Treatment of Patients With Age-Related Macular Degeneration With Anecortave Acetate

NCT ID: NCT00349739

Last Updated: 2010-01-15

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

NO_LONGER_AVAILABLE

Study Classification

EXPANDED_ACCESS

Study Start Date

2003-01-31

Study Completion Date

2008-08-31

Brief Summary

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The purpose of this study is to evaluate whether anecortave acetate can slow or stop the progression of age-related macular degeneration in patients who do not qualify for other studies and have no other treatment options.

Detailed Description

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Choroidal neovascularization (CNV), a complication of age-related macular degeneration (AMD), is the most common cause of profound visual loss in the United States. Laser photocoagulation has been shown to retard visual loss, however only about 20% of patients with CNV are eligible for laser treatment.

Conventional laser photocoagulation of CNV requires laser intensities adequate to coagulate proteins in the target tissue. Due to the proximity of CNV to the center of vision (fovea), the intensity of laser irradiation necessary, and the heat conduction in the ocular tissues, collateral tissue damage often results which further compromises vision. Recently, a new class of compounds (angiostatic steroids) have been found to inhibit the formation of new blood vessels (i.e. neovascularization) in the eye. One new angiostatic steroid, anecortave acetate (AL-3789), may represent a breakthrough in the therapy of ocular neovascular diseases such as AMD and diabetic retinopathy. Anecortave acetate suppresses the formation of new blood vessels in a variety of models of neovascularization. Unlike some of the angiostatic steroids, anecortave acetate appears to be lacking in the pharmacological activities typical of the steroid family (i.e. glucocorticoid, anti-inflammatory, cardiovascular, neurologic, diuretic, etc.) Additionally, anecortave acetate has been shown to arrest lipopolysaccharide (LPS) and basic fibroblast growth factors (bFGH) induced corneal neovascularization, to attenuate oxygen-induced retinopathy and to inhibit the growth of a highly vascularized intraocular tumor.

The purpose of this study is to treat a small number of patients who would not normally qualify for the other anecortave acetate studies. These patients who are not eligible for the other anecortave acetate studies have no other treatment options and will likely experience a poor visual outcome as a result of their AMD. These patients are excluded from other studies because of "occult" neovascularization and visual acuity worse than 20/40. Sub-Tenon's injection of either 15 mg or 30 mg of anecortave acetate will be administered at the University of Iowa. The patients will be followed for a minimum of 24 months.

Conditions

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Macular Degeneration

Interventions

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Anecortave Acetate

Dosage: 0.5ml of 30mg/ml(15 mg) or 60mg/ml(30mg) Duration of action: 159-180 days

Intervention Type DRUG

Other Intervention Names

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Retaane

Eligibility Criteria

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Inclusion Criteria

* Choroidal neovascularization, mostly occult
* Visual acuity \> 20/40 in study eye

Exclusion Criteria

* Allergy to fluorescein dye
* Inability to stop warfarin prior to treatment
* Pregnancy
* Other serious ocular diseases or conditions
Minimum Eligible Age

55 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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University of Iowa

OTHER

Sponsor Role lead

Responsible Party

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University of Iowa

Principal Investigators

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Edwin M Stone, MD, PhD

Role: PRINCIPAL_INVESTIGATOR

University of Iowa

Stephen R. Russell, MD

Role: STUDY_DIRECTOR

University of Iowa

Locations

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University of Iowa Hospitals and Clinics, Ophthalmology

Iowa City, Iowa, United States

Site Status

Countries

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United States

Related Links

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Other Identifiers

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AL-3789

Identifier Type: -

Identifier Source: secondary_id

UIOWA200101096

Identifier Type: -

Identifier Source: org_study_id

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