The Influence of FP-10 on the Eradication Rates of H. Pylori by a Triple Therapy

NCT ID: NCT00281047

Last Updated: 2006-01-27

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

UNKNOWN

Clinical Phase

PHASE2/PHASE3

Total Enrollment

138 participants

Study Classification

INTERVENTIONAL

Study Start Date

2006-01-31

Study Completion Date

2006-05-31

Brief Summary

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FP-10 is a food ingredient derived from milk casein. FP-10 can inhibit H. pylori to attach to the gastric epithelium. FP-10 has been made clear to decrease the intragastric urease activity (which is assumed to be produced by H. pylori) measured by the urea breath test. FP-10 can also detach H. pylori from gastric epithelium. We have hypothesized that FP-10 increases the eradication rates by a triple therapy with a proton pump inhibitor, amoxicillin, and clarithromycin.

Detailed Description

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H. pylori -positive patients older than 15 years of age with gastritis, gastric ulcer, duodenal ulcer, or gastroduodenal ulcer are invited to participate in the study. These patients had endoscopically and histologically proven ulcers or active chronic gastritis and are all H. pylori-positive. Written informed consent to participation must be obtained from each patient before the study.

During gastroduodenoscopy, biopsy specimens obtained from both the antrum and the corpus of the greater curvature are subjected to the bacterial susceptibility to clarithromycin by culture test or measurements of 23S rRNA mutations at positions 2142 and 2143 (from adenine to guanine).

Patients are treated with 30 mg of lansoprazole bid, 200 mg of clarithromycin bid, and 750 mg of amoxicillin bid for one week. In addition, they take placebo bid, FP10 1 g bid, or FP-10 2 g bid (2 hour after breakfast and at the bed time) for the same one week. Administration of placebo, FP-10 1 g or FP-10 2 g are performed in a double blinded manner.

Eradication of H. pylori was confirmed by a 13C-urea breath test performed one month after eradication therapy. Throughout the study period, the investigators involved in the assessment of H. pylori eradication are blinded to susceptibility to clarithromycin H. pylori strains.

Conditions

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Helicobacter Pylori

Keywords

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Helicobacter pylori (H. pylori) FP-10 amoxicillin clarithromycin lansoprazole

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

TREATMENT

Blinding Strategy

DOUBLE

Interventions

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FP-10

Intervention Type DRUG

Eligibility Criteria

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Inclusion Criteria

H. pylori-positive patients who have never undergo the H. pylori eradication therapy -

Exclusion Criteria

Patients not infected with H. pylori, Patients who are allergic to amoxicillin, clarithromycin, lansoprazole, 13C-urea, or milk casein

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Minimum Eligible Age

15 Years

Maximum Eligible Age

90 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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Oita University

OTHER

Sponsor Role collaborator

Hamamatsu University

OTHER

Sponsor Role lead

Principal Investigators

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Takahisa Furuta, MD, PhD

Role: STUDY_DIRECTOR

Center for Clinical Research, Hamamatsu University School of Medicine

Kazunrai Murakami, MD, PhD

Role: STUDY_DIRECTOR

Department of Gastroenterology, Oita University Faculty of Medicine

Toshio Fujioka, MD, PhD

Role: STUDY_CHAIR

Oita University

Locations

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Oita Kouseiren Tsurumi Hospital

Beppu, Oita Prefecture, Japan

Site Status RECRUITING

University Hospital of Oita University Faculty of Medicine

Ōita, Oita Prefecture, Japan

Site Status RECRUITING

Senoo Clinic for Internal Medicine and Gastroenterology

Hamamatsu, Shizuoka, Japan

Site Status RECRUITING

University Hospital of Hamamatsu University School of Medicine

Hamamatsu, Shizuoka, Japan

Site Status RECRUITING

Matsushita Clinic

Hamamatsu, Shizuoka, Japan

Site Status RECRUITING

Kumagai Clinic for Internal Medicine and Gastroenterology

Hamamatsu, Shizuoka, Japan

Site Status RECRUITING

Nakajima Clinic

Kakegawa, Shizuoka, Japan

Site Status RECRUITING

Countries

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Japan

Central Contacts

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Takahisa Furuta, MD, PhD

Role: CONTACT

Phone: 81-53-435-2850

Email: [email protected]

