Potential Impact of Cigarette Smoking on Platelet Reactivity in Patients on Dual Antiplatelet Therapy With P2Y12 Inhibitors

NCT ID: NCT02026713

Last Updated: 2015-06-09

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

PHASE4

Total Enrollment

150 participants

Study Classification

INTERVENTIONAL

Study Start Date

2014-07-31

Study Completion Date

2015-07-31

Brief Summary

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All consecutive patients undergone PTCA in the period between July 2013 and December 2013 are eligible to be enrolled. Study population are divided in :

Group 1: smokers on dual antiplatelet therapy with ASA (100 mg once a day) and Prasugrel (10 mg die); Group 2: smokers on dual antiplatelet therapy with ASA(100 mg once a day) and Ticagrelor (90 mg twice a day); Control group: smokers on dual antiplatelet therapy with ASA(100 mg once a day) and Clopidogrel (75 mg once a day).

Platelet function is evaluated using a validated method: the VerifyNow System (Accumetrics Inc., San Diego, CA), which is a point-of-care turbidimetry-based optical detection system that measures platelet-induced aggregation. These value are measured Platelet function, measured with the VerifyNow P2Y12. All patients on chronic dual antiplatelet therapy (\>1 month) quit smoking for a 2 weeks period. Therefore PRU values are obtained at baseline (at the enrollment time) and after 2 weeks after smoking cessation. Platelet reactivity are expressed in P2Y12 reaction units (PRU). PRU values \>240 are suggestive of high platelet reactivity.

Primary outcomes: P2Y12 reaction units (PRU) at baseline and two weeks after quitting smoking.

Detailed Description

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All consecutive patients undergone PTCA in the period between July 2013 and December 2013 are eligible to be enrolled. Study population are divided in :

Group 1: smokers on dual antiplatelet therapy with ASA (100 mg once a day) and Prasugrel (10 mg die); Group 2: smokers on dual antiplatelet therapy with ASA(100 mg once a day) and Ticagrelor (90 mg twice a day); Control group: smokers on dual antiplatelet therapy with ASA(100 mg once a day) and Clopidogrel (75 mg once a day).

Platelet function is evaluated using a validated method: the VerifyNow System (Accumetrics Inc., San Diego, CA), which is a point-of-care turbidimetry-based optical detection system that measures platelet-induced aggregation. These value are measured Platelet function, measured with the VerifyNow P2Y12. All patients on chronic dual antiplatelet therapy (\>1 month) quit smoking for a 2 weeks period. Therefore PRU values are obtained at baseline (at the enrollment time) and after 2 weeks after smoking cessation. Platelet reactivity are expressed in P2Y12 reaction units (PRU). PRU values \>240 are suggestive of high platelet reactivity.

Primary outcomes: P2Y12 reaction units (PRU) at baseline and two weeks after quitting smoking.

Conditions

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Coronary Artery Disease

Study Design

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Allocation Method

NON_RANDOMIZED

Intervention Model

PARALLEL

Blinding Strategy

NONE

Study Groups

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smokers on dual antiplatelet therapy with ASA and Prasugrel or

All patients quit smoking for a 2 weeks period

Group Type EXPERIMENTAL

quit smoking for a 2 weeks period

Intervention Type OTHER

evaluate the platelet reactivity after 15 days by quit smoking

smokers on dual antiplatelet therapy with ASA and ticagrelor

All patients on chronic dual antiplatelet therapy (\>1 month) quit smoking for a 2 weeks period

Group Type EXPERIMENTAL

quit smoking for a 2 weeks period

Intervention Type OTHER

evaluate the platelet reactivity after 15 days by quit smoking

smokers on dual antiplatelet therapy with ASA and Clopidogrel

All patients on chronic dual antiplatelet therapy (\>1 month) quit smoking for a 2 weeks period

Group Type ACTIVE_COMPARATOR

quit smoking for a 2 weeks period

Intervention Type OTHER

evaluate the platelet reactivity after 15 days by quit smoking

Interventions

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quit smoking for a 2 weeks period

evaluate the platelet reactivity after 15 days by quit smoking

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* All consecutive patients undergone PTCA in our institution in the period between July 2013 and December 2013 will be eligible to be enrolled.
* smokers will be all patients smoking 15±10 cigarettes daily.
* patients on dual antiplatelet therapy with ASA and P2Y12 inhibitors.
* recent (\<12 months) percutaneous coronary interventions
* angiographically-proven coronary artery disease

Exclusion Criteria

\- people unable to understand and willing to sign the informed consent form
Minimum Eligible Age

18 Years

Maximum Eligible Age

90 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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University of Roma La Sapienza

OTHER

Sponsor Role lead

Responsible Party

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Polacco Marina

MD

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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marina MD polacco, medicine

Role: PRINCIPAL_INVESTIGATOR

University of Roma La Sapienza

Locations

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Sapienza University of Rome

Rome, Roma, Italy

Site Status RECRUITING

Countries

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Italy

Central Contacts

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Marina Polacco

Role: CONTACT

3333347960

Facility Contacts

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Marina Polacco

Role: primary

3333347960

References

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Patti G, Polacco M, Taurino E, Gaudio C, Greco C. Effects of cigarette smoking on platelet reactivity during P2Y12 inhibition in patients with myocardial infarction undergoing drug-eluting stent implantation: results from the prospective cigarette smoking on platelet reactivity (COPTER) study. J Thromb Thrombolysis. 2016 May;41(4):648-53. doi: 10.1007/s11239-016-1341-8.

Reference Type DERIVED
PMID: 26849144 (View on PubMed)

Other Identifiers

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WD-131213

Identifier Type: -

Identifier Source: org_study_id

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