2005). It has been shown, for many years, that patients with schizophrenia exhibit abnormal patterns of sensorimotor integration (Manschreck et al., 1982), which is the capacity to integrate different sensory stimuli into appropriate motor actions. It is clinically relevant, in terms of early diagnosis and prevention, whether deficient sensorimotor integration is present in the prodromal phase of schizophrenia, and whether this constitutes a vulnerability marker for the disease.

Our global objective is to study the interactions and related substratum of oculomotor movements during force control task.

The secondary objectives:

(i) To show that increased motor noise is indeed present in schizophrenia. (ii) To show by TMS that cortical excitability in the primary motor cortex (M1) is task-modulated and decreased in schizophrenia.

(iii) Assess the role of deficient cortical inhibition in these behavioral deficits To this end, three different groups of subjects will be studied: schizophrenic patients, non-affected siblings, ultra high risk patients, non-treated schizophrenic patients and healthy control subjects.

Conditions

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Schizophrenia

Keywords

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Sensorimotor Integration Schizophrenia TMS Oculomotor Motor Control

Study Design

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Allocation Method

NON_RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

BASIC_SCIENCE

Blinding Strategy

NONE

Study Groups

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Schizophrenia

40 patients with diagnosis of schizophrenia (25 medicated - 15 non-medicated)

Group Type OTHER

Manual dexterity

Intervention Type DEVICE

Control of manual dexterity will be assessed by a force sensor (Power Grip Manipulandum, PGM)

Oculomotor movements

Intervention Type DEVICE

Oculomotor movements during behavioral task will be recorded using a video-oculography device

TMS coupled to EMG recording

Intervention Type DEVICE

The involvement of cortical inhibition in this volitional inhibition task will be studied by neuronavigation guided TMS coupled to EMG recording

Psychopathological evaluations

Intervention Type OTHER

Neuropsychological evaluations

Intervention Type OTHER

Healthy sibling

25 healthy siblings

Group Type OTHER

Manual dexterity

Intervention Type DEVICE

Control of manual dexterity will be assessed by a force sensor (Power Grip Manipulandum, PGM)

Oculomotor movements

Intervention Type DEVICE

Oculomotor movements during behavioral task will be recorded using a video-oculography device

TMS coupled to EMG recording

Intervention Type DEVICE

The involvement of cortical inhibition in this volitional inhibition task will be studied by neuronavigation guided TMS coupled to EMG recording

Psychopathological evaluations

Intervention Type OTHER

Neuropsychological evaluations

Intervention Type OTHER

Ultra high risk for developing Schizophrenia

15 patients with ultra high risk for developing Schizophrenia

Group Type OTHER

Manual dexterity

Intervention Type DEVICE

Control of manual dexterity will be assessed by a force sensor (Power Grip Manipulandum, PGM)

Oculomotor movements

Intervention Type DEVICE

Oculomotor movements during behavioral task will be recorded using a video-oculography device

TMS coupled to EMG recording

Intervention Type DEVICE

The involvement of cortical inhibition in this volitional inhibition task will be studied by neuronavigation guided TMS coupled to EMG recording

Psychopathological evaluations

Intervention Type OTHER

Neuropsychological evaluations

Intervention Type OTHER

Controls

-25 age and gender-matched healthy controls

Group Type OTHER

Manual dexterity

Intervention Type DEVICE

Control of manual dexterity will be assessed by a force sensor (Power Grip Manipulandum, PGM)

Oculomotor movements

Intervention Type DEVICE

Oculomotor movements during behavioral task will be recorded using a video-oculography device

TMS coupled to EMG recording

Intervention Type DEVICE

The involvement of cortical inhibition in this volitional inhibition task will be studied by neuronavigation guided TMS coupled to EMG recording

Psychopathological evaluations

Intervention Type OTHER

Neuropsychological evaluations

Intervention Type OTHER

Interventions

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Manual dexterity

Control of manual dexterity will be assessed by a force sensor (Power Grip Manipulandum, PGM)

Intervention Type DEVICE

Oculomotor movements

Oculomotor movements during behavioral task will be recorded using a video-oculography device