Kazunari Murakami, MD, PhD

Role: CONTACT

Phone: 81-97-586-6193

Email: [email protected]

Facility Contacts

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Takayuki Nagai, MD. PhD

Role: primary

Kazunari Murakami, MD, PhD

Role: primary

Kazutaka Senoo, MD

Role: primary

Takahisa Furuta, MD, PhD

Role: primary

Fumiaki Matsushita, MD, PhD

Role: primary

Junichi Kumagai, MD, PhD

Role: primary

Hiroshi Nakamura, MD, PhD

Role: primary

References

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Furuta T, Sagehashi Y, Shirai N, Sugimoto M, Nakamura A, Kodaira M, Kenmotsu K, Nagano M, Egashira T, Ueda K, Yoneyama M, Ohashi K, Ishizaki T, Hishida A. Influence of CYP2C19 polymorphism and Helicobacter pylori genotype determined from gastric tissue samples on response to triple therapy for H pylori infection. Clin Gastroenterol Hepatol. 2005 Jun;3(6):564-73. doi: 10.1016/s1542-3565(04)00779-7.

Reference Type BACKGROUND
PMID: 15952098 (View on PubMed)

Shirai N, Furuta T, Sugimoto M, Nakamura A. [High dose dual PPI/AMPC therapy for the treatment of Helicobacter pylori infection after failure of usual standard triple PPI/AMPC/CAM therapy]. Nihon Rinsho. 2005 Nov;63 Suppl 11:438-41. No abstract available. Japanese.

Reference Type BACKGROUND
PMID: 16363575 (View on PubMed)

Okudaira K, Miura S, Furuta T, Sugimoto M, Shirai N. [Concomitant dosing of a H2 receptor antagonist (H2RA) with a triple therapy increases the cure rate of Helicobacter pylori infection]. Nihon Rinsho. 2005 Nov;63 Suppl 11:391-6. No abstract available. Japanese.

Reference Type BACKGROUND
PMID: 16363566 (View on PubMed)

Furuta T, Shirai N, Sugimoto M, Nakamura A, Hishida A, Ishizaki T. Influence of CYP2C19 pharmacogenetic polymorphism on proton pump inhibitor-based therapies. Drug Metab Pharmacokinet. 2005 Jun;20(3):153-67. doi: 10.2133/dmpk.20.153.

Reference Type BACKGROUND
PMID: 15988117 (View on PubMed)

Furuta T, Shirai N, Sugimoto M, Nakamura A, Okudaira K, Kajimura M, Hishida A. Effect of concomitant dosing of famotidine with lansoprazole on gastric acid secretion in relation to CYP2C19 genotype status. Aliment Pharmacol Ther. 2005 Jul 1;22(1):67-74. doi: 10.1111/j.1365-2036.2005.02523.x.

Reference Type BACKGROUND
PMID: 15963082 (View on PubMed)

Okudaira K, Furuta T, Shirai N, Sugimoto M, Miura S. Concomitant dosing of famotidine with a triple therapy increases the cure rates of Helicobacter pylori infections in patients with the homozygous extensive metabolizer genotype of CYP2C19. Aliment Pharmacol Ther. 2005 Feb 15;21(4):491-7. doi: 10.1111/j.1365-2036.2005.02353.x.

Reference Type BACKGROUND
PMID: 15710002 (View on PubMed)

Murakami K, Fujioka T. [Drug resistant H. pylori in Japan: general remarks]. Nihon Rinsho. 2005 Nov;63 Suppl 11:198-202. No abstract available. Japanese.

Reference Type BACKGROUND
PMID: 16363531 (View on PubMed)

Murakami K, Kodama M, Sato R, Okimoto T, Watanabe K, Fujioka T. Helicobacter pylori eradication and associated changes in the gastric mucosa. Expert Rev Anti Infect Ther. 2005 Oct;3(5):757-64. doi: 10.1586/14787210.3.5.757.

