Effect of Ethanol and Genetic Polymorphisms on Bupropion Metabolism

NCT ID: NCT00330434

Last Updated: 2020-11-10

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

WITHDRAWN

Clinical Phase

NA

Study Classification

INTERVENTIONAL

Study Start Date

2005-12-31

Study Completion Date

2008-04-30

Brief Summary

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The two purposes of this study are

1. to determine what effect the chronic and moderate/heavy drinking of alcoholic beverages has

1. on the blood level of bupropion and chlorzoxazone and their major breakdown products in the blood and
2. on the stimulant effect of bupropion and
2. to determine what effect a normal and common (25% frequency) genetic variation of a specific liver enzyme (that breaks down bupropion) has

1. on the blood levels of bupropion and its major breakdown products in the blood and
2. on the stimulant effect of bupropion.

Two groups of volunteers will be recruited for this study:

1. volunteers who drink moderate to heavy amounts of alcohol frequently and
2. volunteers who usually do not drink alcohol.

Volunteers will NOT be asked to change their drinking (or nondrinking) habits during the study.

Detailed Description

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Conditions

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Alcohol Drinking Depression Smoking Cessation Attention Deficit Disorder

Keywords

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Alcohol Bupropion Chlorzoxazone Interaction Induction Depression Smoking Drinking Cytochrome P450 Enzyme CYP2B6 CYP2E1 Pharmacogenetics Pharmacokinetics Pharmacodynamics Boston Tufts University

Study Design

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Allocation Method

NON_RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

TREATMENT

Blinding Strategy

NONE

Study Groups

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Trial Withdrawn 2

Trial Withdrawn 2

Group Type OTHER

Trial Withdrawn 2

Intervention Type OTHER

Trial Withdrawn 2

Trial Withdrawn 1

Trial Withdrawn 1

Group Type OTHER

Trial Withdrawn 1

Intervention Type OTHER

Trial Withdrawn 1

Interventions

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Trial Withdrawn 1

Trial Withdrawn 1

Intervention Type OTHER

Trial Withdrawn 2

Trial Withdrawn 2

Intervention Type OTHER

Eligibility Criteria

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Inclusion Criteria

* Healthy adults who are 21 - 55 years of age.
* Either 1) Moderate-to-heavy drinkers who drink on average more than 14 but less than 28 drinks per week; OR 2) adults who normally abstain from drinking alcohol.

Exclusion Criteria

* Participants who are currently taking prescription medications (including oral contraceptives)
* Pregnancy
* Body mass index (BMI) greater than 30
* History of seizures or eating disorders
Minimum Eligible Age

21 Years

Maximum Eligible Age

55 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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National Institute on Alcohol Abuse and Alcoholism (NIAAA)

NIH

Sponsor Role collaborator

Tufts University

OTHER

Sponsor Role lead

Responsible Party

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Responsibility Role SPONSOR

Principal Investigators

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David J. Greenblatt, MD

Role: PRINCIPAL_INVESTIGATOR

Tufts University; Chair of Department of Pharmacology and Experimental Therapeutics, Sackler School

Michael H. Court, BVsc, PhD

Role: PRINCIPAL_INVESTIGATOR

Tufts University, Department of Pharmacology and Experimental Therapeutics, Sackler School

Locations

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Tufts University School of Medicine

Boston, Massachusetts, United States

Site Status

Countries

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United States

References

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Hesse LM, Venkatakrishnan K, Court MH, von Moltke LL, Duan SX, Shader RI, Greenblatt DJ. CYP2B6 mediates the in vitro hydroxylation of bupropion: potential drug interactions with other antidepressants. Drug Metab Dispos. 2000 Oct;28(10):1176-83.

Reference Type BACKGROUND
PMID: 10997936 (View on PubMed)

Hesse LM, He P, Krishnaswamy S, Hao Q, Hogan K, von Moltke LL, Greenblatt DJ, Court MH. Pharmacogenetic determinants of interindividual variability in bupropion hydroxylation by cytochrome P450 2B6 in human liver microsomes. Pharmacogenetics. 2004 Apr;14(4):225-38. doi: 10.1097/00008571-200404000-00002.

Reference Type BACKGROUND
PMID: 15083067 (View on PubMed)

Other Identifiers

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F32AA015647-01A1

Identifier Type: NIH

Identifier Source: secondary_id

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1F32AA015647-01A1

Identifier Type: NIH

Identifier Source: secondary_id

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NIAAAGRE15647

Identifier Type: -

Identifier Source: org_study_id