Irbesartan and Adhesion Molecules in AF

NCT ID: NCT00613496

Last Updated: 2009-05-29

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

PHASE4

Total Enrollment

60 participants

Study Classification

INTERVENTIONAL

Study Start Date

2009-05-31

Study Completion Date

2010-05-31

Brief Summary

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Experimental data suggest that angiotensin II-antagonists reduce the atrial expression of prothrombotic adhesion molecules and oxidative stress parameters. The present study is designed to investigate the effects on angiotensin II-antagonist irbesartan to reduce the amounts of circulating oxidative stress markers and adhesion molecules in patients with persistent atrial fibrillation.

Detailed Description

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Primary Objective:

The aim of the study is to assess that blocking the angiotensin II type 1 receptor reduces systemic levels of oxidative stress markers and adhesion molecules by more than 25% compared to placebo in patients with persistent/permanent atrial fibrillation.

Primary Target Parameter:

The primary target parameter is defined as reduction of systemic levels of oxidative stress markers and adhesion molecules (hsCRP, ICAM, VCAM, MCP-1, vWF, TGFβ1, TNF-α, Interleukin-6, 8isoProstaglandinF2α)

Secondary Target Parameter:

The secondary Target Parameters are defined as number of cerebrovascular events, number of intermediate medical visits for cardiovascular reasons without hospitalisation, number of hospitalisations for cardiovascular reasons and GFR.

Conditions

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Persistent Atrial Fibrillation

Keywords

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Atrial Fibrillation irbesartan AT-II-antagonist Adhesion Molecules oxidative stress markers hsCRP ICAM VCAM MCP-1 vWF TGFβ1 TNF-α Interleukin-6 8isoProstaglandinF2α

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

CROSSOVER

Primary Study Purpose

TREATMENT

Blinding Strategy

QUADRUPLE

Participants Caregivers Investigators Outcome Assessors

Study Groups

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2

Placebo treatment in each patient during the study (9 weeks) using an intraindividual cross-over design

Group Type PLACEBO_COMPARATOR

placebo

Intervention Type DRUG

Placebo-tablet, 1 in the morning for 7 days, 2 tablets (1 in the morning and 1 in the evening) after day 8.

1

Irbesartan treatment in each patient during the study (9 weeks) using an intraindividual cross-over design

Group Type ACTIVE_COMPARATOR

irbesartan

Intervention Type DRUG

Irbesartan-tablet (150 mg) 1 in the morning for 7 days, 2 tablets (1 in the morning and 1 in the evening) after day 8 if no contraindication for up titration (investigator will decide on the basis of creatinin, urea and potassium after taking a blood sample) for 9 weeks.

Interventions

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irbesartan

Irbesartan-tablet (150 mg) 1 in the morning for 7 days, 2 tablets (1 in the morning and 1 in the evening) after day 8 if no contraindication for up titration (investigator will decide on the basis of creatinin, urea and potassium after taking a blood sample) for 9 weeks.

Intervention Type DRUG

placebo

Placebo-tablet, 1 in the morning for 7 days, 2 tablets (1 in the morning and 1 in the evening) after day 8.

Intervention Type DRUG

Other Intervention Names

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Avapro Aprovel Carvea

Eligibility Criteria

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Inclusion Criteria

* Patients with persistent/permanent AF (\>2 months)
* CHADS2 Score ≥2
* Age ≥18
* Patient informed orally and in writing
* Written informed consent of the patient
* Patients who are anticipated to show sufficient compliance in following the study protocol
* Patients must agree to undergo the 148 days clinical follow-up
* Patients who are mentally and linguistically able to understand the aim of the study and the associated risks and benefits of the treatment. The patients, by providing informed consent, agree to this treatment as stated in the patient informed consent document.

Exclusion Criteria

* Strong clinical evidence that prevents the temporary pause of therapy with AT II antagonists
* Symptomatic bradycardia
* Implanted pacemaker or implanted cardioverter/defibrillator with any antitachycardia algorithm in use
* Cardiac surgery or cardiac catheter ablation within the last 3 months prior to randomisation
* Typical angina pectoris symptoms at rest or during exercise
* Known coronary artery disease with indication for intervention
* Symptomatic peripheral vascular disease
* Left ventricular ejection fraction \<35%
* Myocardial infarction within 6 months prior to randomisation
* Diastolic blood pressure \>110mmHg at rest
* Symptomatic arterial hypotension
* Known renal artery stenosis
* Serum creatinin \>1.8mval/l
* Chronic inflammatory disease
* Acute inflammatory disease (CRP \>20mg/L)
* Relevant hepatic or pulmonary disorders
* Hyperthyreosis manifested clinically and in laboratory
* Known drug intolerance for AT II inhibitors
* Females who are pregnant or breast feeding
* Females of childbearing potential who are not using a scientifically accepted method of contraception
* Participation in a clinical trial within the last 30 days prior to randomisation
* Drug addiction or chronic alcohol abuse
* Cancer or other disease, which inevitably leads to death
* Legal incapacity, or other circumstances which would prevent the patient from understanding the aim, nature or extent of the clinical study, evidence of an uncooperative attitude
Minimum Eligible Age

18 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Sanofi

INDUSTRY

Sponsor Role collaborator

University of Magdeburg

OTHER

Sponsor Role lead

Responsible Party

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University Hospital Magdeburg

Principal Investigators

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Andreas Goette, MD

Role: PRINCIPAL_INVESTIGATOR

University Hospital Magdeburg; Div of Cardiology

Locations

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University Hospital Magdeburg; Div. of Cardiology

Magdeburg, , Germany

Site Status RECRUITING

Countries

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Germany

Central Contacts

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Andreas Goette, MD

Role: CONTACT

Phone: 00493916713225

Email: [email protected]

Facility Contacts

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Veronika Raetzel

Role: primary

Other Identifiers

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EUDRACTN: 2007-003262-17

Identifier Type: -

Identifier Source: secondary_id

AG-1-2007

Identifier Type: -

Identifier Source: org_study_id