Reference Type BACKGROUND
PMID: 16207167 (View on PubMed)

Murakami K, Sato R, Okimoto T, Watanabe K, Nasu M, Fujioka T, Kodama M, Kagawa J. Influence of anti-ulcer drugs used in Japan on the result of (13)C-urea breath test for the diagnosis of Helicobacter pylori infection. J Gastroenterol. 2003;38(10):937-41. doi: 10.1007/s00535-003-1176-x.

Reference Type BACKGROUND
PMID: 14614600 (View on PubMed)

Hiramoto S, Itoh K, Shizuuchi S, Kawachi Y, Morishita Y, Nagase M, Suzuki Y, Nobuta Y, Sudou Y, Nakamura O, Kagaya I, Goshima H, Kodama Y, Icatro FC, Koizumi W, Saigenji K, Miura S, Sugiyama T, Kimura N. Melanoidin, a food protein-derived advanced maillard reaction product, suppresses Helicobacter pylori in vitro and in vivo. Helicobacter. 2004 Oct;9(5):429-35. doi: 10.1111/j.1083-4389.2004.00263.x.

Reference Type BACKGROUND
PMID: 15361082 (View on PubMed)

Murakami K, Sato R, Okimoto T, Nasu M, Fujioka T, Kodama M, Kagawa J. Efficacy of triple therapy comprising rabeprazole, amoxicillin and metronidazole for second-line Helicobacter pylori eradication in Japan, and the influence of metronidazole resistance. Aliment Pharmacol Ther. 2003 Jan;17(1):119-23. doi: 10.1046/j.1365-2036.2003.01401.x.

Reference Type BACKGROUND
PMID: 12492740 (View on PubMed)

Murakami K, Sato R, Okimoto T, Nasu M, Fujioka T, Kodama M, Kagawa J, Sato S, Abe H, Arita T. Eradication rates of clarithromycin-resistant Helicobacter pylori using either rabeprazole or lansoprazole plus amoxicillin and clarithromycin. Aliment Pharmacol Ther. 2002 Nov;16(11):1933-8. doi: 10.1046/j.1365-2036.2002.01368.x.

Reference Type BACKGROUND
PMID: 12390102 (View on PubMed)

Murakami K, Nasu M. [Clarithromycin (CAM)]. Nihon Rinsho. 2002 Feb;60 Suppl 2:667-70. No abstract available. Japanese.

Reference Type BACKGROUND
PMID: 11979867 (View on PubMed)

Murakami K, Nasu M. [Drug sensitivity test for Helicobacter pylori]. Nihon Rinsho. 2002 Feb;60 Suppl 2:350-3. No abstract available. Japanese.

Reference Type BACKGROUND
PMID: 11979806 (View on PubMed)

Murakami K, Fujioka T, Okimoto T, Sato R, Kodama M, Nasu M. Drug combinations with amoxycillin reduce selection of clarithromycin resistance during Helicobacter pylori eradication therapy. Int J Antimicrob Agents. 2002 Jan;19(1):67-70. doi: 10.1016/s0924-8579(01)00456-3.

Reference Type BACKGROUND
PMID: 11814770 (View on PubMed)

Asaka M, Satoh K, Sugano K, Sugiyama T, Takahashi S, Fukuda Y, Ota H, Murakami K, Kimura K, Shimoyama T. Guidelines in the management of Helicobacter pylori infection in Japan. Helicobacter. 2001 Sep;6(3):177-86. doi: 10.1046/j.1523-5378.2001.00027.x.

Reference Type BACKGROUND
PMID: 11683920 (View on PubMed)

Murakami K, Fujioka T, Kodama R, Kubota T, Tokieda M, Nasu M. Helicobacter pylori infection accelerates human gastric mucosal cell proliferation. J Gastroenterol. 1997 Apr;32(2):184-8. doi: 10.1007/BF02936365.

Reference Type BACKGROUND
PMID: 9085165 (View on PubMed)

Other Identifiers

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Hp.FP-10.01

Identifier Type: -

Identifier Source: org_study_id