Pharmacodynamic Effects of Ranolazine Versus Amlodipine on Platelet Reactivity

NCT ID: NCT01490255

Last Updated: 2014-03-26

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

PHASE4

Total Enrollment

100 participants

Study Classification

INTERVENTIONAL

Study Start Date

2012-01-31

Study Completion Date

2014-06-30

Brief Summary

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No previous study has assessed the potential of ranolazine to interfere with levels of platelet reactivity in patients with coronary artery disease who are treated with dual antiplatelet therapy.

Aim of this study is to compare the pharmacodynamic effects of maintenance doses of ranolazine versus amlodipine on platelet reactivity in patients with coronary artery disease who are treated with dual antiplatelet therapy.

Detailed Description

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Patients with coronary artery disease (CAD) are often treated with dual antiplatelet therapy (DAT), including aspirin and clopidogrel, to prevent from recurrent atherothrombotic events.

Levels of platelet reactivity in patients on DAT can be influenced by concomitant treatment with medications that inhibit the CYP3A4 system involved in the activation of clopidogrel, such as calcium channel blockers, potentially interfering with its clinical benefits. Importantly, calcium channel blockers, such as amlodipine, are commonly used for relief of ischemic symptoms in patients with CAD.

Ranolazine is a novel antianginal drug that reduces intracellular sodium and calcium accumulation and constitutes a pharmacologic alternative to calcium channel blockade.

However, no previous study has assessed the potential of ranolazine to interfere with levels of platelet reactivity in CAD patients on DAT.

The primary objective of this study is to compare the pharmacodynamic effects of maintenance doses of ranolazine versus amlodipine on platelet reactivity in patients with CAD on DAT.

Conditions

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Coronary Artery Disease

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

CROSSOVER

Primary Study Purpose

DIAGNOSTIC

Blinding Strategy

SINGLE

Participants

Study Groups

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Ranolazine

Patients will receive ranolazine (750 mg bid) for 15 days

Group Type ACTIVE_COMPARATOR

Ranolazine

Intervention Type DRUG

os, 750 mg, twice per day, for 15 days

Amlodipine

Patients will receive amlodipine (10 mg once daily) for 15 days

Group Type ACTIVE_COMPARATOR

Amlodipine

Intervention Type DRUG

os, 10 mg, once daily, 15 days

Interventions

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Ranolazine

os, 750 mg, twice per day, for 15 days

Intervention Type DRUG

Amlodipine

os, 10 mg, once daily, 15 days

Intervention Type DRUG

Other Intervention Names

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Ranexa®, Gilead, USA Norvasc®, Pfizer, USA

Eligibility Criteria

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Inclusion Criteria

* Angiographically-proven coronary artery disease
* Class I indication to dual antiplatelet therapy because of recent (\<12 months) percutaneous coronary intervention and/or recent acute coronary syndrome (\<12 months)
* Stable clinical conditions
* Able to understand and willing to sign the informed consent form

Exclusion Criteria

* Use of other drug interfering with CYP activity such as proton pump inhibitors
* Women of child bearing potential patients must demonstrate a negative pregnancy test performed within 24 hours before
Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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University of Roma La Sapienza

OTHER

Sponsor Role lead

Responsible Party

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Francesco Pelliccia

Assistant Professor

Responsibility Role PRINCIPAL_INVESTIGATOR

Principal Investigators

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Francesco Pelliccia, MD

Role: PRINCIPAL_INVESTIGATOR

University La Sapienza, Rome, IT

Locations

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San Raffaele Pisana

Rome, , Italy

Site Status RECRUITING

University La Sapienza

Rome, , Italy

Site Status RECRUITING

Countries

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Italy

Central Contacts

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Francesco Pelliccia, MD

Role: CONTACT

+393483392006

Facility Contacts

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Giuseppe Marazzi, MD

Role: primary

+39 335 8381320

Francesco Pelliccia, MD

Role: primary

+393483392006

Other Identifiers

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654/2011/D

Identifier Type: -

Identifier Source: org_study_id

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