Effects of Obstructive Sleep Apnea and/or Cigarette Smoking on Endothelial Function

NCT ID: NCT00957463

Last Updated: 2014-12-30

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Total Enrollment

150 participants

Study Classification

OBSERVATIONAL

Study Start Date

2008-06-30

Brief Summary

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The hypothesis of this study is that either obstructive sleep apnea (OSA) or cigarette smoking (CS) exposure would produce oxidative stress and inflammation leading to endothelial injury, and the combined exposure would be additive or synergistic.

Detailed Description

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Conditions

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Obstructive Sleep Apnea Smoking

Keywords

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endothelial function

Study Design

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Observational Model Type

CASE_CONTROL

Study Time Perspective

CROSS_SECTIONAL

Study Groups

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smoker, with high apnea-hypopnea index

subjects who smoke either in the past or currently, with confirmed moderate-severe OSA

No interventions assigned to this group

smoker, mild OSA or without OSA

subjects who smoke either in the past or currently, with mild OSA or without OSA

No interventions assigned to this group

nonsmoker, with high apnea-hypopnea index

subjects who never smoke, with confirmed moderate-severe OSA

No interventions assigned to this group

nonsmoker, with mild OSA or without OSA

subjects who never smoke, with mild OSA or without OSA

No interventions assigned to this group

Eligibility Criteria

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Inclusion Criteria

* No acute illness
* No hospitalization in past 4 weeks
* Absence of medical comorbidity such as hypertension, ischemic heart disease, diabetes and hyperlipidemia, renal impairment, vasculitis or Raynaud's syndrome, peripheral vascular disease especially Buerger's disease, aortic disease (aortic aneurysm or dilatation), cancer, thrombocytopenia, coagulation disorder or other chronic medical illness

Exclusion Criteria

* Mentally incapacitated
Minimum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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The University of Hong Kong

OTHER

Sponsor Role lead

Responsible Party

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The University of Hong Kong

Principal Investigators

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Mary SM Ip, MD

Role: PRINCIPAL_INVESTIGATOR

The University of Hong Kong

Locations

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Queen Mary Hospital

Hong Kong, , Hong Kong

Site Status

Countries

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Hong Kong

References

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Barnoya J, Glantz SA. Cardiovascular effects of secondhand smoke: nearly as large as smoking. Circulation. 2005 May 24;111(20):2684-98. doi: 10.1161/CIRCULATIONAHA.104.492215.

Reference Type BACKGROUND
PMID: 15911719 (View on PubMed)

Kashyap R, Hock LM, Bowman TJ. Higher prevalence of smoking in patients diagnosed as having obstructive sleep apnea. Sleep Breath. 2001 Dec;5(4):167-72. doi: 10.1007/s11325-001-0167-5.

Reference Type BACKGROUND
PMID: 11868156 (View on PubMed)

Lavie L, Lavie P. Smoking interacts with sleep apnea to increase cardiovascular risk. Sleep Med. 2008 Mar;9(3):247-53. doi: 10.1016/j.sleep.2007.03.018. Epub 2007 May 21.

Reference Type BACKGROUND
PMID: 17513169 (View on PubMed)

El Solh AA, Akinnusi ME, Baddoura FH, Mankowski CR. Endothelial cell apoptosis in obstructive sleep apnea: a link to endothelial dysfunction. Am J Respir Crit Care Med. 2007 Jun 1;175(11):1186-91. doi: 10.1164/rccm.200611-1598OC. Epub 2007 Feb 1.

Reference Type BACKGROUND
PMID: 17272785 (View on PubMed)

Bernhard D, Wang XL. Smoking, oxidative stress and cardiovascular diseases--do anti-oxidative therapies fail? Curr Med Chem. 2007;14(16):1703-12. doi: 10.2174/092986707781058959.

Reference Type BACKGROUND
PMID: 17627508 (View on PubMed)

Itzhaki S, Lavie L, Pillar G, Tal G, Lavie P. Endothelial dysfunction in obstructive sleep apnea measured by peripheral arterial tone response in the finger to reactive hyperemia. Sleep. 2005 May;28(5):594-600. doi: 10.1093/sleep/28.5.594.

Reference Type BACKGROUND
PMID: 16171272 (View on PubMed)

Other Identifiers

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UW 08-023

Identifier Type: -

Identifier Source: org_study_id