Erlotinib for Treatment of Psoriasis

NCT ID: NCT01006096

Last Updated: 2014-11-10

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

WITHDRAWN

Clinical Phase

PHASE2

Study Classification

INTERVENTIONAL

Brief Summary

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The purpose of this study is to determine whether erlotinib is effective in the treatment of psoriasis.

Detailed Description

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Psoriasis vulgaris is a disease that affects 25 million people in North America and Europe. It often presents in late adolescence and usually persists for life. Current therapies target specific immune molecules that are implicated in the cause of this disease. For example, biologic agents that are used in severe psoriasis are aimed at inflammatory mediators. These therapies have been proven to be effective but also have their limitations.

Conditions

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Psoriasis

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

TREATMENT

Blinding Strategy

QUADRUPLE

Participants Caregivers Investigators Outcome Assessors

Study Groups

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Erlotinib

Group Type EXPERIMENTAL

erlotinib

Intervention Type DRUG

100mg tablet, once daily for 16 weeks

Placebo tablets

Group Type PLACEBO_COMPARATOR

placebo tablet

Intervention Type OTHER

placebo tablet (lactose), once daily for 16 weeks

Interventions

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erlotinib

100mg tablet, once daily for 16 weeks

Intervention Type DRUG

placebo tablet

placebo tablet (lactose), once daily for 16 weeks

Intervention Type OTHER

Other Intervention Names

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Tarceva

Eligibility Criteria

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Inclusion Criteria

* Clinical diagnosis of moderate to severe psoriasis
* Must have documented moderate to severe psoriasis by the Physician's Global Assessment (PGA) and the Psoriasis Area Severity Index (PASI)
* Must be able to swallow tablets
* Must be able to provide written informed consent
* Subjects with reproductive potential (menopausal for less than 1 year and not surgically sterilized) must practice effective contraceptive measures throughout the study and thirty days after discontinuation of study drug. Women of childbearing potential must provide negative pregnancy test (serum or urine) within 14 days prior to randomization.

Exclusion Criteria

* Use of concurrent agents/therapies for psoriasis
* Bilirubin \> 3 X ≥ ULN or moderate to severe hepatic impairment
* Pregnant or breast-feeding females
* Subjects currently receiving other anticancer treatments
* Subjects currently receiving other biologic treatments
* Subjects currently receiving blood thinners (warfarin or heparin)
* Subjects who currently smoke
* Subjects with other skin disease which in the opinion of the investigator, would inhibit the ability to use the PGA and PASI evaluation methods
Minimum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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OSI Pharmaceuticals

INDUSTRY

Sponsor Role collaborator

Northwestern University

OTHER

Sponsor Role lead

Responsible Party

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Northwestern University

Principal Investigators

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Anne E Laumann, MBChB, MRCP (UK)

Role: PRINCIPAL_INVESTIGATOR

Northwestern University

Locations

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Northwestern University Feinberg School of Medicine Department of Dermatology

Chicago, Illinois, United States

Site Status

Countries

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United States

References

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Lowes MA, Bowcock AM, Krueger JG. Pathogenesis and therapy of psoriasis. Nature. 2007 Feb 22;445(7130):866-73. doi: 10.1038/nature05663.

Reference Type BACKGROUND
PMID: 17314973 (View on PubMed)

Shepherd FA, Rodrigues Pereira J, Ciuleanu T, Tan EH, Hirsh V, Thongprasert S, Campos D, Maoleekoonpiroj S, Smylie M, Martins R, van Kooten M, Dediu M, Findlay B, Tu D, Johnston D, Bezjak A, Clark G, Santabarbara P, Seymour L; National Cancer Institute of Canada Clinical Trials Group. Erlotinib in previously treated non-small-cell lung cancer. N Engl J Med. 2005 Jul 14;353(2):123-32. doi: 10.1056/NEJMoa050753.

