Trial Outcomes & Findings for Effects of Incretins on Human Platelet Function (NCT NCT01408862)

NCT ID: NCT01408862

Last Updated: 2014-09-23

Results Overview

1.1 Measurement of GLP-1 and GIP receptors at the platelet RNA level by Real Time-PCR 1.2 GLP-1 and GIP receptors detection at the protein level: 1.2.1 Expression on platelet membrane by flow cytometry. 1.2.2 Detection in total platelet proteins by western-blot. Data of flow cytometry are provided below

Recruitment status

COMPLETED

Target enrollment

20 participants

Primary outcome timeframe

Platelets drawn from a volunteer were evaluated 1 time (in 1-2 days)

Results posted on

2014-09-23

Participant Flow

Platelets from 20 healthy human subjects who were not taken any medication in the previous 10 days were studied during 18 month in our institution.

Participant milestones

Participant milestones
Measure
Controls
20 healthy volunteers were asked to donor a 10-80 ml blood through a venous puncture venous puncture: Blood for in vitro studies were drawn
Overall Study
STARTED
20
Overall Study
COMPLETED
20
Overall Study
NOT COMPLETED
0

Reasons for withdrawal

Withdrawal data not reported

Baseline Characteristics

Effects of Incretins on Human Platelet Function

Baseline characteristics by cohort

Baseline characteristics by cohort
Measure
Controls
n=20 Participants
Platelets from healthy human subjects who were not taken any medication in the previous 10 days were studied. Blood samples (30 ml) were drawn with i) ACD-C (9:1, v/v) (7 mmol/L citric acid, 93 mmol/L citrate, 139 mmol/L dextrose, pH 6.4) for gene expression and western blot studies; ii) with 1% EDTA for flow cytometry and iii) with 3.8% sodium citrate for functional studies. Blood samples were centrifuged at 150 g for 10 min to obtain platelet-rich plasma (PRP).
Age, Continuous
42 years
STANDARD_DEVIATION 10 • n=5 Participants
Age, Categorical
<=18 years
0 Participants
n=5 Participants
Age, Categorical
Between 18 and 65 years
20 Participants
n=5 Participants
Age, Categorical
>=65 years
0 Participants
n=5 Participants
Sex: Female, Male
Female
10 Participants
n=5 Participants
Sex: Female, Male
Male
10 Participants
n=5 Participants
Region of Enrollment
Argentina
20 participants
n=5 Participants

PRIMARY outcome

Timeframe: Platelets drawn from a volunteer were evaluated 1 time (in 1-2 days)

1.1 Measurement of GLP-1 and GIP receptors at the platelet RNA level by Real Time-PCR 1.2 GLP-1 and GIP receptors detection at the protein level: 1.2.1 Expression on platelet membrane by flow cytometry. 1.2.2 Detection in total platelet proteins by western-blot. Data of flow cytometry are provided below

Outcome measures

Outcome measures
Measure
Controls
n=20 Participants
Flow cytometry studies In order to test the presence of GLP1 and GIP receptors on normal platelet membrane, indirect immunodetection was carried out in platelet samples from 20 normal donors.
Expression of Glucagon Like Peptide -1 (GLP-1) and Gastric Inhibitory Peptide (GIP) Receptors in Normal Human Platelets
GIPR
4.42 percentage of positive cells
Standard Deviation 1.70
Expression of Glucagon Like Peptide -1 (GLP-1) and Gastric Inhibitory Peptide (GIP) Receptors in Normal Human Platelets
GLP1R
3.73 percentage of positive cells
Standard Deviation 2.43

PRIMARY outcome

Timeframe: Platelets drawn from a volunteer will be evaluated 1 time (in 1-2 days)

Population: We assume that with 10 samples per condition divided in 5 categories (1 control plus 4 concentrations of GLP1 and GIP agonists) for a standardized difference of 0.5 between groups and a p=0.05, the statistical power would be higher than 90%. We used a similar design to test the effect of GLP1 and GIP agonists on platelet aggregants.

1 Direct effect of GLP-1-(7-36)NH2 and its metabolite (GLP-1-(9-36)NH2) and GIP agonist on platelet aggregation, with and without preincubation at different glucose concentrations. 2\. GLP-1-(7-36)NH2 and its metabolite (GLP-1-(9-36)NH2) and GIP agonist modulation (with and without 300 mg/dL glucose added to the media) of platelet activation and aggregation induced by classical platelet agonists such as ADP, collagen and bovine von Willebrand factor. Data points from various conditions were combined (averaged),

Outcome measures

Outcome measures
Measure
Controls
n=20 Participants
Flow cytometry studies In order to test the presence of GLP1 and GIP receptors on normal platelet membrane, indirect immunodetection was carried out in platelet samples from 20 normal donors.
Changes in Basal or Aggregant-induced Platelet Activation (With and Without Glucose Added to the Media, 200 mg/dl, 400 mg/dL) by GLP-1-(7-36)NH2 and Its Metabolite (GLP-1-(9-36)NH2)and a GIP Agonist at Different Concentrations.
GIP
0 % of Inhibition
Standard Deviation 8
Changes in Basal or Aggregant-induced Platelet Activation (With and Without Glucose Added to the Media, 200 mg/dl, 400 mg/dL) by GLP-1-(7-36)NH2 and Its Metabolite (GLP-1-(9-36)NH2)and a GIP Agonist at Different Concentrations.
GLP1
0 % of Inhibition
Standard Deviation 10

Adverse Events

Controls

Serious events: 0 serious events
Other events: 0 other events
Deaths: 0 deaths

Serious adverse events

Adverse event data not reported

Other adverse events

Adverse event data not reported

Additional Information

Dr. Carlos Jose Pirola

Institute of Medical Research (UBA-CONICET)

Phone: 541145148701

Results disclosure agreements

  • Principal investigator is a sponsor employee
  • Publication restrictions are in place

Restriction type: LTE60