dnaJ Peptide for Relieving Rheumatoid Arthritis

NCT ID: NCT00000435

Last Updated: 2007-07-31

Study Results

Results pending

The study team has not published outcome measurements, participant flow, or safety data for this trial yet. Check back later for updates.

Basic Information

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Recruitment Status

COMPLETED

Clinical Phase

PHASE2

Total Enrollment

160 participants

Study Classification

INTERVENTIONAL

Study Start Date

1999-09-30

Study Completion Date

2004-09-30

Brief Summary

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A small protein called dnaJ peptide may help people with rheumatoid arthritis (RA) by preventing their immune system cells from attacking their own tissues. The purpose of this study is to determine if small amounts of dnaJ peptide can "re-educate" immune cells in people with RA so that the cells stop attacking joint tissues.

Detailed Description

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Immune modulation is a promising new approach for the treatment of RA. Studies have shown that immune cells in the joints of people in the early stages of RA react strongly against dnaJ peptides from bacteria. These immune cells may also cross-react with human dnaJ peptides in the joints to cause inflammation. dnaJ may help RA by "re-educating" the immune system and dampening the abnormal inflammatory immune response in RA.

This study will last 7 months. Participants will be randomly assigned to receive either dnaJ or placebo by mouth. At screening, participants will have medical history, physical, and medication assessment. At screening, at 6 study visits every month after the start of treatment, and at 1 month follow-up, participants will have a joint exam, blood and urine collection, and will fill out a questionnaire about their condition.

Conditions

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Rheumatoid Arthritis

Keywords

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RA Immune Modulation Oral Tolerance Peptide dnaJ

Study Design

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Allocation Method

RANDOMIZED

Intervention Model

PARALLEL

Primary Study Purpose

TREATMENT

Blinding Strategy

DOUBLE

Study Groups

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A

Subjects randomized to arm A received 25mg/day po of placebo

Group Type PLACEBO_COMPARATOR

None-placebo

Intervention Type DRUG

placebo was taken in pill form at 25mg/day for 6 months

B

Subjects randomized to Arm B received 25mg/day po of peptide dnaJP1

Group Type ACTIVE_COMPARATOR

dnaJ peptide

Intervention Type DRUG

dnaJP1 was taken in pill form at 25mg/day for 6 months

Interventions

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dnaJ peptide

dnaJP1 was taken in pill form at 25mg/day for 6 months

Intervention Type DRUG

None-placebo

placebo was taken in pill form at 25mg/day for 6 months

Intervention Type DRUG

Eligibility Criteria

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Inclusion Criteria

* Active rheumatoid arthritis as defined by the revised American College of Rheumatology (ACR) 1987 criteria. Evidence of active disease will be based on at least six swollen or nine tender joints.
* Diagnosis of rheumatoid arthritis of less than 5 years
* Reactivity to dnaJ
* Agree to use acceptable methods of contraception
* Able to understand and sign informed consent

Exclusion Criteria

* Patients taking more 7.5 mg of prednisone or disease modifying agents other than hydrochloroquine or sulfasalazine (i.e., gold, penicillamine, azathioprine, cyclophosphamide, methotrexate, cyclosporine, or anti-TNF agents)
* Serum creatinine greater than 1.5 mg/dl
* SGOT less than SGPT
* Alkaline phosphatase greater than 2 times age/sex adjusted normal values
* Hematocrit of less than 30
* Platelets less than 130,000
* History of lymphoma
* Any active malignancy or cancer requiring treatment in the last 5 years, except for nonmelanoma skin cancers and carcinoma of the cervix in situ
* Medical or psychiatric condition or active serious infection
* Pregnant or breastfeeding
Minimum Eligible Age

18 Years

Maximum Eligible Age

85 Years

Eligible Sex

ALL

Accepts Healthy Volunteers

No

Sponsors

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National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)

NIH

Sponsor Role lead

Principal Investigators

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Salvatore Albani, MD

Role: PRINCIPAL_INVESTIGATOR

University of California, San Diego

Locations

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University of Arizona Health Sciences Center

Tucson, Arizona, United States

Site Status

University of California, Irvine Medical Center

Orange, California, United States

Site Status

Stanford University

Palo Alto, California, United States

Site Status

Denver Arthritis Center

Denver, Colorado, United States

Site Status

Johns Hopkins University

Baltimore, Maryland, United States

Site Status

Mayo Clinic

Rochester, Minnesota, United States

Site Status

Guthrie Clinic

Sayre, Pennsylvania, United States

Site Status

Virginia Mason Research Center

Seattle, Washington, United States

Site Status

Countries

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United States

References

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Prakken BJ, Samodal R, Le TD, Giannoni F, Yung GP, Scavulli J, Amox D, Roord S, de Kleer I, Bonnin D, Lanza P, Berry C, Massa M, Billetta R, Albani S. Epitope-specific immunotherapy induces immune deviation of proinflammatory T cells in rheumatoid arthritis. Proc Natl Acad Sci U S A. 2004 Mar 23;101(12):4228-33. doi: 10.1073/pnas.0400061101. Epub 2004 Mar 15.

Reference Type BACKGROUND
PMID: 15024101 (View on PubMed)

Koffeman EC, Genovese M, Amox D, Keogh E, Santana E, Matteson EL, Kavanaugh A, Molitor JA, Schiff MH, Posever JO, Bathon JM, Kivitz AJ, Samodal R, Belardi F, Dennehey C, van den Broek T, van Wijk F, Zhang X, Zieseniss P, Le T, Prakken BA, Cutter GC, Albani S. Epitope-specific immunotherapy of rheumatoid arthritis: clinical responsiveness occurs with immune deviation and relies on the expression of a cluster of molecules associated with T cell tolerance in a double-blind, placebo-controlled, pilot phase II trial. Arthritis Rheum. 2009 Nov;60(11):3207-16. doi: 10.1002/art.24916.

Reference Type DERIVED
PMID: 19877047 (View on PubMed)

Other Identifiers

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NIAMS-042

Identifier Type: -

Identifier Source: secondary_id

N01 AR92241

Identifier Type: -

Identifier Source: org_study_id