Rosuvastatin Affecting Aortic Valve Endothelium

NCT ID: NCT00114491

Last Updated: 2006-10-31

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Total Enrollment

200 participants

Study Classification

OBSERVATIONAL

Study Start Date

2003-09-30

Study Completion Date

2005-05-31

Brief Summary

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Recent studies support the hypothesis that aortic stenosis (AS) develops due to atherosclerosis affecting the aortic valve endothelium. The study's aim was to assess Rosuvastatin on the hemodynamic progression and inflammatory markers of AS by treating low-density lipoprotein (LDL) in patients with AS according to the National Cholesterol Education Program Adult Treatment Panel III (NCEP-ATPIII) guidelines for one year.

Detailed Description

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Background: Recent retrospective studies support the hypothesis that statins slow the progression of aortic stenosis. The aim of this study was to assess the effect of Rosuvastatin on hemodynamic progression of aortic stenosis by treating patients with aortic stenosis and elevated LDL-cholesterol for 18 months.

Methods: We performed an open-label, prospective study evaluating 121 consecutive patients with asymptomatic moderate to severe aortic stenosis (AVA≥ 1.0 cm2), (mean age 73.7±8.9 years; 57 men and 64 women), treated with and without Rosuvastatin according to the NCEP-ATPIII guidelines. Echocardiographic, serum lipid, and inflammatory markers were measured at baseline and every 6 months for 18 months.

Conditions

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Atherosclerosis Hypercholesterolemia

Keywords

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Aortic Stenosis Statins Hypercholesterolemia

Study Design

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Observational Model Type

DEFINED_POPULATION

Study Time Perspective

PROSPECTIVE

Interventions

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Rosuvastatin

Intervention Type DRUG

Eligibility Criteria

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Inclusion Criteria

* Asymptomatic AS
* Normal ejection fraction
* Elevated LDL \>130 mg/dl

Exclusion Criteria

* Echocardiographic evidence of rheumatic mitral valve disease,
* Previous statin therapy,
* Congenital heart disease (bicuspid aortic valve),
* Subaortic obstruction,
* Creatinine ≥ 2,0 mg/dl (to avoid the potential confounder of an elevated serum \[CaP04\]),
* Evidence of liver disease,
* Greater than mild aortic regurgitation and previous aortic valve surgery
Minimum Eligible Age

50 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

Yes

Sponsors

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Northwestern University

OTHER

Sponsor Role collaborator

Hospital Pedro Hispano

OTHER

Sponsor Role lead

Principal Investigators

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Luis M Moura

Role: PRINCIPAL_INVESTIGATOR

Hospital Pedro Hispano

Locations

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Hospital Pedro Hispano

Porto, , Portugal

Site Status

Countries

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Portugal

References

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Rajamannan NM, Subramaniam M, Stock SR, Stone NJ, Springett M, Ignatiev KI, McConnell JP, Singh RJ, Bonow RO, Spelsberg TC. Atorvastatin inhibits calcification and enhances nitric oxide synthase production in the hypercholesterolaemic aortic valve. Heart. 2005 Jun;91(6):806-10. doi: 10.1136/hrt.2003.029785.

Reference Type RESULT
PMID: 15894785 (View on PubMed)

Makkena B, Salti H, Subramaniam M, Thennapan S, Bonow RH, Caira F, Bonow RO, Spelsberg TC, Rajamannan NM. Atorvastatin decreases cellular proliferation and bone matrix expression in the hypercholesterolemic mitral valve. J Am Coll Cardiol. 2005 Feb 15;45(4):631-3. doi: 10.1016/j.jacc.2004.11.023. No abstract available.

Reference Type RESULT
PMID: 15708716 (View on PubMed)

Rajamannan NM. Is it time for medical therapy for aortic valve disease? Expert Rev Cardiovasc Ther. 2004 Nov;2(6):845-54. doi: 10.1586/14779072.2.6.845.

Reference Type RESULT
PMID: 15500430 (View on PubMed)

Rajamannan NM, Edwards WD, Spelsberg TC. Hypercholesterolemic aortic-valve disease. N Engl J Med. 2003 Aug 14;349(7):717-8. doi: 10.1056/NEJMc031360. No abstract available.

Reference Type RESULT
PMID: 12917318 (View on PubMed)

Otto CM. Why is aortic sclerosis associated with adverse clinical outcomes? J Am Coll Cardiol. 2004 Jan 21;43(2):176-8. doi: 10.1016/j.jacc.2003.10.027. No abstract available.

Reference Type RESULT
PMID: 14736433 (View on PubMed)

Rajamannan NM, Otto CM. Targeted therapy to prevent progression of calcific aortic stenosis. Circulation. 2004 Sep 7;110(10):1180-2. doi: 10.1161/01.CIR.0000140722.85490.EA. No abstract available.

Reference Type RESULT
PMID: 15353511 (View on PubMed)

Rajamannan NM, Subramaniam M, Rickard D, Stock SR, Donovan J, Springett M, Orszulak T, Fullerton DA, Tajik AJ, Bonow RO, Spelsberg T. Human aortic valve calcification is associated with an osteoblast phenotype. Circulation. 2003 May 6;107(17):2181-4. doi: 10.1161/01.CIR.0000070591.21548.69. Epub 2003 Apr 28.

Reference Type RESULT
PMID: 12719282 (View on PubMed)

Moura LM, Ramos SF, Zamorano JL, Barros IM, Azevedo LF, Rocha-Goncalves F, Rajamannan NM. Rosuvastatin affecting aortic valve endothelium to slow the progression of aortic stenosis. J Am Coll Cardiol. 2007 Feb 6;49(5):554-61. doi: 10.1016/j.jacc.2006.07.072. Epub 2007 Jan 22.

Reference Type DERIVED
PMID: 17276178 (View on PubMed)

Other Identifiers

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22352

Identifier Type: -

Identifier Source: org_study_id