Intervention Type DEVICE

TMS coupled to EMG recording

The involvement of cortical inhibition in this volitional inhibition task will be studied by neuronavigation guided TMS coupled to EMG recording

Intervention Type DEVICE

Psychopathological evaluations

Intervention Type OTHER

Neuropsychological evaluations

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* All groups:

1. 18\>yrs\<50
2. Medical visit completed
3. Visual acuity (9/10 for each eye or corrected)
4. Provided written informed consent
* Group of patient suffering from schizophrenia:

4\. DSM-IV-TR diagnostic criteria for schizophrenia 5. Treatment: stable atypical anti-psychotic medication for \>3 months prior to the study
* Group of UHR patient:

6\. 18\>yrs\<30 7. Fulfill at risk criteria of CAARMS diagnostic tool

Exclusion Criteria

• All groups:

1. IQ\<70,
2. Contraindications for TMS protocol: no previous history of neurosurgery or seizures or 1st degree relative with history of seizures, heart disease, drug abuse or addiction in the last 12 months, medications that lower seizure threshold including clozapine, bupropion, méthadone or theophylline.
3. Metallic implant in head (except dental fillings)
4. Pacemaker, or other electronic implanted devices
5. Central neurological disease: parkinsonism, x
6. Severe heart attack
7. Instable clinical state (e.g. stroke)
8. Previous history of drug abuse lasting more than 5 years or during the last year
9. Life event with a moderate to severe impact
10. Caffeine intake in the last two hours preceding visuomotor assessment

• Groups of Siblings and Healthy controls:
11. No previous history of psychiatric disease, psychotic spectrum disorder (according to DIGS 3.0)
12. No previous history of antipsychotic medication (entire life)

• Groups of UHR patient:
13. Chlorpromazine dose \>100mg over more than 12 weeks
14. No previous history of autism spectrum disorder, bipolar disorder or diagnozed schizophrenia (according to DSM-IV-TR criteria), isolated anxiety disorders (e.g. social phobia, agoraphobia)
Minimum Eligible Age

18 Years

Maximum Eligible Age

50 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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University of Paris 5 - Rene Descartes

OTHER

Sponsor Role collaborator

Institut National de la Santé Et de la Recherche Médicale, France

OTHER_GOV

Sponsor Role collaborator

Centre Hospitalier St Anne

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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Isabelle Amado, Dr

Role: STUDY_DIRECTOR

CHSA

Pavel Lindberg, PhD

Role: STUDY_DIRECTOR

Institut National de la Santé Et de la Recherche Médicale, France

Locations

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Centre de Recherche Clinique (CRC) - CHSA

Paris, , France

Site Status RECRUITING

Service Hospitalo-Universitaire (SHU) - CHSA

Paris, , France

Site Status NOT_YET_RECRUITING

Countries

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France

Central Contacts

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Isabelle Amado, Dr

Role: CONTACT

Phone: 00 33 1 45 65 81 79

Email: [email protected]

Marie GODARD

Role: CONTACT

Phone: 00 33 1 45 65 77 28

Email: [email protected]

Facility Contacts

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Macarena CUENCA

Role: primary

Cecile Bergot

Role: backup

Isabelle Amado

Role: primary

Marie GODARD

Role: backup

References

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Teremetz M, Amado I, Bendjemaa N, Krebs MO, Lindberg PG, Maier MA. Deficient grip force control in schizophrenia: behavioral and modeling evidence for altered motor inhibition and motor noise. PLoS One. 2014 Nov 4;9(11):e111853. doi: 10.1371/journal.pone.0111853. eCollection 2014.

Reference Type BACKGROUND
PMID: 25369465 (View on PubMed)

Amado I, Landgraf S, Bourdel MC, Leonardi S, Krebs MO. Predictive saccades are impaired in biological nonpsychotic siblings of schizophrenia patients. J Psychiatry Neurosci. 2008 Jan;33(1):17-22.

Reference Type BACKGROUND
PMID: 18197268 (View on PubMed)

Other Identifiers

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D16-P01

Identifier Type: -

Identifier Source: org_study_id