Reference Type BACKGROUND
PMID: 16014882 (View on PubMed)

Lacouture ME. Mechanisms of cutaneous toxicities to EGFR inhibitors. Nat Rev Cancer. 2006 Oct;6(10):803-12. doi: 10.1038/nrc1970.

Reference Type BACKGROUND
PMID: 16990857 (View on PubMed)

Wierzbicka E, Tourani JM, Guillet G. Improvement of psoriasis and cutaneous side-effects during tyrosine kinase inhibitor therapy for renal metastatic adenocarcinoma. A role for epidermal growth factor receptor (EGFR) inhibitors in psoriasis? Br J Dermatol. 2006 Jul;155(1):213-4. doi: 10.1111/j.1365-2133.2006.07299.x. No abstract available.

Reference Type BACKGROUND
PMID: 16792781 (View on PubMed)

Ben-Bassat H, Vardi DV, Gazit A, Klaus SN, Chaouat M, Hartzstark Z, Levitzki A. Tyrphostins suppress the growth of psoriatic keratinocytes. Exp Dermatol. 1995 Apr;4(2):82-8. doi: 10.1111/j.1600-0625.1995.tb00227.x.

Reference Type BACKGROUND
PMID: 7640880 (View on PubMed)

Powell TJ, Ben-Bassat H, Klein BY, Chen H, Shenoy N, McCollough J, Narog B, Gazit A, Harzstark Z, Chaouat M, Levitzki R, Tang C, McMahon J, Shawver L, Levitzki A. Growth inhibition of psoriatic keratinocytes by quinazoline tyrosine kinase inhibitors. Br J Dermatol. 1999 Nov;141(5):802-10. doi: 10.1046/j.1365-2133.1999.03152.x.

Reference Type BACKGROUND
PMID: 10583160 (View on PubMed)

Ben-Bassat H, Klein BY. Inhibitors of tyrosine kinases in the treatment of psoriasis. Curr Pharm Des. 2000 Jun;6(9):933-42. doi: 10.2174/1381612003400182.

Reference Type BACKGROUND
PMID: 10828317 (View on PubMed)

Ben-Bassat H, Levitzki A. Inhibitors of tyrosine kinases in the treatment of psoriasis. Isr Med Assoc J. 2000 Jul;2 Suppl:69-73. No abstract available.

Reference Type BACKGROUND
PMID: 10909421 (View on PubMed)

Halin C, Fahrngruber H, Meingassner JG, Bold G, Littlewood-Evans A, Stuetz A, Detmar M. Inhibition of chronic and acute skin inflammation by treatment with a vascular endothelial growth factor receptor tyrosine kinase inhibitor. Am J Pathol. 2008 Jul;173(1):265-77. doi: 10.2353/ajpath.2008.071074. Epub 2008 Jun 5.

Reference Type BACKGROUND
PMID: 18535184 (View on PubMed)

Forsberg S, Ostman A, Rollman O. Regeneration of human epidermis on acellular dermis is impeded by small-molecule inhibitors of EGF receptor tyrosine kinase. Arch Dermatol Res. 2008 Oct;300(9):505-16. doi: 10.1007/s00403-008-0853-2. Epub 2008 Apr 30.

Reference Type BACKGROUND
PMID: 18446355 (View on PubMed)

Neyns B, Meert V, Vandenbroucke F. Cetuximab treatment in a patient with metastatic colorectal cancer and psoriasis. Curr Oncol. 2008 Aug;15(4):196-7. doi: 10.3747/co.v15i4.228.

Reference Type BACKGROUND
PMID: 18769609 (View on PubMed)

Trivin F, Boucher E, Raoul JL. Complete sustained regression of extensive psoriasis with cetuximab combination chemotherapy. Acta Oncol. 2004;43(6):592-3. doi: 10.1080/02841860410020211. No abstract available.

Reference Type BACKGROUND
PMID: 15370619 (View on PubMed)

Other Identifiers

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MEL-041509

Identifier Type: -

Identifier Source: org_study_id